Kilpatrick Bethan S相关文献与解析，生知库

Penny, Christopher J; Vassileva, Kristin; Jha, Archana; Yuan, Yu; Chee, Xavier; Yates, Elizabeth; Mazzon, Michela; Kilpatrick, Bethan S; Muallem, Shmuel; Marsh, Mark; Rahman, Taufiq; Patel, Sandip

微生物学

发表日期：2019

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Endoplasmic reticulum and lysosomal Ca²⁺ stores are remodelled in GBA1-linked Parkinson disease patient fibroblasts

在GBA1相关帕金森病患者的成纤维细胞中，内质网和溶酶体Ca²⁺储存发生重塑。

期刊:Cell Calcium

影响因子:4

doi:10.1016/j.ceca.2015.11.002

Kilpatrick, Bethan S; Magalhaes, Joana; Beavan, Michelle S; McNeill, Alisdair; Gegg, Matthew E; Cleeter, Michael W J; Bloor-Young, Duncan; Churchill, Grant C; Duchen, Michael R; Schapira, Anthony H; Patel, Sandip

Endo-lysosomal TRP mucolipin-1 channels trigger global ER Ca2+ release and Ca2+ influx

内溶酶体TRP粘脂蛋白-1通道触发内质网整体Ca2+释放和Ca2+内流

期刊:Journal of Cell Science

影响因子:3.6

doi:10.1242/jcs.190322

Kilpatrick, Bethan S; Yates, Elizabeth; Grimm, Christian; Schapira, Anthony H; Patel, Sandip

发表日期：2016

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Connecting Ca(2+) and lysosomes to Parkinson disease

将Ca(2+)和溶酶体与帕金森病联系起来

期刊:

影响因子:

doi:10.1166/msr.2016.1059

Dysregulation of lysosomal morphology by pathogenic LRRK2 is corrected by TPC2 inhibition.

致病性 LRRK2 引起的溶酶体形态失调可通过 TPC2 抑制得到纠正

期刊:Journal of Cell Science

影响因子:3.6

doi:10.1242/jcs.164152

Hockey Leanne N, Kilpatrick Bethan S, Eden Emily R, Lin-Moshier Yaping, Brailoiu G Cristina, Brailoiu Eugen, Futter Clare E, Schapira Anthony H, Marchant Jonathan S, Patel Sandip

其它

发表日期：2015

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A "mix-and-match" approach to designing Ca(2+) microdomains at membrane-contact sites

在膜接触位点设计Ca(2+)微区的“混合搭配”方法

期刊:Communicative and Integrative Biology

影响因子:

doi:10.4161/cib.29586

Penny, Christopher J; Kilpatrick, Bethan S; Min Han, Jung; Sneyd, James; Patel, Sandip

发表日期：2014

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Direct mobilisation of lysosomal Ca2+ triggers complex Ca2+ signals

溶酶体Ca2+的直接动员会触发复杂的Ca2+信号。

期刊:Journal of Cell Science

影响因子:3.6

doi:10.1242/jcs.118836

Kilpatrick, Bethan S; Eden, Emily R; Schapira, Anthony H; Futter, Clare E; Patel, Sandip

信号转导

发表日期：2013

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Mining of Ebola virus entry inhibitors identifies approved drugs as two-pore channel pore blockers

对埃博拉病毒入侵抑制剂的研究发现，已批准的药物是双孔通道阻滞剂。

Endoplasmic reticulum and lysosomal Ca²⁺ stores are remodelled in GBA1-linked Parkinson disease patient fibroblasts

在GBA1相关帕金森病患者的成纤维细胞中，内质网和溶酶体Ca²⁺储存发生重塑。

Endo-lysosomal TRP mucolipin-1 channels trigger global ER Ca2+ release and Ca2+ influx

内溶酶体TRP粘脂蛋白-1通道触发内质网整体Ca2+释放和Ca2+内流

Connecting Ca(2+) and lysosomes to Parkinson disease

将Ca(2+)和溶酶体与帕金森病联系起来

Dysregulation of lysosomal morphology by pathogenic LRRK2 is corrected by TPC2 inhibition.

致病性 LRRK2 引起的溶酶体形态失调可通过 TPC2 抑制得到纠正

A "mix-and-match" approach to designing Ca(2+) microdomains at membrane-contact sites

在膜接触位点设计Ca(2+)微区的“混合搭配”方法

Direct mobilisation of lysosomal Ca2+ triggers complex Ca2+ signals

溶酶体Ca2+的直接动员会触发复杂的Ca2+信号。