Kwon Hae Ryong相关文献与解析，生知库 | 链接生命科学的每一步

Overactive PDGFRÎ± and PDGFRÎ² promote distinct yet overlapping phenotypes of skeletal muscle fibrosis and stiffness, with PDGFRÎ² also driving drastic muscle growth.

PDGFRα 和 PDGFRβ 过度活跃会促进骨骼肌纤维化和僵硬的不同但又相互重叠的表型，其中 PDGFRβ 还会驱动肌肉的剧烈生长。

doi:10.64898/2026.01.02.697406

Gimla Mariola, Olszewski Szczepan, Brown Jacob L, Raymond-Pope Christiana, Rigsby Sandra, Peelor Frederick F 3rd, Kwon Hae Ryong, Yao Longbiao, Olson Lorin E, Fuqua Jordan D, Miller Benjamin F

发表日期：2026

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PDGFRÎ± signaling regulates cartilage and fibrous tissue differentiation during synovial joint development.

PDGFRα信号传导调节滑膜关节发育过程中软骨和纤维组织的分化

期刊:Nature Communications

影响因子:15.7

doi:10.1038/s41467-025-59207-1

Woods John P, Rackley Alex, Kwon Hae Ryong, Olson Lorin E

Imatinib on target in stroke recovery

伊马替尼在卒中康复中发挥靶向作用

期刊:Journal of Clinical Investigation

影响因子:13.6

doi:10.1172/JCI190024

Kwon, Hae Ryong; Olson, Lorin E

发表日期：2025

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Mouse scalp development requires Rac1 and SRF for the maintenance of mechanoresponsive mesenchyme

小鼠头皮发育需要 Rac1 和 SRF 来维持机械反应性间充质。

期刊:Development

影响因子:3.6

doi:10.1242/dev.204965

Rathnakar, Bharath H; Rackley, Alex; Kwon, Hae Ryong; Kim, Jang H; Berry, William L; Olson, Lorin E

Mouse scalp development requires Rac1 and SRF for the maintenance of mechanosensing mesenchyme

小鼠头皮发育需要 Rac1 和 SRF 来维持机械感受间充质。

期刊:

影响因子:

doi:10.1101/2025.02.11.637680

Rathnakar, Bharath H; Rackley, Alex; Kwon, Hae Ryong; Berry, William L; Olson, Lorin E

STAT1 modulates tissue wasting or overgrowth downstream from PDGFRβ

STAT1 调控 PDGFRβ 下游的组织萎缩或过度增生

期刊:Genes & Development

影响因子:7.7

doi:10.1101/gad.300384.117

He, Chaoyong; Medley, Shayna C; Kim, Jang; Sun, Chengyi; Kwon, Hae Ryong; Sakashita, Hiromi; Pincu, Yair; Yao, Longbiao; Eppard, Danielle; Dai, Bojie; Berry, William L; Griffin, Timothy M; Olson, Lorin E

The contribution of specific cell subpopulations to submandibular salivary gland branching morphogenesis

特定细胞亚群对颌下腺分支形态发生的贡献

期刊:Current Opinion in Genetics & Development

影响因子:3.6

doi:10.1016/j.gde.2015.01.007

Kwon, Hae Ryong; Larsen, Melinda

细胞生物学

发表日期：2015

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Connective tissue growth in a mouse model of Kosaki overgrowth syndrome is limited by STAT1

在 Kosaki 过度生长综合征小鼠模型中，结缔组织生长受 STAT1 限制。

Overactive PDGFRÎ± and PDGFRÎ² promote distinct yet overlapping phenotypes of skeletal muscle fibrosis and stiffness, with PDGFRÎ² also driving drastic muscle growth.

PDGFRα 和 PDGFRβ 过度活跃会促进骨骼肌纤维化和僵硬的不同但又相互重叠的表型，其中 PDGFRβ 还会驱动肌肉的剧烈生长。

PDGFRÎ± signaling regulates cartilage and fibrous tissue differentiation during synovial joint development.

PDGFRα信号传导调节滑膜关节发育过程中软骨和纤维组织的分化

Imatinib on target in stroke recovery

伊马替尼在卒中康复中发挥靶向作用

Mouse scalp development requires Rac1 and SRF for the maintenance of mechanoresponsive mesenchyme

小鼠头皮发育需要 Rac1 和 SRF 来维持机械反应性间充质。

Mouse scalp development requires Rac1 and SRF for the maintenance of mechanosensing mesenchyme

小鼠头皮发育需要 Rac1 和 SRF 来维持机械感受间充质。

STAT1 modulates tissue wasting or overgrowth downstream from PDGFRβ

STAT1 调控 PDGFRβ 下游的组织萎缩或过度增生

The contribution of specific cell subpopulations to submandibular salivary gland branching morphogenesis

特定细胞亚群对颌下腺分支形态发生的贡献