日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Aryl hydrocarbon receptor antagonism mitigates cytokine-mediated inflammatory signalling in primary human fibroblast-like synoviocytes

芳烃受体拮抗作用可减轻原代人成纤维细胞样滑膜细胞中细胞因子介导的炎症信号传导

Lahoti, Tejas S; John, Kaarthik; Hughes, Jarod M; Kusnadi, Ann; Murray, Iain A; Krishnegowda, Gowdahalli; Amin, Shantu; Perdew, Gary H

Ah receptor antagonism represses head and neck tumor cell aggressive phenotype

Ah受体拮抗剂抑制头颈部肿瘤细胞的侵袭性表型

DiNatale, Brett C; Smith, Kayla; John, Kaarthik; Krishnegowda, Gowdahalli; Amin, Shantu G; Perdew, Gary H

Selective aryl hydrocarbon receptor modulator-mediated repression of CD55 expression induced by cytokine exposure

选择性芳烃受体调节剂介导的细胞因子暴露诱导的CD55表达抑制

Narayanan, Gitanjali A; Murray, Iain A; Krishnegowda, Gowdahalli; Amin, Shantu; Perdew, Gary H

Facile syntheses of O(2)-[4-(3-pyridyl-4-oxobut-1-yl]thymidine, the major adduct formed by tobacco specific nitrosamine 4-methylnitrosamino-1-(3-pyridyl)-1-butanone (NNK) in vivo, and its site-specifically adducted oligodeoxynucleotides

简便合成 O(2)-[4-(3-吡啶基-4-氧代丁-1-基]胸苷,它是烟草特异性亚硝胺 4-甲基亚硝胺-1-(3-吡啶基)-1-丁酮 (NNK) 在体内形成的主要加合物,以及其位点特异性加合的寡脱氧核苷酸

Krishnegowda, Gowdahalli; Sharma, Arun K; Krzeminski, Jacek; Gowda, A S Prakasha; Lin, Jyh-Ming; Desai, Dhimant; Spratt, Thomas E; Amin, Shantu

Proinflammatory responses by glycosylphosphatidylinositols (GPIs) of Plasmodium falciparum are mainly mediated through the recognition of TLR2/TLR1.

恶性疟原虫糖基磷脂酰肌醇(GPI)的促炎反应主要通过识别TLR2/TLR1介导

Zhu Jianzhong, Krishnegowda Gowdahalli, Li Guangfu, Gowda D Channe

Development of a selective modulator of aryl hydrocarbon (Ah) receptor activity that exhibits anti-inflammatory properties

开发一种具有抗炎特性的芳烃(Ah)受体活性选择性调节剂

Murray, Iain A; Krishnegowda, Gowdahalli; DiNatale, Brett C; Flaveny, Colin; Chiaro, Chris; Lin, Jyh-Ming; Sharma, Arun K; Amin, Shantu; Perdew, Gary H

Plasmodium falciparum GPI mannosyltransferase-III has novel signature sequence and is functional

恶性疟原虫GPI甘露糖基转移酶-III具有新的特征序列,且具有功能活性。

Basagoudanavar, Suresh H; Feng, Xiaorong; Krishnegowda, Gowdahalli; Muthusamy, Arivalagan; Gowda, D Channe

Naturally elicited antibodies to glycosylphosphatidylinositols (GPIs) of Plasmodium falciparum require intact GPI structures for binding and are directed primarily against the conserved glycan moiety

天然产生的针对恶性疟原虫糖基磷脂酰肌醇(GPI)的抗体需要完整的GPI结构才能结合,并且主要针对保守的聚糖部分。

Naik, Ramachandra S; Krishnegowda, Gowdahalli; Ockenhouse, Christian F; Gowda, D Channe

Induction of proinflammatory responses in macrophages by the glycosylphosphatidylinositols of Plasmodium falciparum: the requirement of extracellular signal-regulated kinase, p38, c-Jun N-terminal kinase and NF-kappaB pathways for the expression of proinflammatory cytokines and nitric oxide

恶性疟原虫糖基磷脂酰肌醇诱导巨噬细胞产生促炎反应:促炎细胞因子和一氧化氮表达需要细胞外信号调节激酶、p38、c-Jun N端激酶和NF-κB通路

Zhu, Jianzhong; Krishnegowda, Gowdahalli; Gowda, D Channe

Induction of nitric oxide synthase in Anopheles stephensi by Plasmodium falciparum: mechanism of signaling and the role of parasite glycosylphosphatidylinositols

恶性疟原虫诱导斯氏按蚊体内一氧化氮合酶的表达:信号传导机制及寄生虫糖基磷脂酰肌醇的作用

Lim, Junghwa; Gowda, D Channe; Krishnegowda, Gowdahalli; Luckhart, Shirley