Katharina E Hayer相关文献与解析，生知库

An Alternatively Spliced Gain-of-Function NT5C2 Isoform Contributes to Chemoresistance in Acute Lymphoblastic Leukemia

选择性剪接的获得功能 NT5C2 亚型导致急性淋巴细胞白血病产生化学耐药性

Manuel Torres-Diz, Clara Reglero, Catherine D Falkenstein, Annette Castro, Katharina E Hayer, Caleb M Radens, Mathieu Quesnel-Vallières, Zhiwei Ang, Priyanka Sehgal, Marilyn M Li, Yoseph Barash, Sarah K Tasian, Adolfo Ferrando, Andrei Thomas-Tikhonenko

免疫

The Cyclin D3 Protein Enforces Monogenic TCRβ Expression by Mediating TCRβ Protein-Signaled Feedback Inhibition of Vβ Recombination

细胞周期蛋白 D3 蛋白通过介导 TCRβ 蛋白信号反馈抑制 Vβ 重组来促进单基因 TCRβ 表达

期刊:Journal of Immunology

影响因子:3.6

doi:10.4049/jimmunol.2300623

Erica J Culberson, Kymberle C Shields, Rebecca A Glynn, Brittney M Allyn, Katharina E Hayer, Craig H Bassing

ATM-dependent Phosphorylation of Nemo SQ Motifs Is Dispensable for Nemo-mediated Gene Expression Changes in Response to DNA Double-Strand Breaks

ATM依赖的Nemo SQ基序磷酸化对于Nemo介导的DNA双链断裂诱导的基因表达变化并非必需

期刊:

影响因子:3.6

doi:PMC11348802

Rebecca A Glynn，Katharina E Hayer ，Craig H Bassing

Characterization of aberrant splicing in pediatric central nervous system tumors reveals CLK1 as a candidate oncogenic dependency

儿童中枢神经系统肿瘤异常剪接的表征揭示 CLK1 为候选致癌依赖性

期刊:

影响因子:

doi:10.1101/2024.08.03.606419

Ammar S Naqvi, Ryan J Corbett, Priyanka Seghal, Karina L Conkrite, Komal S Rathi, Brian M Ennis, Katharina E Hayer, Bo Zhang, Miguel A Brown, Daniel P Miller, Adam A Kraya, Joseph M Dybas, Zhuangzhuang Geng, Christopher Blackden, Shehbeel Arif, Antonia Chroni, Aditya Lahiri, Madison L Hollawell, Phi

Alternative splicing of its 5'-UTR limits CD20 mRNA translation and enables resistance to CD20-directed immunotherapies

5'-UTR 的选择性剪接限制了 CD20 mRNA 翻译，并导致对 CD20 靶向免疫疗法的耐药性

期刊:Blood

影响因子:21

doi:10.1182/blood.2023020400

Zhiwei Ang, Luca Paruzzo, Katharina E Hayer, Carolin Schmidt, Manuel Torres Diz, Feng Xu, Urvi Zankharia, Yunlin Zhang, Samantha Soldan, Sisi Zheng, Catherine D Falkenstein, Joseph P Loftus, Scarlett Y Yang, Mukta Asnani, Patricia King Sainos, Vinodh Pillai, Emeline Chong, Marilyn M Li, Sarah K Tasi

免疫

发表日期：2023

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FBXW7β isoform drives transcriptional activation of the proinflammatory TNF cluster in human pro-B cells

FBXW7β 同工酶驱动人类前 B 细胞中促炎性 TNF 簇的转录激活

期刊:Blood Advances

影响因子:7.4

doi:10.1182/bloodadvances.2022007910

Scarlett Y Yang, Katharina E Hayer, Hossein Fazelinia, Lynn A Spruce, Mukta Asnani, Kathryn L Black, Ammar S Naqvi, Vinodh Pillai, Yoseph Barash, Kojo S J Elenitoba-Johnson, Andrei Thomas-Tikhonenko

FBXW7β isoform drives transcriptional activation of the proinflammatory TNF cluster in human pro-B cells

