日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Rotenone accelerates endogenous α-synuclein spreading and enhances neurodegeneration in an intra-striatal α-synuclein preformed fibril injected mouse model of Parkinson's disease.

鱼藤酮可加速内源性α-突触核蛋白的扩散,并增强纹状体内注射α-突触核蛋白预成纤维的帕金森病小鼠模型的神经退行性变。

Khan Engila, Radwan Nada, Ardah Mustafa T, Kitada Tohru, Haque M Emdadul

Ellagic Acid Prevents α-Synuclein Spread and Mitigates Toxicity by Enhancing Autophagic Flux in an Animal Model of Parkinson's Disease.

鞣花酸通过增强帕金森病动物模型中的自噬通量来阻止α-突触核蛋白扩散并减轻毒性

Radwan Nada, Khan Engila, Ardah Mustafa T, Kitada Tohru, Haque M Emdadul

History of Parkinson's Disease-Associated Gene, Parkin: Research over a Quarter Century in Quest of Finding the Physiological Substrate

帕金森病相关基因帕金蛋白的历史:二十五年来寻找其生理基础的研究

Kitada, Tohru; Ardah, Mustafa T; Haque, M Emdadul

Ellagic Acid Prevents Dopamine Neuron Degeneration from Oxidative Stress and Neuroinflammation in MPTP Model of Parkinson's Disease.

鞣花酸可预防 MPTP 帕金森病模型中氧化应激和神经炎症引起的多巴胺神经元退化

Ardah Mustafa T, Bharathan Greeshma, Kitada Tohru, Haque M Emdadul

Loss of DJ-1 does not affect mitochondrial respiration but increases ROS production and mitochondrial permeability transition pore opening

DJ-1的缺失不影响线粒体呼吸作用,但会增加活性氧(ROS)的产生和线粒体通透性转换孔的开放。

Giaime, Emilie; Yamaguchi, Hiroo; Gautier, Clement A; Kitada, Tohru; Shen, Jie

Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress

PINK1 的缺失会导致线粒体功能缺陷和对氧化应激的敏感性增加。

Gautier, Clement A; Kitada, Tohru; Shen, Jie

Impaired dopamine release and synaptic plasticity in the striatum of PINK1-deficient mice

PINK1缺陷小鼠纹状体多巴胺释放和突触可塑性受损

Kitada, Tohru; Pisani, Antonio; Porter, Douglas R; Yamaguchi, Hiroo; Tscherter, Anne; Martella, Giuseppina; Bonsi, Paola; Zhang, Chen; Pothos, Emmanuel N; Shen, Jie