Kevin P Foley相关文献与解析，生知库 | 链接生命科学的每一步

Matthias P Mayer, Laura Blair, Gregory L Blatch, Thiago J Borges, Ahmed Chadli, Gabriela Chiosis, Aurélie de Thonel, Albena Dinkova-Kostova, Heath Ecroyd, Adrienne L Edkins, Takanori Eguchi, Monika Fleshner, Kevin P Foley, Sotirios Fragkostefanakis, Jason Gestwicki, Pierre Goloubinoff, Jennifer A He

信号转导

发表日期：2024

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Human gut microbiota after bariatric surgery alters intestinal morphology and glucose absorption in mice independently of obesity

减肥手术后人类肠道微生物群独立于肥胖而改变小鼠的肠道形态和葡萄糖吸收

期刊:Gut

影响因子:23

doi:10.1136/gutjnl-2022-328185

Fernando F Anhê, Soumaya Zlitni, Song-Yang Zhang, Béatrice So-Yun Choi, Cassandra Y Chen, Kevin P Foley, Nicole G Barra, Michael G Surette, Laurent Biertho, Denis Richard, André Tchernof, Tony K T Lam, Andre Marette, Jonathan Schertzer

微生物学

发表日期：2023

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Lower brown adipose tissue activity is associated with non-alcoholic fatty liver disease but not changes in the gut microbiota

棕色脂肪组织活性降低与非酒精性脂肪肝有关，但与肠道微生物群变化无关

期刊:Cell Reports Medicine

影响因子:11.7

doi:10.1016/j.xcrm.2021.100397

Basma A Ahmed, Frank J Ong, Nicole G Barra, Denis P Blondin, Elizabeth Gunn, Stephan M Oreskovich, Jake C Szamosi, Saad A Syed, Emily K Hutchings, Norman B Konyer, Nina P Singh, Julian M Yabut, Eric M Desjardins, Fernando F Anhê, Kevin P Foley, Alison C Holloway, Michael D Noseworthy, Francois Haman

Inflammation promotes adipocyte lipolysis via IRE1 kinase

炎症通过IRE1激酶促进脂肪细胞脂肪分解。

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1016/j.jbc.2021.100440

Kevin P Foley ，Yong Chen ，Nicole G Barra ，Mark Heal ，Kieran Kwok ，Akhilesh K Tamrakar ，Wendy Chi ，Brittany M Duggan ，Brandyn D Henriksbo ，Yong Liu ，Jonathan D Schertzer

Immunology/Inflammation

Gut microbiota impairs insulin clearance in obese mice

肠道菌群损害肥胖小鼠的胰岛素清除率

期刊:Molecular Metabolism

影响因子:7

doi:10.1016/j.molmet.2020.101067

Kevin P Foley, Soumaya Zlitni, Brittany M Duggan, Nicole G Barra, Fernando F Anhê, Joseph F Cavallari, Brandyn D Henriksbo, Cassandra Y Chen, Michael Huang, Trevor C Lau, Roxanne Plante, Michael Schwab, André Marette, Jonathan D Schertzer

微生物学

发表日期：2020

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MALT1 Protease Plays a Dual Role in the Allergic Response by Acting in Both Mast Cells and Endothelial Cells

MALT1 蛋白酶在肥大细胞和内皮细胞中起作用，在过敏反应中发挥双重作用

期刊:Journal of Immunology

影响因子:3.6

doi:10.4049/jimmunol.1900281

Danielle N Alfano, Linda R Klei, Hanna B Klei, Matthew Trotta, Peter J Gough, Kevin P Foley, John Bertin, Tina L Sumpter, Peter C Lucas, Linda M McAllister-Lucas

Adipose Tissue Inflammation Is Directly Linked to Obesity-Induced Insulin Resistance, while Gut Dysbiosis and Mitochondrial Dysfunction Are Not Required

脂肪组织炎症与肥胖引起的胰岛素抵抗直接相关，而肠道菌群失调和线粒体功能障碍并非必要因素

期刊:Function

影响因子:

doi:10.1093/function/zqaa013

Heather L Petrick, Kevin P Foley, Soumaya Zlitni, Henver S Brunetta, Sabina Paglialunga, Paula M Miotto, Valerie Politis-Barber, Conor O'Dwyer, Diana J Philbrick, Morgan D Fullerton, Jonathan D Schertzer, Graham P Holloway

