Land Benjamin B相关文献与解析，生知库 | 链接生命科学的每一步

P2X(7) receptor-dependent increase in endocannabinoid 2-arachidonoyl glycerol production by neuronal cells in culture: Dynamics and mechanism.

P2X(7)受体依赖性增加培养神经元细胞内源性大麻素2-花生四烯酸甘油酯的产生：动力学和机制

Singh Simar, Sarroza Dennis, English Anthony, Whittington Dale, Dong Ao, Malamas Michael, Makriyannis Alexandros, van der Stelt Mario, Li Yulong, Zweifel Larry, Bruchas Michael R, Land Benjamin B, Stella Nephi

神经科学

发表日期：2024

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Analgesic Properties of Next-Generation Modulators of Endocannabinoid Signaling: Leveraging Modern Tools for the Development of Novel Therapeutics

新一代内源性大麻素信号调节剂的镇痛特性：利用现代工具开发新型疗法

期刊:Journal of Pharmacology and Experimental Therapeutics

影响因子:3.8

doi:10.1124/jpet.124.002119

Singh, Simar; Ellioff, Kaylin J; Bruchas, Michael R; Land, Benjamin B; Stella, Nephi

信号转导

发表日期：2024

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Pharmacological Characterization of the Endocannabinoid Sensor GRAB(eCB2.0).

内源性大麻素传感器 GRAB(eCB2.0) 的药理学特征

期刊:Cannabis and Cannabinoid Research

影响因子:2.7

doi:10.1089/can.2023.0036

Singh Simar, Sarroza Dennis, English Anthony, McGrory Maya, Dong Ao, Zweifel Larry, Land Benjamin B, Li Yulong, Bruchas Michael R, Stella Nephi

发表日期：2024

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Endogenous opioids facilitate stress-induced binge eating via an insular cortex-claustrum pathway

内源性阿片类物质通过岛叶皮层-屏状核通路促进应激诱发的暴食行为。

期刊:

影响因子:

doi:10.1101/2024.06.10.598168

Chen, Jingyi; Mangieri, Leandra; Mar, Sophia; Piantadosi, Sean; Marcus, David; Davis, Phoenix; Anger, Bennett; Land, Benjamin B; Bruchas, Michael R

信号转导

发表日期：2024

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Stress decreases serotonin tone in the nucleus accumbens in male mice to promote aversion and potentiate cocaine preference via decreased stimulation of 5-HT(1B) receptors

压力会降低雄性小鼠伏隔核中的血清素水平，从而通过减少 5-HT(1B) 受体的刺激来促进厌恶感并增强对可卡因的偏好。

期刊:Neuropsychopharmacology

影响因子:7.1

doi:10.1038/s41386-021-01178-0

Fontaine, Harrison M; Silva, Phillip R; Neiswanger, Carlie; Tran, Rachelle; Abraham, Antony D; Land, Benjamin B; Neumaier, John F; Chavkin, Charles

Release of endogenous dynorphin opioids in the prefrontal cortex disrupts cognition

前额叶皮层内源性强啡肽类阿片的释放会扰乱认知功能。

期刊:Neuropsychopharmacology

影响因子:7.1

doi:10.1038/s41386-021-01168-2

Abraham, Antony D; Casello, Sanne M; Schattauer, Selena S; Wong, Brenden A; Mizuno, Grace O; Mahe, Karan; Tian, Lin; Land, Benjamin B; Chavkin, Charles

神经科学

发表日期：2021

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Orally consumed cannabinoids provide long-lasting relief of allodynia in a mouse model of chronic neuropathic pain

口服大麻素可持久缓解慢性神经性疼痛小鼠模型中的痛觉过敏。

期刊:Neuropsychopharmacology

影响因子:7.1

doi:10.1038/s41386-019-0585-3

Abraham, Antony D; Leung, Edward J Y; Wong, Brenden A; Rivera, Zeena M G; Kruse, Lauren C; Clark, Jeremy J; Land, Benjamin B

发表日期：2020

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G Protein Inactivation as a Mechanism for Addiction Treatment

G蛋白失活作为成瘾治疗的一种机制

The Pro- But Not Antinociceptive Effects of Cannabidiol Depend on Trpa1b in Larval Zebrafish

大麻二酚的促伤害感受作用而非镇痛作用依赖于斑马鱼幼体中的Trpa1b。

Differential increase in endocannabinoid levels at the plasma and intracellular membranes

血浆膜和细胞内膜内源性大麻素水平的差异性升高

P2X(7) receptor-dependent increase in endocannabinoid 2-arachidonoyl glycerol production by neuronal cells in culture: Dynamics and mechanism.

P2X(7)受体依赖性增加培养神经元细胞内源性大麻素2-花生四烯酸甘油酯的产生：动力学和机制

Analgesic Properties of Next-Generation Modulators of Endocannabinoid Signaling: Leveraging Modern Tools for the Development of Novel Therapeutics

新一代内源性大麻素信号调节剂的镇痛特性：利用现代工具开发新型疗法

Pharmacological Characterization of the Endocannabinoid Sensor GRAB(eCB2.0).

内源性大麻素传感器 GRAB(eCB2.0) 的药理学特征

Endogenous opioids facilitate stress-induced binge eating via an insular cortex-claustrum pathway

内源性阿片类物质通过岛叶皮层-屏状核通路促进应激诱发的暴食行为。

Stress decreases serotonin tone in the nucleus accumbens in male mice to promote aversion and potentiate cocaine preference via decreased stimulation of 5-HT(1B) receptors

压力会降低雄性小鼠伏隔核中的血清素水平，从而通过减少 5-HT(1B) 受体的刺激来促进厌恶感并增强对可卡因的偏好。

Release of endogenous dynorphin opioids in the prefrontal cortex disrupts cognition

前额叶皮层内源性强啡肽类阿片的释放会扰乱认知功能。

Orally consumed cannabinoids provide long-lasting relief of allodynia in a mouse model of chronic neuropathic pain

口服大麻素可持久缓解慢性神经性疼痛小鼠模型中的痛觉过敏。