Lebish相关文献与解析，生知库 | 链接生命科学的每一步

Wong, Jasmine C; Weinfurtner, Kelley M; Westover, Tamara; Kim, Jangkyung; Lebish, Eric J; Del Pilar Alzamora, Maria; Huang, Benjamin J; Walsh, Michael; Abdelhamed, Sherif; Ma, Jing; Klco, Jeffery M; Shannon, Kevin

肿瘤

肿瘤免疫

发表日期：2024

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Mucosal-associated invariant T cells modulate innate immune cells and inhibit colon cancer growth

黏膜相关不变T细胞调节固有免疫细胞并抑制结肠癌生长

期刊:Scandinavian Journal of Immunology

影响因子:4.1

doi:10.1111/sji.13391

Olivia J Cheng ，Eric J Lebish ，Owen Jensen ，Damian Jacenik ，Shubhanshi Trivedi ，Jackson G Cacioppo ，Jeffrey Aubé ，Ellen J Beswick ，Daniel T Leung

MAIT Cells Modulate Innate Immune Cells and Inhibit Colon Cancer Growth

MAIT 细胞调节先天免疫细胞并抑制结肠癌生长

期刊:

影响因子:

doi:10.1101/2024.01.16.575894

Olivia J Cheng, Eric J Lebish, Owen Jensen, Damian Jacenik, Shubhanshi Trivedi, Jackson Cacioppo, Jeffrey Aubé, Ellen J Beswick, Daniel T Leung

MK2 drives progression of pancreas and colon cancers by suppressing CD8(+) T cell cytotoxic function and is a potential immunotherapy target

MK2通过抑制CD8(+) T细胞的细胞毒性功能促进胰腺癌和结肠癌的进展，是一种潜在的免疫治疗靶点。

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2023.1212100

Jacenik, Damian; Lebish, Eric J; Beswick, Ellen J

Prenatal inflammation perturbs murine fetal hematopoietic development and causes persistent changes to postnatal immunity

产前炎症会扰乱小鼠胎儿造血发育，并导致出生后免疫力发生持续性改变。

期刊:Cell Reports

影响因子:7.5

doi:10.1016/j.celrep.2022.111677

Diego A López ，April C Apostol ，Eric J Lebish ，Clint H Valencia ，Mari Carmen Romero-Mulero ，Polina V Pavlovich ，Gloria E Hernandez ，E Camilla Forsberg ，Nina Cabezas-Wallscheid ，Anna E Beaudin

MK2 Promotes the Development and Progression of Pancreatic Neuroendocrine Tumors Mediated by Macrophages and Metabolomic Factors

MK2通过巨噬细胞和代谢组学因子促进胰腺神经内分泌肿瘤的发生发展

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms232113561

Jacenik, Damian; Lebish, Eric J; Beswick, Ellen J

MK2 Inhibitors as a Potential Crohn's Disease Treatment Approach for Regulating MMP Expression, Cleavage of Checkpoint Molecules and T Cell Activity

MK2 抑制剂作为克罗恩病的潜在治疗方法，可调节 MMP 表达、检查点分子的裂解和 T 细胞活性

期刊:Pharmaceuticals

影响因子:

doi:10.3390/ph15121508

Eric J Lebish, Natalie J Morgan, John F Valentine, Ellen J Beswick

5G2 mutant mice model loss of a commonly deleted segment of chromosome 7q22 in myeloid malignancies

5G2突变小鼠模型模拟了髓系恶性肿瘤中常见的7q22染色体片段缺失。

Mucosal-associated invariant T cells modulate innate immune cells and inhibit colon cancer growth

黏膜相关不变T细胞调节固有免疫细胞并抑制结肠癌生长

MAIT Cells Modulate Innate Immune Cells and Inhibit Colon Cancer Growth

MAIT 细胞调节先天免疫细胞并抑制结肠癌生长

MK2 drives progression of pancreas and colon cancers by suppressing CD8(+) T cell cytotoxic function and is a potential immunotherapy target

MK2通过抑制CD8(+) T细胞的细胞毒性功能促进胰腺癌和结肠癌的进展，是一种潜在的免疫治疗靶点。

Prenatal inflammation perturbs murine fetal hematopoietic development and causes persistent changes to postnatal immunity

产前炎症会扰乱小鼠胎儿造血发育，并导致出生后免疫力发生持续性改变。

MK2 Promotes the Development and Progression of Pancreatic Neuroendocrine Tumors Mediated by Macrophages and Metabolomic Factors

MK2通过巨噬细胞和代谢组学因子促进胰腺神经内分泌肿瘤的发生发展

MK2 Inhibitors as a Potential Crohn's Disease Treatment Approach for Regulating MMP Expression, Cleavage of Checkpoint Molecules and T Cell Activity

MK2 抑制剂作为克罗恩病的潜在治疗方法，可调节 MMP 表达、检查点分子的裂解和 T 细胞活性