日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Nonsense-mediated mRNA decay safeguards telomeres in pluripotent stem cells.

无义介导的mRNA衰变保护多能干细胞中的端粒。

Markiewicz-Potoczny Marta, Lee Si Young, Chatterjee Soniya, Mabin Justin W, Zinsser Anna, Sandhu Ranjodh, Tricola Gianna, Hogg J Robert, Lazzerini Denchi Eros

Control of telomerase recruitment and end protection by independent shelterin components

端粒酶募集和末端保护由独立的端粒保护蛋白组分控制

Sandhu, Ranjodh; Tricola, Gianna M; Lee, Si Young; Tran, Andy; Lazzerini Denchi, Eros

Identification of modulators of the ALT pathway through a native FISH-based optical screen.

通过基于天然 FISH 的光学筛选鉴定 ALT 通路的调节因子

Azeroglu Benura, Khurana Simran, Wang Shih-Chun, Tricola Gianna M, Sharma Shalu, Jubelin Camille, Cortolezzis Ylenia, Pegoraro Gianluca, Miller Kyle M, Stracker Travis H, Lazzerini Denchi Eros

Conserved and unique features of terminal telomeric sequences in ALT-positive cancer cells.

ALT阳性癌细胞末端端粒序列的保守性和独特性

Azeroglu Benura, Wu Wei, Pavani Raphael, Sandhu Ranjodh Singh, Matsumoto Tadahiko, Nussenzweig André, Lazzerini-Denchi Eros

Stem cells at odds with telomere maintenance and protection

干细胞与端粒的维持和保护存在冲突

Penev, Alex; Markiewicz-Potoczny, Marta; Sfeir, Agnel; Lazzerini Denchi, Eros

Corrigendum: Hepatocytes with extensive telomere deprotection and fusion remain viable and regenerate liver mass through endoreduplication

更正:端粒广泛去保护和融合的肝细胞仍能存活,并通过内复制再生肝脏组织。

Lazzerini Denchi, Eros; Celli, Giulia; de Lange, Titia

Stop pulling my strings - what telomeres taught us about the DNA damage response

别再操纵我了——端粒教会我们关于DNA损伤反应的哪些知识

Lazzerini-Denchi, Eros; Sfeir, Agnel

Isolation of chromatin from dysfunctional telomeres reveals an important role for Ring1b in NHEJ-mediated chromosome fusions

从功能异常的端粒中分离染色质揭示了Ring1b在NHEJ介导的染色体融合中的重要作用

Bartocci, Cristina; Diedrich, Jolene K; Ouzounov, Iliana; Li, Julia; Piunti, Andrea; Pasini, Diego; Yates, John R 3rd; Lazzerini Denchi, Eros

RNF4 interacts with both SUMO and nucleosomes to promote the DNA damage response

RNF4 与 SUMO 和核小体相互作用,从而促进 DNA 损伤反应。

Groocock, Lynda M; Nie, Minghua; Prudden, John; Moiani, Davide; Wang, Tao; Cheltsov, Anton; Rambo, Robert P; Arvai, Andrew S; Hitomi, Chiharu; Tainer, John A; Luger, Karolin; Perry, J Jefferson P; Lazzerini-Denchi, Eros; Boddy, Michael N

Hepatocytes with extensive telomere deprotection and fusion remain viable and regenerate liver mass through endoreduplication

端粒保护作用广泛丧失并发生融合的肝细胞仍能存活,并通过内复制再生肝脏组织。

Lazzerini Denchi, Eros; Celli, Giulia; de Lange, Titia