Oligodendrocyte-specific overexpression of human alpha-synuclein results in elevated MBP levels and inflammatory responses in TgM83 mice, mimicking the pathological features of multiple system atrophy.
人类α-突触核蛋白在少突胶质细胞中的特异性过表达导致TgM83小鼠体内MBP水平升高和炎症反应，模拟多系统萎缩的病理特征
期刊:Acta Neuropathologica Communications
影响因子:5.7
doi:10.1186/s40478-025-02014-y
Liu Sam Chi-Hao, Chang Koping, Chen Meng-Ling, Kuo Ming-Che, Wang Teh-Cheng, Wu Ruey-Meei
