Katrin Daniel相关文献与解析，生知库 | 链接生命科学的每一步

The Reproduction Rate of Peptide Transporter PEPT-1 Deficient C. elegans Is Dependent on Dietary Glutamate Supply

缺乏肽转运蛋白 PEPT-1 的秀丽隐杆线虫的繁殖率取决于膳食谷氨酸的供应

doi:10.3389/fmolb.2018.00109

Spanier, Britta; Wallwitz, Jacqueline; Zapoglou, Despoina; Idrissou, Bio Maria Ghéo; Fischer, Christine; Troll, Martina; Petzold, Katrin; Daniel, Hannelore

C. elegans

发表日期：2018

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Quantitative Cell Cycle Analysis Based on an Endogenous All-in-One Reporter for Cell Tracking and Classification

基于内源性一体化报告基因的定量细胞周期分析，用于细胞追踪和分类

期刊:Cell Reports

影响因子:7.5

doi:10.1016/j.celrep.2017.05.022

Thomas Zerjatke, Igor A Gak, Dilyana Kirova, Markus Fuhrmann, Katrin Daniel, Magdalena Gonciarz, Doris Müller, Ingmar Glauche, Jörg Mansfeld

The enigmatic meiotic dense body and its newly discovered component, SCML1, are dispensable for fertility and gametogenesis in mice

神秘的减数分裂密体及其新发现的成分 SCML1 对小鼠的生育力和配子发生并非必不可少

期刊:Chromosoma

影响因子:2.5

doi:10.1007/s00412-016-0598-1

Frantzeskos Papanikos, Katrin Daniel, Angelique Goercharn-Ramlal, Ji-Feng Fei, Thomas Kurth, Lukasz Wojtasz, Ihsan Dereli, Jun Fu, Josef Penninger, Bianca Habermann, Azim Surani, A Francis Stewart, Attila Toth

信号转导

发表日期：2017

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Implementation of meiosis prophase I programme requires a conserved retinoid-independent stabilizer of meiotic transcripts

减数分裂前期 I 程序的实施需要保守的类视黄酸非依赖性减数分裂转录本稳定剂

期刊:Nature Communications

影响因子:14.7

doi:10.1038/ncomms10324

Emilie Abby, Sophie Tourpin, Jonathan Ribeiro, Katrin Daniel, Sébastien Messiaen, Delphine Moison, Justine Guerquin, Jean-Charles Gaillard, Jean Armengaud, Francina Langa, Attila Toth, Emmanuelle Martini, Gabriel Livera

发表日期：2016

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MEI4 – a central player in the regulation of meiotic DNA double-strand break formation in the mouse

MEI4 – 调节小鼠减数分裂 DNA 双链断裂形成的核心因子

期刊:Journal of Cell Science

影响因子:3.3

doi:10.1242/jcs.165464

Rajeev Kumar, Norbert Ghyselinck, Kei-ichiro Ishiguro, Yoshinori Watanabe, Anna Kouznetsova, Christer Höög, Edward Strong, John Schimenti, Katrin Daniel, Attila Toth, Bernard de Massy

Meiotic homologue alignment and its quality surveillance are controlled by mouse HORMAD1

减数分裂同源物比对及其质量监控由小鼠HORMAD1控制

期刊:Nature Cell Biology

影响因子:17.3

doi:10.1038/ncb2213

Katrin Daniel, Julian Lange, Khaled Hached, Jun Fu, Konstantinos Anastassiadis, Ignasi Roig, Howard J Cooke, A Francis Stewart, Katja Wassmann, Maria Jasin, Scott Keeney, Attila Tóth

Mouse HORMAD1 and HORMAD2, two conserved meiotic chromosomal proteins, are depleted from synapsed chromosome axes with the help of TRIP13 AAA-ATPase

小鼠 HORMAD1 和 HORMAD2 是两种保守的减数分裂染色体蛋白，在 TRIP13 AAA-ATPase 的帮助下从突触染色体轴上消失

期刊:PLoS Genetics

影响因子:

doi:10.1371/journal.pgen.1000702

Lukasz Wojtasz, Katrin Daniel, Ignasi Roig, Ewelina Bolcun-Filas, Huiling Xu, Verawan Boonsanay, Christian R Eckmann, Howard J Cooke, Maria Jasin, Scott Keeney, Michael J McKay, Attila Toth

Conditional control of fluorescent protein degradation by an auxin-dependent nanobody

生长素依赖性纳米抗体对荧光蛋白降解的条件控制

The Reproduction Rate of Peptide Transporter PEPT-1 Deficient C. elegans Is Dependent on Dietary Glutamate Supply

缺乏肽转运蛋白 PEPT-1 的秀丽隐杆线虫的繁殖率取决于膳食谷氨酸的供应

Quantitative Cell Cycle Analysis Based on an Endogenous All-in-One Reporter for Cell Tracking and Classification

基于内源性一体化报告基因的定量细胞周期分析，用于细胞追踪和分类

The enigmatic meiotic dense body and its newly discovered component, SCML1, are dispensable for fertility and gametogenesis in mice

神秘的减数分裂密体及其新发现的成分 SCML1 对小鼠的生育力和配子发生并非必不可少

Implementation of meiosis prophase I programme requires a conserved retinoid-independent stabilizer of meiotic transcripts

减数分裂前期 I 程序的实施需要保守的类视黄酸非依赖性减数分裂转录本稳定剂

MEI4 – a central player in the regulation of meiotic DNA double-strand break formation in the mouse

MEI4 – 调节小鼠减数分裂 DNA 双链断裂形成的核心因子

Meiotic homologue alignment and its quality surveillance are controlled by mouse HORMAD1

减数分裂同源物比对及其质量监控由小鼠HORMAD1控制

Mouse HORMAD1 and HORMAD2, two conserved meiotic chromosomal proteins, are depleted from synapsed chromosome axes with the help of TRIP13 AAA-ATPase

小鼠 HORMAD1 和 HORMAD2 是两种保守的减数分裂染色体蛋白，在 TRIP13 AAA-ATPase 的帮助下从突触染色体轴上消失