Epigenetic regulation by HDAC11-driven STAT3 activation promotes pro-inflammatory cytokine production and endothelial dysfunction upon bacterial infection in diabetes
在糖尿病中，细菌感染后，HDAC11驱动的STAT3激活介导的表观遗传调控促进促炎细胞因子的产生和内皮功能障碍。
期刊:Cell Communication and Signaling
影响因子:8.9
doi:10.1186/s12964-026-02696-4
Setyaningsih, Wiwit Ananda Wahyu; Naderi-Meshkin, Hojjat; Yacoub, Andrew; Cornelius, Victoria A; Silva, Johnatas Dutra; Fitriawan, Akbar Satria; Guduric-Fuchs, Jasenka; Tashkandi, Asim Jamalabdalnaser A; Kuburas, Refik; Zampetaki, Anna; Lois, Noemi; Stitt, Alan W; Valvano, Miguel A; Grieve, David J; Atlasi, Yaser; Krasnodembskaya, Anna; Margariti, Andriana
HDAC1
表观遗传
炎症/感染
信号转导
JAK/STAT
代谢
微生物学
STAT3
糖尿病
细胞生物学
免疫/内分泌
