日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Editorial: Cell death programs in the pathogenesis of heart disease

社论:心脏病发病机制中的细胞死亡程序

Kwong, Jennifer Q; Correll, Robert N; Liu, Qinghang

Lipid peroxidation intrinsically induces mitochondrial iron overload via Bach1-HO-1 signaling to promote cardiac ferroptosis

脂质过氧化通过 Bach1-HO-1 信号通路诱导线粒体铁过载,从而促进心脏铁死亡。

Guo, Xiaoyun; Chen, Yi; Zeng, Yachang; Mo, Xiaoliang; Hong, Siqi; He, Hui; Li, Jing; Zhang, Kevin; Liu, Qinghang

Reformulation of the Necroptosis Pathway in Reperfused Myocardial Infarction

再灌注心肌梗死中坏死性凋亡途径的重新构建

Qin, Dongze; Modanwal, Radheshyam; Ghazi, Madeeha; Corbalan, J Jose; Jia, Xiaotong F; Axelrod, Joshua L; Mesidor, Roneldine; Jang, Hee Jae; Humeres, Claudio; Hanna, Anis; Varzideh, Fahimeh; Tallquist, Michelle D; Santulli, Gaetano; Liu, Qinghang; Kirshenbaum, Lorrie A; Ni, Hongmin; Kamp, Timothy J; Gundry, Rebekah L; Balachandran, Siddharth; Degterev, Alexei; Calvert, John W; Kitsis, Richard N

Role of ferroptosis in the pathogenesis of heart disease

铁死亡在心脏病发病机制中的作用

Fatima, Sulail; Zhou, Haiyan; Chen, Yi; Liu, Qinghang

Necroptosis in heart disease: Molecular mechanisms and therapeutic implications

心脏病中的坏死性凋亡:分子机制和治疗意义

Guo, Xiaoyun; Chen, Yi; Liu, Qinghang

ITCH puts the brakes on septic cardiomyopathy

ITCH 可抑制脓毒性心肌病

Del Re, Dominic P; Liu, Qinghang

CYLD exaggerates pressure overload-induced cardiomyopathy via suppressing autolysosome efflux in cardiomyocytes

CYLD通过抑制心肌细胞中的自溶酶体外排,加剧压力超负荷诱发的心肌病。

Qi, Lei; Zang, Huimei; Wu, Weiwei; Nagarkatti, Prakash; Nagarkatti, Mitzi; Liu, Qinghang; Robbins, Jeffrey; Wang, Xuejun; Cui, Taixing

Fundamental Mechanisms of Regulated Cell Death and Implications for Heart Disease

调控性细胞死亡的基本机制及其对心脏病的影响

Del Re, Dominic P; Amgalan, Dulguun; Linkermann, Andreas; Liu, Qinghang; Kitsis, Richard N

Cardioprotective Role of Tumor Necrosis Factor Receptor-Associated Factor 2 by Suppressing Apoptosis and Necroptosis.

肿瘤坏死因子受体相关因子 2 通过抑制细胞凋亡和坏死性凋亡发挥心脏保护作用

Guo Xiaoyun, Yin Haifeng, Li Lei, Chen Yi, Li Jing, Doan Jessica, Steinmetz Rachel, Liu Qinghang

Loss of AKAP150 promotes pathological remodelling and heart failure propensity by disrupting calcium cycling and contractile reserve

AKAP150 的缺失会破坏钙循环和收缩储备,从而促进病理性重塑和心力衰竭倾向。

Li, Lei; Li, Jing; Drum, Benjamin M; Chen, Yi; Yin, Haifeng; Guo, Xiaoyun; Luckey, Stephen W; Gilbert, Merle L; McKnight, G Stanley; Scott, John D; Santana, L Fernando; Liu, Qinghang