微生物学

发表日期：2020

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TNF, but not hyperinsulinemia or hyperglycemia, is a key driver of obesity-induced monocytosis revealing that inflammatory monocytes correlate with insulin in obese male mice

TNF（而非高胰岛素血症或高血糖症）是肥胖诱导单核细胞增多症的关键驱动因素，这表明炎症性单核细胞与肥胖雄性小鼠的胰岛素水平相关。

期刊:Physiological Reports

影响因子:2.2

doi:10.14814/phy2.13937

Jessica A Breznik ，Avee Naidoo ，Kevin P Foley ，Christian Schulz ，Trevor C Lau ，Dessi Loukov ，Deborah M Sloboda ，Dawn M E Bowdish ，Jonathan D Schertzer

Targeting macrophage scavenger receptor 1 promotes insulin resistance in obese male mice

靶向巨噬细胞清道夫受体 1 可促进肥胖雄性小鼠产生胰岛素抵抗

期刊:Physiological Reports

影响因子:2.2

doi:10.14814/phy2.13930

Joseph F Cavallari, Fernando F Anhê, Kevin P Foley, Emmanuel Denou, Rebecca W Chan, Dawn M E Bowdish, Jonathan D Schertzer

Age-Associated Microbial Dysbiosis Promotes Intestinal Permeability, Systemic Inflammation, and Macrophage Dysfunction

与年龄相关的微生物菌群失调会促进肠道通透性增加、全身炎症和巨噬细胞功能障碍

期刊:Cell Host & Microbe

影响因子:20.6

doi:10.1016/j.chom.2017.03.002

Netusha Thevaranjan ，Alicja Puchta ，Christian Schulz ，Avee Naidoo ，J C Szamosi ，Chris P Verschoor ，Dessi Loukov ，Louis P Schenck ，Jennifer Jury ，Kevin P Foley ，Jonathan D Schertzer ，Maggie J Larché ，Donald J Davidson ，Elena F Verdú ，Michael G Surette ，Dawn M E Bowdish

Immunology/Inflammation

巨噬细胞

发表日期：2017

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Stress biology: Complexity and multifariousness in health and disease

压力生物学：健康和疾病的复杂性和多样性

Human gut microbiota after bariatric surgery alters intestinal morphology and glucose absorption in mice independently of obesity

减肥手术后人类肠道微生物群独立于肥胖而改变小鼠的肠道形态和葡萄糖吸收

Lower brown adipose tissue activity is associated with non-alcoholic fatty liver disease but not changes in the gut microbiota

棕色脂肪组织活性降低与非酒精性脂肪肝有关，但与肠道微生物群变化无关

Inflammation promotes adipocyte lipolysis via IRE1 kinase

炎症通过IRE1激酶促进脂肪细胞脂肪分解。

Gut microbiota impairs insulin clearance in obese mice

肠道菌群损害肥胖小鼠的胰岛素清除率

MALT1 Protease Plays a Dual Role in the Allergic Response by Acting in Both Mast Cells and Endothelial Cells

MALT1 蛋白酶在肥大细胞和内皮细胞中起作用，在过敏反应中发挥双重作用

Adipose Tissue Inflammation Is Directly Linked to Obesity-Induced Insulin Resistance, while Gut Dysbiosis and Mitochondrial Dysfunction Are Not Required

脂肪组织炎症与肥胖引起的胰岛素抵抗直接相关，而肠道菌群失调和线粒体功能障碍并非必要因素

TNF, but not hyperinsulinemia or hyperglycemia, is a key driver of obesity-induced monocytosis revealing that inflammatory monocytes correlate with insulin in obese male mice

TNF（而非高胰岛素血症或高血糖症）是肥胖诱导单核细胞增多症的关键驱动因素，这表明炎症性单核细胞与肥胖雄性小鼠的胰岛素水平相关。

Targeting macrophage scavenger receptor 1 promotes insulin resistance in obese male mice

靶向巨噬细胞清道夫受体 1 可促进肥胖雄性小鼠产生胰岛素抵抗

Age-Associated Microbial Dysbiosis Promotes Intestinal Permeability, Systemic Inflammation, and Macrophage Dysfunction

与年龄相关的微生物菌群失调会促进肠道通透性增加、全身炎症和巨噬细胞功能障碍