日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

MOS is a key regulator of meiotic midbody assembly and prevents abnormal divisions in mouse eggs and their polar bodies

MOS是减数分裂中体组装的关键调控因子,能够防止小鼠卵子及其极体发生异常分裂。

Cairo, Gisela; Haseeb, Muhammad A; Boyce Joy, Zachary; Schindler, Karen; Lacefield, Soni

A cross-center comparison of the relationship between matriline fragmentation, grooming cohesion, and agonistic behavior in captive rhesus macaque (Macaca mulatta) social groups

一项跨中心比较研究,旨在探讨圈养恒河猴(Macaca mulatta)社会群体中母系分裂、梳理行为凝聚力和攻击行为之间的关系。

Beisner, B A; McCowan, B; Bloomsmith, M A; Lacefield, L; Ethun, K F

Aneuploidy of specific chromosomes is beneficial to cells lacking spindle checkpoint protein Bub3

特定染色体的非整倍体对缺乏纺锤体检查点蛋白Bub3的细胞是有益的。

Gadgil, Pallavi; Ballew, Olivia; Sullivan, Timothy J; Lacefield, Soni

Disrupted MOS signaling alters meiotic cell cycle regulation and the egg transcriptome.

MOS信号传导紊乱会改变减数分裂细胞周期调控和卵子转录组

Cairo Gisela, Kholod Olha, Palmer Olivia, Meytin Sophia, Goods Brittany A, Lacefield Soni

The B-type cyclin Clb4 prevents meiosis I sister centromere separation in budding yeast

B型细胞周期蛋白Clb4可阻止出芽酵母减数分裂I期姐妹着丝粒的分离。

Lumbroso, Gal; Cairo, Gisela; Lacefield, Soni; Murray, Andrew W

Disrupted MOS signaling alters meiotic cell cycle regulation and the egg transcriptome

MOS信号传导紊乱会改变减数分裂细胞周期调控和卵子转录组。

Cairo, Gisela; Kholod, Olha; Palmer, Olivia; Goods, Brittany A; Lacefield, Soni

Antidepressants reactivate developmental plasticity through remodeling of extracellular matrix

抗抑郁药通过重塑细胞外基质来重新激活发育可塑性

Nguyen, Phi T; Sun, Emily; Shi, Yuecheng; Tamura, Shogo; Xiao, Yang; Lacefield, Clay; Turi, Gergely F; Hen, René

A minimal model for the role of Rim4 in regulating meiotic exit in budding yeast

Rim4在调控出芽酵母减数分裂退出中的作用的最小模型

Marquez Davila, V Abigail; Gadgil, Pallavi; Zike, Anna; Cairo, Gisela; Wang, Renyu; Setayeshgar, Sima; Lacefield, Soni

Safety learning induces postsynaptic potentiation of direct pathway spiny projection neurons in the tail of the striatum.

安全学习可诱导纹状体尾部直接通路棘状投射神经元的突触后增强

Stanley Adrien, Choi Se Joon, Frank Anika, Makowicz Emily, Cataldi Stefano, N Shashaank, Mosharov Eugene V, Lacefield Clay, Miniaci Maria Concetta, Sulzer David

A persistent intracellular bridge and cell cycle misregulation enable polar body cell divisions and tumor formation in Mos-deficient eggs.

在缺乏 Mos 的卵子中,持续存在的细胞内桥和细胞周期失调使得极体细胞分裂和肿瘤形成成为可能

Cairo Gisela, Haseeb Muhammad A, Schindler Karen, Lacefield Soni