日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

The immune landscape of systemic sclerosis: from pathogenic mechanisms to precision therapeutic breakthroughs

系统性硬化症的免疫图谱:从致病机制到精准治疗突破

Liu, Mengguo

miR-3606-3p alleviates skin fibrosis by integratively suppressing the integrin/FAK, p-AKT/p-ERK, and TGF-β signaling cascades.

miR-3606-3p 通过整合抑制整合素/FAK、p-AKT/p-ERK 和 TGF-β 信号级联来缓解皮肤纤维化。

Chen Yahui, Gong Yiyi, Shi Mengkun, Zhu Haoxing, Tang Yulong, Huang Delin, Wang Wei, Shi Chenyi, Xia Xueyi, Zhang Ying, Liu Jianlan, Huang Jia, Liu Mengguo, Chen Huyan, Ma Yanyun, Wang Ziyu, Wang Lei, Tu Wenzhen, Zhao Yinhuan, Lin Jinran, Jin Li, Distler Jörg Hw, Wu Wenyu, Wang Jiucun, Shi Xiangguang

Ferroptosis-Resistant Adipocytes Drive Keloid Pathogenesis via GPX4-Mediated Adipocyte-Mesenchymal Transition and Iron-Cystine Metabolic Communication.

抗铁死亡脂肪细胞通过 GPX4 介导的脂肪细胞-间质转化和铁-胱氨酸代谢通讯驱动瘢痕疙瘩的发病机制

Shi Xiangguang, Xia Xueyi, Xiao Yang, Shu Huizhen, Xu Zhuoya, Liu Mengguo, Shi Chenyi, Zhang Ying, Wei Yining, Gong Yiyi, Wang Wei, Chen Yahui, Liu Jianlan, Huang Jia, Shi Mengkun, Wang Jiucun, Wu Wenyu

Increased melanin induces aberrant keratinocyte - melanocyte - basal - fibroblast cell communication and fibrogenesis by inducing iron overload and ferroptosis resistance in keloids.

黑色素增多会诱导瘢痕疙瘩中角质形成细胞-黑色素细胞-基底细胞-成纤维细胞之间的异常通讯和纤维化,其机制是通过诱导铁过载和铁死亡抵抗

Shi Xiangguang, Xia Xueyi, Xiao Yang, Zhang Ying, Gong Yiyi, Chen Yahui, Shi Chenyi, Wang Wei, Liu Jianlan, Huang Jia, Liu Mengguo, Xu Zhuoya, Ma Yanyun, Shi Mengkun, Wang Jiucun, Wu Wenyu

Cytokines, chemokines and growth factors involved in keloids pathogenesis

参与瘢痕疙瘩发病机制的细胞因子、趋化因子和生长因子

Liu, Mengguo

Multivariate Analysis and Prediction Model Construction for Dermatoscope Use by Chinese Dermatologists via an Online Survey

通过在线调查对中国皮肤科医生使用皮肤镜的情况进行多元分析和预测模型构建

Liu, Mengguo; Xu, Feng

Effector and regulatory B-cell imbalance in systemic sclerosis: cooperation or competition?

系统性硬化症中效应B细胞和调节性B细胞失衡:是合作还是竞争?

Liu, Mengguo

Dermoscopy of cutaneous sarcoidosis: a cross-sectional study

皮肤结节病的皮肤镜检查:一项横断面研究

Liu, Mengguo; Chen, Huyan; Xu, Feng

Detection of skin thickness and density in healthy Chinese people by using high-frequency ultrasound

利用高频超声检测健康中国人的皮肤厚度和密度

Wang, Shangshang; Yu, Rui-Xing; Fan, Wen; Li, Cheng-Xu; Fei, Wen-Min; Li, Shanshan; Zhou, Junfeng; Hu, Ruiming; Liu, Mengguo; Xu, Feng; Xu, Jinhua; Cui, Yong

CTHRC1+ fibroblasts are stimulated by macrophage-secreted SPP1 to induce excessive collagen deposition in keloids

CTHRC1+成纤维细胞受到巨噬细胞分泌的SPP1刺激,从而诱导瘢痕疙瘩中胶原蛋白过度沉积。

Liu, Jing; Huang, Yan; Gong, Yiyi; Liu, Qingmei; Lin, Jinran; Liu, Jianlan; Liu, Mengguo; Huang, Jia; Pu, Weilin; Ma, Yanyun; Zhang, Yuting; Li, Haiyang; Shi, Xiangguang; Zhang, Yue; Wang, Jian; Zhu, Yifei; Wang, Qin; Wei, Kelu; Wang, Jiayi; Sha, Yu'ou; Wang, Jiucun; Wu, Wenyu