FBXW7β亚型驱动人类前B细胞中促炎性TNF簇的转录激活

期刊:

影响因子:7.4

doi:PMC10111352

Scarlett Y Yang，Katharina E Hayer，Hossein Fazelinia，Lynn A Spruce，Mukta Asnani，Kathryn L Black，Ammar S Naqvi，Vinodh Pillai，Yoseph Barash，Kojo S J Elenitoba-Johnson，Andrei Thomas-Tikhonenko

Intestinal transit-amplifying cells require METTL3 for growth factor signaling and cell survival

肠道转运扩增细胞需要METTL3来进行生长因子信号传导和细胞存活。

期刊:JCI Insight

影响因子:6.3

doi:10.1172/jci.insight.171657

Charles H Danan,Kaitlyn E Naughton,Katharina E Hayer,Sangeevan Vellappan,Emily A McMillan,Yusen Zhou,Rina Matsuda,Shaneice K Nettleford,Kay Katada,Louis R Parham,Xianghui Ma,Afrah Chowdhury,Benjamin J Wilkins,Premal Shah,Matthew D Weitzman,Kathryn E Hamilton

Intestinal transit amplifying cells require METTL3 for growth factor signaling, KRAS expression, and cell survival

肠道转运扩增细胞需要 METTL3 进行生长因子信号传导、KRAS 表达和细胞存活

期刊:

影响因子:

doi:10.1101/2023.04.06.535853

Charles H Danan, Kaitlyn E Naughton, Katharina E Hayer, Sangeevan Vellappan, Emily A McMillan, Yusen Zhou, Rina Matsuda, Shaneice K Nettleford, Kay Katada, Louis R Parham, Xianghui Ma, Afrah Chowdhury, Benjamin J Wilkins, Premal Shah, Matthew D Weitzman, Kathryn E Hamilton

Locus folding mechanisms determine modes of antigen receptor gene assembly

基因座折叠机制决定抗原受体基因组装模式

An Alternatively Spliced Gain-of-Function NT5C2 Isoform Contributes to Chemoresistance in Acute Lymphoblastic Leukemia

选择性剪接的获得功能 NT5C2 亚型导致急性淋巴细胞白血病产生化学耐药性

The Cyclin D3 Protein Enforces Monogenic TCRβ Expression by Mediating TCRβ Protein-Signaled Feedback Inhibition of Vβ Recombination

细胞周期蛋白 D3 蛋白通过介导 TCRβ 蛋白信号反馈抑制 Vβ 重组来促进单基因 TCRβ 表达

ATM-dependent Phosphorylation of Nemo SQ Motifs Is Dispensable for Nemo-mediated Gene Expression Changes in Response to DNA Double-Strand Breaks

ATM依赖的Nemo SQ基序磷酸化对于Nemo介导的DNA双链断裂诱导的基因表达变化并非必需

Characterization of aberrant splicing in pediatric central nervous system tumors reveals CLK1 as a candidate oncogenic dependency

儿童中枢神经系统肿瘤异常剪接的表征揭示 CLK1 为候选致癌依赖性

Alternative splicing of its 5'-UTR limits CD20 mRNA translation and enables resistance to CD20-directed immunotherapies

5'-UTR 的选择性剪接限制了 CD20 mRNA 翻译，并导致对 CD20 靶向免疫疗法的耐药性

FBXW7β isoform drives transcriptional activation of the proinflammatory TNF cluster in human pro-B cells

FBXW7β 同工酶驱动人类前 B 细胞中促炎性 TNF 簇的转录激活

FBXW7β isoform drives transcriptional activation of the proinflammatory TNF cluster in human pro-B cells

FBXW7β亚型驱动人类前B细胞中促炎性TNF簇的转录激活

Intestinal transit-amplifying cells require METTL3 for growth factor signaling and cell survival

肠道转运扩增细胞需要METTL3来进行生长因子信号传导和细胞存活。

Intestinal transit amplifying cells require METTL3 for growth factor signaling, KRAS expression, and cell survival

肠道转运扩增细胞需要 METTL3 进行生长因子信号传导、KRAS 表达和细胞存活