Kashleva相关文献与解析，生知库 | 链接生命科学的每一步

Role of Bcr1-activated genes Hwp1 and Hyr1 in Candida albicans oral mucosal biofilms and neutrophil evasion

Bcr1激活基因Hwp1和Hyr1在白色念珠菌口腔黏膜生物膜和中性粒细胞逃避中的作用

doi:10.1371/journal.pone.0016218

Dwivedi, Prabhat; Thompson, Angela; Xie, Zhihong; Kashleva, Helena; Ganguly, Shantanu; Mitchell, Aaron P; Dongari-Bagtzoglou, Anna

Characterization of mucosal Candida albicans biofilms

黏膜白色念珠菌生物膜的特征分析

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0007967

Dongari-Bagtzoglou, Anna; Kashleva, Helena; Dwivedi, Prabhat; Diaz, Patricia; Vasilakos, John

微生物学

发表日期：2009

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Cytotoxic and cytokine-inducing properties of Candida glabrata in single and mixed oral infection models

念珠菌在单一和混合口腔感染模型中的细胞毒性和细胞因子诱导特性

期刊:Microbial Pathogenesis

影响因子:3.5

doi:10.1016/j.micpath.2006.12.003

Li, Lulu; Kashleva, Helena; Dongari-Bagtzoglou, Anna

Mucosal tissue invasion by Candida albicans is associated with E-cadherin degradation, mediated by transcription factor Rim101p and protease Sap5p

白色念珠菌侵袭黏膜组织与E-钙黏蛋白降解有关，该降解过程由转录因子Rim101p和蛋白酶Sap5p介导。

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.00054-07

Villar, C C; Kashleva, H; Nobile, C J; Mitchell, A P; Dongari-Bagtzoglou, A

蛋白降解

发表日期：2007

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Development of a highly reproducible three-dimensional organotypic model of the oral mucosa

建立高度可重复的口腔黏膜三维器官模型

期刊:Nature Protocols

影响因子:13.1

doi:10.1038/nprot.2006.323

Anna Dongari-Bagtzoglou, Helena Kashleva

发表日期：2006

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Invasive phenotype of Candida albicans affects the host proinflammatory response to infection

白色念珠菌的侵袭性表型会影响宿主对感染的促炎反应。

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.73.8.4588-4595.2005

Villar, C C; Kashleva, H; Mitchell, A P; Dongari-Bagtzoglou, A

炎症/感染

发表日期：2005

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T cell vaccination induces T cell receptor Vbeta-specific Qa-1-restricted regulatory CD8(+) T cells

T细胞疫苗接种诱导T细胞受体Vβ特异性Qa-1限制性调节性CD8(+) T细胞

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.95.8.4533

Jiang, H; Kashleva, H; Xu, L X; Forman, J; Flaherty, L; Pernis, B; Braunstein, N S; Chess, L

A quantitative real-time RT-PCR assay for mature C. albicans biofilms

成熟白色念珠菌生物膜的定量实时RT-PCR检测

Role of Bcr1-activated genes Hwp1 and Hyr1 in Candida albicans oral mucosal biofilms and neutrophil evasion

Bcr1激活基因Hwp1和Hyr1在白色念珠菌口腔黏膜生物膜和中性粒细胞逃避中的作用

Characterization of mucosal Candida albicans biofilms

黏膜白色念珠菌生物膜的特征分析

Cytotoxic and cytokine-inducing properties of Candida glabrata in single and mixed oral infection models

念珠菌在单一和混合口腔感染模型中的细胞毒性和细胞因子诱导特性

Mucosal tissue invasion by Candida albicans is associated with E-cadherin degradation, mediated by transcription factor Rim101p and protease Sap5p

白色念珠菌侵袭黏膜组织与E-钙黏蛋白降解有关，该降解过程由转录因子Rim101p和蛋白酶Sap5p介导。

Development of a highly reproducible three-dimensional organotypic model of the oral mucosa

建立高度可重复的口腔黏膜三维器官模型

Invasive phenotype of Candida albicans affects the host proinflammatory response to infection

白色念珠菌的侵袭性表型会影响宿主对感染的促炎反应。

T cell vaccination induces T cell receptor Vbeta-specific Qa-1-restricted regulatory CD8(+) T cells

T细胞疫苗接种诱导T细胞受体Vβ特异性Qa-1限制性调节性CD8(+) T细胞