Enhanced primary ciliogenesis via mitochondrial oxidative stress activates AKT to prevent neurotoxicity in HSPA9/mortalin-depleted SH-SY5Y cells
通过线粒体氧化应激增强的原发性纤毛生成可激活 AKT,从而防止 HSPA9/mortalin 耗竭的 SH-SY5Y 细胞产生神经毒性
期刊:Molecular Brain
影响因子:3.3
doi:10.1186/s13041-023-01029-7
Ji-Eun Bae, Soyoung Jang, Joon Bum Kim, Hyejin Hyung, Na Yeon Park, Yong Hwan Kim, So Hyun Kim, Seong Hyun Kim, Jin Min Ha, Gyeong Seok Oh, Kyuhee Park, Kwiwan Jeong, Jae Seon Jang, Doo Sin Jo, Pansoo Kim, Hyun-Shik Lee, Zae Young Ryoo, Dong-Hyung Cho