Liu Zheng-Gang相关文献与解析，生知库 | 链接生命科学的每一步

TGFB signaling induces mitophagy via PLSCR3-mediated cardiolipin externalization in conjunction with a BNIP3L/NIX-, BNIP3-, and FUNDC1-dependent mechanism.

TGFB 信号通过 PLSCR3 介导的心磷脂外化诱导线粒体自噬，并依赖于 BNIP3L/NIX、BNIP3 和 FUNDC1 机制

期刊:Autophagy

影响因子:14.3

doi:10.1080/15548627.2025.2483441

Yan Jiong, Chen Xin, Choksi Swati, Liu Zheng-Gang

ATF4-mediated expression of NOXA is critical for Necroptosis driven by Glucose Deprivation

ATF4介导的NOXA表达对于葡萄糖剥夺驱动的坏死性凋亡至关重要

期刊:

影响因子:

doi:10.3389/fceld.2024.1507960

Sonkaew, Sasiprapa; Rajna, Ruwaida; Park, Yeon-Ji; Yan, Jiong; Liu, Zhaoshan; Jitkaew, Siriporn; Liu, Zheng-Gang; Choksi, Swati

Tumor necroptosis-mediated shedding of cell surface proteins promotes metastasis of breast cancer by suppressing anti-tumor immunity

肿瘤细胞坏死介导的细胞表面蛋白脱落通过抑制抗肿瘤免疫促进乳腺癌转移。

期刊:Breast Cancer Research

影响因子:6.1

doi:10.1186/s13058-023-01604-9

Zhaoshan Liu # ，Swati Choksi # ，Hyung-Joon Kwon ，Delong Jiao ，Chengyu Liu ，Zheng-Gang Liu

Comparative proteomic analysis of exosomes derived from endothelial cells and Schwann cells

内皮细胞和雪旺细胞外泌体的比较蛋白质组学分析

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0290155

Lei Wang, XueRong Lu, Michael Chopp, Chao Li, Yi Zhang, Alexandra Szalad, Xian Shuang Liu, Zheng Gang Zhang

Mitochondria engage the integrated stress response to promote tumor growth

线粒体启动整合应激反应以促进肿瘤生长

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.28404

Liu, Zhaoshan; Choksi, Swati; Liu, Zheng-Gang; Boulton, Dillon P; Caino, M Cecilia

Transition from TNF-Induced Inflammation to Death Signaling

从TNF诱导的炎症到死亡信号的转变

期刊:Methods in Molecular Biology

影响因子:

doi:10.1007/978-1-0716-1130-2_5

Choksi, Swati; Choudhary, Gourav; Liu, Zheng-Gang

炎症/感染

信号转导

Immunology/Inflammation

炎症

发表日期：2021

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G9A promotes gastric cancer metastasis by upregulating ITGB3 in a SET domain-independent manner

G9A以SET结构域独立的方式上调ITGB3促进胃癌转移

期刊:Cell Death & Disease

影响因子:8.1

doi:10.1038/s41419-018-0322-6

Lei Hu, Ming-de Zang, He-Xiao Wang, Bao-Gui Zhang, Zhen-Qiang Wang, Zhi-Yuan Fan, Huo Wu, Jian-Fang Li, Li-Ping Su, Min Yan, Zhi-Qiang Zhu, Qiu-Meng Yang, Qiang Huang, Bing-Ya Liu, Zheng-Gang Zhu

The cytoplasmic nuclear receptor RARγ controls RIP1 initiated cell death when cIAP activity is inhibited

当 cIAP 活性受到抑制时，细胞质核受体 RARγ 控制 RIP1 启动的细胞死亡

期刊:Nature Communications

影响因子:14.7

doi:10.1038/s41467-017-00496-6

Qing Xu, Siriporn Jitkaew, Swati Choksi, Chamila Kadigamuwa, Jianhui Qu, Moran Choe, Jonathan Jang, Chengyu Liu, Zheng-Gang Liu

Biglycan stimulates VEGF expression in endothelial cells by activating the TLR signaling pathway

Biglycan 通过激活 TLR 信号通路刺激内皮细胞中的 VEGF 表达

期刊:Molecular Oncology

影响因子:5

doi:10.1016/j.molonc.2016.08.002

Lei Hu, Ming-de Zang, He-Xiao Wang, Jian-Fang Li, Li-Ping Su, Min Yan, Chen Li, Qiu-Meng Yang, Bing-Ya Liu, Zheng-Gang Zhu

Soluble tissue factor generated by necroptosis-triggered shedding is responsible for thrombosis.

坏死性凋亡触发的脱落产生的可溶性组织因子是血栓形成的原因。

TGFB signaling induces mitophagy via PLSCR3-mediated cardiolipin externalization in conjunction with a BNIP3L/NIX-, BNIP3-, and FUNDC1-dependent mechanism.

TGFB 信号通过 PLSCR3 介导的心磷脂外化诱导线粒体自噬，并依赖于 BNIP3L/NIX、BNIP3 和 FUNDC1 机制

ATF4-mediated expression of NOXA is critical for Necroptosis driven by Glucose Deprivation

ATF4介导的NOXA表达对于葡萄糖剥夺驱动的坏死性凋亡至关重要

Tumor necroptosis-mediated shedding of cell surface proteins promotes metastasis of breast cancer by suppressing anti-tumor immunity

肿瘤细胞坏死介导的细胞表面蛋白脱落通过抑制抗肿瘤免疫促进乳腺癌转移。

Comparative proteomic analysis of exosomes derived from endothelial cells and Schwann cells

内皮细胞和雪旺细胞外泌体的比较蛋白质组学分析

Mitochondria engage the integrated stress response to promote tumor growth

线粒体启动整合应激反应以促进肿瘤生长

Transition from TNF-Induced Inflammation to Death Signaling

从TNF诱导的炎症到死亡信号的转变

G9A promotes gastric cancer metastasis by upregulating ITGB3 in a SET domain-independent manner

G9A以SET结构域独立的方式上调ITGB3促进胃癌转移

The cytoplasmic nuclear receptor RARγ controls RIP1 initiated cell death when cIAP activity is inhibited

当 cIAP 活性受到抑制时，细胞质核受体 RARγ 控制 RIP1 启动的细胞死亡

Biglycan stimulates VEGF expression in endothelial cells by activating the TLR signaling pathway

Biglycan 通过激活 TLR 信号通路刺激内皮细胞中的 VEGF 表达