日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Recombinant growth hormone improves growth and adult height: a comparison between treated and untreated patients with idiopathic growth hormone deficiency

重组生长激素可改善生长发育和成年身高:特发性生长激素缺乏症患者治疗组与未治疗组的比较

Zhao, Qianqian; Li, Rong; Shao, Qian; Zhang, Mei; Ban, Bo

Human CD56(+)CD39(+) dNK cells support fetal survival through controlling trophoblastic cell fate: immune mechanisms of recurrent early pregnancy loss

人CD56(+)CD39(+) dNK细胞通过控制滋养层细胞命运来支持胎儿存活:复发性早期妊娠丢失的免疫机制

Jia, Wentong; Ma, Liyang; Yu, Xin; Wang, Feiyang; Yang, Qian; Wang, Xiaoye; Fan, Mengjie; Gu, Yan; Meng, Ran; Wang, Jian; Li, Yuxia; Li, Rong; Shao, Xuan; Wang, Yan-Ling

Sulforaphane Activates a lysosome-dependent transcriptional program to mitigate oxidative stress

萝卜硫素激活溶酶体依赖性转录程序以减轻氧化应激

Dan Li, Rong Shao, Na Wang, Nan Zhou, Kaili Du, Jiahui Shi, Yihan Wang, Zhuangzhuang Zhao, Xin Ye, Xiaoli Zhang, Haoxing Xu

Rapamycin directly activates lysosomal mucolipin TRP channels independent of mTOR

雷帕霉素直接激活溶酶体粘脂蛋白TRP通道,不依赖于mTOR

Xiaoli Zhang, Wei Chen, Qiong Gao, Junsheng Yang, Xueni Yan, Han Zhao, Lin Su, Meimei Yang, Chenlang Gao, Yao Yao, Ken Inoki, Dan Li, Rong Shao, Shiyi Wang, Nirakar Sahoo, Fumitaka Kudo, Tadashi Eguchi, Benfang Ruan, Haoxing Xu

The Glycolytic Metabolite, Fructose-1,6-bisphosphate, Blocks Epileptiform Bursts by Attenuating Voltage-Activated Calcium Currents in Hippocampal Slices

糖酵解代谢物果糖-1,6-二磷酸通过减弱海马切片中的电压激活钙电流来阻断癫痫样发作

Li-Rong Shao, Guangxin Wang, Carl E Stafstrom

Glycolytic inhibition by 2-deoxy-d-glucose abolishes both neuronal and network bursts in an in vitro seizure model

2-脱氧-D-葡萄糖的糖酵解抑制可消除体外癫痫模型中的神经元和网络爆发

Li-Rong Shao, Carl E Stafstrom

Synaptic and extrasynaptic plasticity in glutamatergic circuits involving dentate granule cells following chronic N-methyl-D-aspartate receptor inhibition

慢性 N-甲基-D-天冬氨酸受体抑制后,涉及齿状颗粒细胞的谷氨酸能回路的突触和突触外可塑性

Shuijin He, Li-Rong Shao, Yu Wang, Suzanne B Bausch

Increased Kv1 channel expression may contribute to decreased sIPSC frequency following chronic inhibition of NR2B-containing NMDAR

长期抑制含有 NR2B 的 NMDAR 后,Kv1 通道表达增加可能导致 sIPSC 频率降低

Shuijin He, Li-Rong Shao, W Bradley Rittase, Suzanne B Bausch