日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Pilot study of modified LMB-based therapy for children with ataxia-telangiectasia and advanced stage high grade mature B-cell malignancies

改良型LMB疗法治疗伴有毛细血管扩张性共济失调和晚期高级别成熟B细胞恶性肿瘤的儿童的试点研究

Sandlund, J T; Hudson, M M; Kennedy, W; Onciu, M; Kastan, M B

ATM-dependent phosphorylation of Mdm2 on serine 395: role in p53 activation by DNA damage.

ATM 依赖的 Mdm2 在丝氨酸 395 上的磷酸化:在 DNA 损伤激活 p53 中的作用

Maya R, Balass M, Kim S T, Shkedy D, Leal J F, Shifman O, Moas M, Buschmann T, Ronai Z, Shiloh Y, Kastan M B, Katzir E, Oren M

Defective potassium currents in ataxia telangiectasia fibroblasts

共济失调毛细血管扩张症成纤维细胞中钾电流缺陷

Rhodes, N; D'Souza, T; Foster, C D; Ziv, Y; Kirsch, D G; Shiloh, Y; Kastan, M B; Reinhart, P H; Gilmer, T M

DNA damage induces phosphorylation of the amino terminus of p53

DNA损伤诱导p53氨基末端磷酸化

Siliciano, J D; Canman, C E; Taya, Y; Sakaguchi, K; Appella, E; Kastan, M B

Fragments of ATM which have dominant-negative or complementing activity

具有显性负效应或互补活性的ATM片段

Morgan, S E; Lovly, C; Pandita, T K; Shiloh, Y; Kastan, M B

Control of G1 arrest after DNA damage

DNA损伤后G1期阻滞的控制

Kastan, M B; Kuerbitz, S J

Human papillomavirus 16 E6 expression disrupts the p53-mediated cellular response to DNA damage

人乳头瘤病毒16 E6的表达会破坏p53介导的细胞对DNA损伤的反应。

Kessis, T D; Slebos, R J; Nelson, W G; Kastan, M B; Plunkett, B S; Han, S M; Lorincz, A T; Hedrick, L; Cho, K R

Wild-type p53 is a cell cycle checkpoint determinant following irradiation

野生型p53是辐射后细胞周期检查点的决定因素。

Kuerbitz, S J; Plunkett, B S; Walsh, W V; Kastan, M B