Kesh Kousik相关文献与解析，生知库 | 链接生命科学的每一步

日期影响因子

Targeting Triple-Negative Breast Cancer with Momordicine-I for Therapeutic Gain in Preclinical Models.

利用苦参碱-I靶向治疗三阴性乳腺癌，在临床前模型中获得治疗效果

影响因子:4.4

doi:10.3390/cancers17142342

Kesh Kousik, Tran Ellen T, Patel Ruchi A, Ma Cynthia X, Ray Ratna B

发表日期：2025

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Obesity enriches for tumor protective microbial metabolites and treatment refractory cells to confer therapy resistance in PDAC

肥胖会增加肿瘤保护性微生物代谢产物和难治性细胞的含量，从而导致胰腺导管腺癌（PDAC）产生治疗耐药性。

期刊:Gut Microbes

影响因子:11

doi:10.1080/19490976.2022.2096328

Kesh, Kousik; Mendez, Roberto; Mateo-Victoriano, Beatriz; Garrido, Vanessa T; Durden, Brittany; Gupta, Vineet K; Oliveras Reyes, Alfredo; Merchant, Nipun; Datta, Jashodeep; Banerjee, Santanu; Banerjee, Sulagna

细胞生物学

发表日期：2022

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Opioid Use in Murine Model Results in Severe Gastric Pathology that May Be Attenuated by Proton Pump Inhibition

在小鼠模型中，阿片类药物的使用会导致严重的胃部病变，而质子泵抑制剂可能减轻这种病变。

期刊:American Journal of Pathology

影响因子:3.6

doi:10.1016/j.ajpath.2022.04.005

Ghosh, Nillu; Kesh, Kousik; Ramakrishnan, Sundaram; Roy, Sabita

发表日期：2022

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Type 2 diabetes induced microbiome dysbiosis is associated with therapy resistance in pancreatic adenocarcinoma

2型糖尿病引起的肠道菌群失调与胰腺腺癌的治疗耐药性相关

期刊:Microbial Cell Factories

影响因子:4.9

doi:10.1186/s12934-020-01330-3

Kesh, Kousik; Mendez, Roberto; Abdelrahman, Leila; Banerjee, Santanu; Banerjee, Sulagna

发表日期：2020

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Therapy Resistance, Cancer Stem Cells and ECM in Cancer: The Matrix Reloaded

癌症中的治疗耐药性、癌症干细胞和细胞外基质：基质重装上阵

影响因子:4.4

doi:10.3390/cancers12103067

Kesh, Kousik; Gupta, Vineet K; Durden, Brittany; Garrido, Vanessa; Mateo-Victoriano, Beatriz; Lavania, Shweta P; Banerjee, Sulagna

细胞生物学

发育与干细胞

发表日期：2020

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Microbial dysbiosis and polyamine metabolism as predictive markers for early detection of pancreatic cancer

微生物菌群失调和多胺代谢作为胰腺癌早期检测的预测标志物

期刊:Carcinogenesis

影响因子:2.9

doi:10.1093/carcin/bgz116

Mendez, Roberto; Kesh, Kousik; Arora, Nivedita; Di Martino, Leá; McAllister, Florencia; Merchant, Nipun; Banerjee, Sulagna; Banerjee, Santanu

发表日期：2020

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Long non-coding RNA GAS5 acts as proliferation "brakes" in CD133+ cells responsible for tumor recurrence

长链非编码RNA GAS5在CD133+细胞中起到增殖“刹车”的作用，而CD133+细胞是肿瘤复发的罪魁祸首。

期刊:Oncogenesis

影响因子:6.4

doi:10.1038/s41389-019-0177-4

Sharma, Nikita S; Gnamlin, Prisca; Durden, Brittany; Gupta, Vineet K; Kesh, Kousik; Garrido, Vanessa T; Dudeja, Vikas; Saluja, Ashok; Banerjee, Sulagna

细胞生物学

发表日期：2019

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Inactivation of Cancer-Associated-Fibroblasts Disrupts Oncogenic Signaling in Pancreatic Cancer Cells and Promotes Its Regression

抑制癌相关成纤维细胞可破坏胰腺癌细胞中的致癌信号传导并促进其消退

期刊:Cancer Research

影响因子:16.6

doi:10.1158/0008-5472.CAN-17-2320

Dauer, Patricia; Zhao, Xianda; Gupta, Vineet K; Sharma, Nikita; Kesh, Kousik; Gnamlin, Prisca; Dudeja, Vikas; Vickers, Selwyn M; Banerjee, Sulagna; Saluja, Ashok

成纤维细胞

细胞生物学

发表日期：2018

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Association of MMP7 -181AâG Promoter Polymorphism with Gastric Cancer Risk: INFLUENCE OF NICOTINE IN DIFFERENTIAL ALLELE-SPECIFIC TRANSCRIPTION VIA INCREASED PHOSPHORYLATION OF cAMP-RESPONSE ELEMENT-BINDING PROTEIN (CREB).

MMP7 -181Aâ†'G 启动子多态性与胃癌风险的关联：尼古丁通过增加 cAMP 反应元件结合蛋白 (CREB) 的磷酸化作用影响差异等位基因特异性转录

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M114.630129

Kesh Kousik, Subramanian Lakshmi, Ghosh Nillu, Gupta Vinayak, Gupta Arnab, Bhattacharya Samir, Mahapatra Nitish R, Swarnakar Snehasikta

发表日期：2015

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Matrix metalloproteinase-1 (MMP-1) Promoter polymorphisms are well linked with lower stomach tumor formation in eastern Indian population

基质金属蛋白酶-1 (MMP-1) 启动子多态性与印度东部人群的下胃肿瘤形成密切相关

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0088040

Dey, Sanjib; Ghosh, Nillu; Saha, Debjit; Kesh, Kousik; Gupta, Arnab; Swarnakar, Snehasikta

多发性骨髓瘤

发表日期：2014

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