A Chronic Increase in Blood-Brain Barrier Permeability Facilitates Intraneuronal Deposition of Exogenous Bloodborne Amyloid-Beta1-42 Peptide in the Brain and Leads to Alzheimer's Disease-Relevant Cognitive Changes in a Mouse Model

血脑屏障通透性的慢性增加促进外源性血源性淀粉样蛋白-β1-42 肽在脑内的神经元内沉积，并导致小鼠模型中与阿尔茨海默病相关的认知变化