日期 影响因子
日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:
–

Lipid flippases ATP9A and ATP9B form a complex and contribute to the exocytic pathway from the Golgi

脂质翻转酶ATP9A和ATP9B形成复合物，参与高尔基体的胞吐途径。

期刊:Life Science Alliance
影响因子:2.9
doi:10.26508/lsa.202403163
Yagi, Tsukasa; Nakabuchi, Riki; Muranaka, Yumeka; Tanaka, Gaku; Katoh, Yohei; Nakayama, Kazuhisa; Takatsu, Hiroyuki; Shin, Hye-Won
发表日期：2025
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Coordinated roles of the CEP164 homodimer and TTBK2 are required for recruitment of the IFT machinery to the mother centriole for ciliogenesis

CEP164同源二聚体和TTBK2的协同作用对于将IFT机制募集到母中心粒以进行纤毛发生是必需的。

期刊:Molecular Biology of the Cell
影响因子:2.7
doi:10.1091/mbc.E24-12-0536
Mori, Kohei; Yamazaki, Shingo; Yoshida, Kosei; Shirota, Rin; Chiba, Shuhei; Shin, Hye-Won; Katoh, Yohei; Nakayama, Kazuhisa
发表日期：2025
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Mutually independent and cilia-independent assembly of IFT-A and IFT-B complexes at mother centriole

在母中心粒处，IFT-A 和 IFT-B 复合物的组装是相互独立且不依赖于纤毛的。

期刊:Molecular Biology of the Cell
影响因子:2.7
doi:10.1091/mbc.E24-11-0509
Tasaki, Koshi; Satoda, Yuuki; Chiba, Shuhei; Shin, Hye-Won; Katoh, Yohei; Nakayama, Kazuhisa
发表日期：2025
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Assembly and mother centriole recruitment of IFT-B subcomplexes to form IFT-B holocomplex

IFT-B亚复合物的组装和母中心粒募集，形成IFT-B全复合物。

期刊:Cell Structure and Function
影响因子:2.2
doi:10.1247/csf.25027
Tasaki, Koshi; Katoh, Yohei; Shin, Hye-Won; Nakayama, Kazuhisa
发表日期：2025
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Positive regulation of Hedgehog signaling via phosphorylation of GLI2/GLI3 by DYRK2 kinase

DYRK2激酶通过磷酸化GLI2/GLI3正向调控Hedgehog信号通路

期刊:Proceedings of the National Academy of Sciences of the United States of America
影响因子:9.1
doi:10.1073/pnas.2320070121
Yoshida, Saishu; Kawamura, Akira; Aoki, Katsuhiko; Wiriyasermkul, Pattama; Sugimoto, Shinya; Tomiyoshi, Junnosuke; Tajima, Ayasa; Ishida, Yamato; Katoh, Yohei; Tsukada, Takehiro; Tsuneoka, Yousuke; Yamada, Kohji; Nagamori, Shushi; Nakayama, Kazuhisa; Yoshida, Kiyotsugu
DYR
信号转导
Hedgehog
DYRK2
磷酸化
发表日期：2024
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Combinations of deletion and missense variations of the dynein-2 DYNC2LI1 subunit found in skeletal ciliopathies cause ciliary defects

骨骼纤毛病中发现的动力蛋白-2 DYNC2LI1亚基的缺失和错义变异组合会导致纤毛缺陷。

期刊:Scientific Reports
影响因子:3.9
doi:10.1038/s41598-021-03950-0
Qiu, Hantian; Tsurumi, Yuta; Katoh, Yohei; Nakayama, Kazuhisa
发表日期：2022
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Anterograde trafficking of ciliary MAP kinase-like ICK/CILK1 by the intraflagellar transport machinery is required for intraciliary retrograde protein trafficking

纤毛内运输机制介导的纤毛MAP激酶样蛋白ICK/CILK1的顺行运输是纤毛内逆行蛋白运输所必需的。

期刊:Journal of Biological Chemistry
影响因子:3.9
doi:10.1074/jbc.RA120.014142
Nakamura, Kentaro; Noguchi, Tatsuro; Takahara, Mariko; Omori, Yoshihiro; Furukawa, Takahisa; Katoh, Yohei; Nakayama, Kazuhisa
信号转导
CILK1
ILK
ILK1
发表日期：2020
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Formation of the B9-domain protein complex MKS1-B9D2-B9D1 is essential as a diffusion barrier for ciliary membrane proteins

B9结构域蛋白复合物MKS1-B9D2-B9D1的形成对于纤毛膜蛋白的扩散屏障至关重要。

期刊:Molecular Biology of the Cell
影响因子:2.7
doi:10.1091/mbc.E20-03-0208
Okazaki, Misato; Kobayashi, Takuya; Chiba, Shuhei; Takei, Ryota; Liang, Luxiaoxue; Nakayama, Kazuhisa; Katoh, Yohei
发表日期：2020
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Interactions of the dynein-2 intermediate chain WDR34 with the light chains are required for ciliary retrograde protein trafficking

动力蛋白-2中间链WDR34与轻链的相互作用是纤毛逆行蛋白运输所必需的。

期刊:Molecular Biology of the Cell
影响因子:2.7
doi:10.1091/mbc.E18-10-0678
Tsurumi, Yuta; Hamada, Yuki; Katoh, Yohei; Nakayama, Kazuhisa
发表日期：2019
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BBS1 is involved in retrograde trafficking of ciliary GPCRs in the context of the BBSome complex

BBS1参与BBSome复合物中纤毛GPCR的逆向运输。

期刊:PLoS One
影响因子:2.6
doi:10.1371/journal.pone.0195005
Nozaki, Shohei; Katoh, Yohei; Kobayashi, Takuya; Nakayama, Kazuhisa
GPCR/Calcium/cAMP
发表日期：2018
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