日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Akap5 links synaptic dysfunction to neuroinflammatory signaling in a mouse model of infantile neuronal ceroid lipofuscinosis

Akap5 在婴儿神经元蜡样脂褐素沉积症小鼠模型中将突触功能障碍与神经炎症信号联系起来

Kevin P Koster, Zach Fyke, Thu T A Nguyen, Amanda Niqula, Lorena Y Noriega-González, Kevin M Woolfrey, Mark L Dell'Acqua, Stephanie M Cologna, Akira Yoshii

Amyloid-β-induced dendritic spine elimination requires Ca2+-permeable AMPA receptors, AKAP-Calcineurin-NFAT signaling, and the NFAT target gene Mdm2

淀粉样蛋白-β诱导的树突棘消除需要 Ca2+ 通透性 AMPA 受体、AKAP-钙调磷酸酶-NFAT 信号传导和 NFAT 靶基因 Mdm2

Tyler P Martinez, Matthew E Larsen, Emily Sullivan, Kevin M Woolfrey, Mark L Dell'Acqua

Loss of EPAC2 alters dendritic spine morphology and inhibitory synapse density

EPAC2 的缺失会改变树突棘的形态和抑制性突触密度

Kelly A Jones, Michiko Sumiya, Kevin M Woolfrey, Deepak P Srivastava, Peter Penzes

AKAP150 Palmitoylation Regulates Synaptic Incorporation of Ca2+-Permeable AMPA Receptors to Control LTP

AKAP150棕榈酰化调控钙离子通透性AMPA受体在突触中的整合,从而控制长时程增强作用(LTP)。

Alicia M Purkey ,Kevin M Woolfrey ,Kevin C Crosby ,Dominik G Stich ,Wallace S Chick ,Jason Aoto ,Mark L Dell'Acqua

Palmitoylation of caveolin-1 is regulated by the same DHHC acyltransferases that modify steroid hormone receptors

Caveolin-1 的棕榈酰化由与修饰类固醇激素受体相同的 DHHC 酰基转移酶调控

Katherine R Tonn Eisinger, Kevin M Woolfrey, Samuel P Swanson, Stephen A Schnell, John Meitzen, Mark Dell'Acqua, Paul G Mermelstein

Social, communication, and cortical structural impairments in Epac2-deficient mice

Epac2 缺陷小鼠的社交、沟通和皮质结构障碍

Deepak P Srivastava, Kelly A Jones, Kevin M Woolfrey, Jeffrey Burgdorf, Theron A Russell, Abigail Kalmbach, Hyerin Lee, Connie Yang, Mazdak M Bradberry, David Wokosin, Joseph R Moskal, Manuel F Casanova, Jack Waters, Peter Penzes

Analysis of dendritic spine morphology in cultured CNS neurons

培养的中枢神经系统神经元树突棘形态分析

Deepak P Srivastava ,Kevin M Woolfrey, Peter Penzes