Nr4a1 and Nr4a3 redundantly control clonal deletion and contribute to an anergy-like transcriptome in auto-reactive thymocytes to impose tolerance in mice

Nr4a1 和 Nr4a3 冗余地控制克隆清除，并在自身反应性胸腺细胞中促成类似无反应性的转录组，从而在小鼠中诱导免疫耐受。