Kucharzik T相关文献与解析，生知库 | 链接生命科学的每一步

Inflammatory Bowel Disease Management During the COVID-19 Outbreak: The Ten Do's and Don'ts from the ECCO-COVID Taskforce

COVID-19 疫情期间炎症性肠病管理：ECCO-COVID 工作组的十项注意事项

Magro, F; Rahier, J-F; Abreu, C; MacMahon, E; Hart, A; van der Woude, C J; Gordon, H; Adamina, M; Viget, N; Vavricka, S; Kucharzik, T; Leone, S; Siegmund, B; Danese, S; Peyrin-Biroulet, L

炎症/感染

炎症

发表日期：2020

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Investigating the role of proinflammatory CD16+ monocytes in the pathogenesis of inflammatory bowel disease

研究促炎性CD16+单核细胞在炎症性肠病发病机制中的作用

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1111/j.1365-2249.2010.04177.x

Koch, S; Kucharzik, T; Heidemann, J; Nusrat, A; Luegering, A

Modulating the intestinal immune system: the role of lymphotoxin and GALT organs

调节肠道免疫系统：淋巴毒素和肠道相关淋巴组织（GALT）的作用

期刊:Gut

影响因子:25.8

doi:10.1136/gut.2003.023671

Spahn, T W; Kucharzik, T

Characterization of M cell formation and associated mononuclear cells during indomethacin-induced intestinal inflammation

吲哚美辛诱导的肠道炎症过程中M细胞形成及相关单核细胞的特征分析

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1111/j.1365-2249.2004.02438.x

Lügering, A; Floer, M; Lügering, N; Cichon, C; Schmidt, M A; Domschke, W; Kucharzik, T

Neutrophil derived human S100A12 (EN-RAGE) is strongly expressed during chronic active inflammatory bowel disease

中性粒细胞来源的人类S100A12（EN-RAGE）在慢性活动性炎症性肠病中强烈表达。

期刊:Gut

影响因子:25.8

doi:10.1136/gut.52.6.847

Foell, D; Kucharzik, T; Kraft, M; Vogl, T; Sorg, C; Domschke, W; Roth, J

Patients with inflammatory bowel disease (IBD) reveal increased induction capacity of intracellular interferon-gamma (IFN-gamma) in peripheral CD8+ lymphocytes co-cultured with intestinal epithelial cells

炎症性肠病（IBD）患者的外周血CD8+淋巴细胞与肠上皮细胞共培养时，其细胞内干扰素-γ（IFN-γ）的诱导能力增强。

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1046/j.1365-2249.2001.01443.x

Bisping, G; Lügering, N; Lütke-Brintrup, S; Pauels, H G; Schürmann, G; Domschke, W; Kucharzik, T

Colonic epithelial cells induce endothelial cell expression of ICAM-1 and VCAM-1 by a NF-kappaB-dependent mechanism

结肠上皮细胞通过NF-κB依赖性机制诱导内皮细胞表达ICAM-1和VCAM-1。

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1046/j.1365-2249.2001.01541.x

Maaser, C; Schoeppner, S; Kucharzik, T; Kraft, M; Schoenherr, E; Domschke, W; Luegering, N

IFN-gamma synergizes with TNF-alpha but not with viable H. pylori in up-regulating CXC chemokine secretion in gastric epithelial cells.

IFN-γ 与 TNF-α 协同作用，但与活的幽门螺杆菌不协同作用，可上调胃上皮细胞中 CXC 趋化因子的分泌

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1046/j.1365-2249.2001.01634.x

Kraft M, Riedel S, Maaser C, Kucharzik T, Steinbuechel A, Domschke W, Luegering N

Human intestinal M cells exhibit enterocyte-like intermediate filaments

人类肠道M细胞表现出类似肠细胞的中间丝

期刊:Gut

影响因子:25.8

doi:10.1136/gut.42.1.54

Kucharzik, T; Lügering, N; Schmid, K W; Schmidt, M A; Stoll, R; Domschke, W

Monitoring of patients with active inflammatory bowel disease

对活动性炎症性肠病患者进行监测

Inflammatory Bowel Disease Management During the COVID-19 Outbreak: The Ten Do's and Don'ts from the ECCO-COVID Taskforce

COVID-19 疫情期间炎症性肠病管理：ECCO-COVID 工作组的十项注意事项

Investigating the role of proinflammatory CD16+ monocytes in the pathogenesis of inflammatory bowel disease

研究促炎性CD16+单核细胞在炎症性肠病发病机制中的作用

Modulating the intestinal immune system: the role of lymphotoxin and GALT organs

调节肠道免疫系统：淋巴毒素和肠道相关淋巴组织（GALT）的作用

Characterization of M cell formation and associated mononuclear cells during indomethacin-induced intestinal inflammation

吲哚美辛诱导的肠道炎症过程中M细胞形成及相关单核细胞的特征分析

Neutrophil derived human S100A12 (EN-RAGE) is strongly expressed during chronic active inflammatory bowel disease

中性粒细胞来源的人类S100A12（EN-RAGE）在慢性活动性炎症性肠病中强烈表达。

Patients with inflammatory bowel disease (IBD) reveal increased induction capacity of intracellular interferon-gamma (IFN-gamma) in peripheral CD8+ lymphocytes co-cultured with intestinal epithelial cells

炎症性肠病（IBD）患者的外周血CD8+淋巴细胞与肠上皮细胞共培养时，其细胞内干扰素-γ（IFN-γ）的诱导能力增强。

Colonic epithelial cells induce endothelial cell expression of ICAM-1 and VCAM-1 by a NF-kappaB-dependent mechanism

结肠上皮细胞通过NF-κB依赖性机制诱导内皮细胞表达ICAM-1和VCAM-1。

IFN-gamma synergizes with TNF-alpha but not with viable H. pylori in up-regulating CXC chemokine secretion in gastric epithelial cells.

IFN-γ 与 TNF-α 协同作用，但与活的幽门螺杆菌不协同作用，可上调胃上皮细胞中 CXC 趋化因子的分泌

Human intestinal M cells exhibit enterocyte-like intermediate filaments

人类肠道M细胞表现出类似肠细胞的中间丝