日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Variation in histone configurations correlates with gene expression across nine inbred strains of mice

组蛋白结构的变化与九种近交系小鼠的基因表达相关

Anna L Tyler #, Catrina Spruce #, Romy Kursawe, Annat Haber, Robyn L Ball, Wendy A Pitman, Alexander D Fine, Narayanan Raghupathy, Michael Walker, Vivek M Philip, Christopher L Baker, J Matthew Mahoney, Gary A Churchill, Jennifer J Trowbridge, Michael L Stitzel, Kenneth Paigen, Petko M Petkov, Grego

Sexual dimorphism in the meiotic requirement for PRDM9: A mammalian evolutionary safeguard

PRDM9 减数分裂要求中的性别二态性:哺乳动物的进化保障

Natalie R Powers, Beth L Dumont, Chihiro Emori, Raman Akinyanju Lawal, Catherine Brunton, Kenneth Paigen, Mary Ann Handel, Ewelina Bolcun-Filas, Petko M Petkov, Tanmoy Bhattacharyya

HELLS and PRDM9 form a pioneer complex to open chromatin at meiotic recombination hot spots

HELLS 和 PRDM9 形成先锋复合物,在减数分裂重组热点处打开染色质

Catrina Spruce, Sibongakonke Dlamini, Guruprasad Ananda, Naomi Bronkema, Hui Tian, Kenneth Paigen, Gregory W Carter, Christopher L Baker

Histone methyltransferase PRDM9 is not essential for meiosis in male mice

组蛋白甲基转移酶 PRDM9 对雄性小鼠减数分裂不是必需的

Ondrej Mihola, Florencia Pratto, Kevin Brick, Eliska Linhartova, Tatyana Kobets, Petr Flachs, Christopher L Baker, Radislav Sedlacek, Kenneth Paigen, Petko M Petkov, R Daniel Camerini-Otero, Zdenek Trachtulec

PRDM9 interactions with other proteins provide a link between recombination hotspots and the chromosomal axis in meiosis

PRDM9 与其他蛋白质的相互作用提供了重组热点和减数分裂染色体轴之间的联系

Emil D Parvanov, Hui Tian, Timothy Billings, Ruth L Saxl, Catrina Spruce, Rakesh Aithal, Lumir Krejci, Kenneth Paigen, Petko M Petkov

The Meiotic Recombination Activator PRDM9 Trimethylates Both H3K36 and H3K4 at Recombination Hotspots In Vivo

减数分裂重组激活剂 PRDM9 在体内对重组热点处的 H3K36 和 H3K4 进行三甲基化

Natalie R Powers, Emil D Parvanov, Christopher L Baker, Michael Walker, Petko M Petkov, Kenneth Paigen

Affinity-seq detects genome-wide PRDM9 binding sites and reveals the impact of prior chromatin modifications on mammalian recombination hotspot usage

Affinity-seq 检测全基因组 PRDM9 结合位点,并揭示先前染色质修饰对哺乳动物重组热点使用的影响

Michael Walker #, Timothy Billings #, Christopher L Baker, Natalie Powers, Hui Tian, Ruth L Saxl, Kwangbom Choi, Matthew A Hibbs, Gregory W Carter, Mary Ann Handel, Kenneth Paigen, Petko M Petkov

PRDM9 drives evolutionary erosion of hotspots in Mus musculus through haplotype-specific initiation of meiotic recombination

PRDM9 通过单倍型特异性启动减数分裂重组,推动小鼠热点的进化侵蚀

Christopher L Baker, Shimpei Kajita, Michael Walker, Ruth L Saxl, Narayanan Raghupathy, Kwangbom Choi, Petko M Petkov, Kenneth Paigen

Multimer Formation Explains Allelic Suppression of PRDM9 Recombination Hotspots

多聚体的形成解释了 PRDM9 重组热点的等位基因抑制

Christopher L Baker, Pavlina Petkova, Michael Walker, Petr Flachs, Ondrej Mihola, Zdenek Trachtulec, Petko M Petkov, Kenneth Paigen

PRDM9 binding organizes hotspot nucleosomes and limits Holliday junction migration

PRDM9 结合组织热点核小体并限制霍利迪连接体迁移

Christopher L Baker, Michael Walker, Shimpei Kajita, Petko M Petkov, Kenneth Paigen