日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Antrum Mucosal Protein-18 Peptide Targets Tight Junctions to Protect and Heal Barrier Structure and Function in Models of Inflammatory Bowel Disease

胃窦黏膜蛋白-18肽靶向紧密连接,在炎症性肠病模型中保护和修复屏障结构和功能

Chen, Peili; Bakke, Danika; Kolodziej, Lauren; Lodolce, James; Weber, Christopher R; Boone, David L; Toback, F Gary

Intestinal epithelial expression of TNFAIP3 results in microbial invasion of the inner mucus layer and induces colitis in IL-10-deficient mice

肠上皮表达 TNFAIP3 导致微生物侵入内粘液层并在 IL-10 缺乏的小鼠中诱发结肠炎

Stephen F Murphy, Lesley Rhee, Wesley A Grimm, Christopher R Weber, Jeannette S Messer, James P Lodolce, Jonathan E Chang, Sarah J Bartulis, Thomas Nero, Renata A Kukla, Gordon MacDougall, Charles Binghay, Lauren E Kolodziej, David L Boone

The Crohn's disease: associated ATG16L1 variant and Salmonella invasion

克罗恩病:与ATG16L1变异和沙门氏菌入侵相关

Messer, Jeannette S; Murphy, Stephen F; Logsdon, Mark F; Lodolce, James P; Grimm, Wesley A; Bartulis, Sarah J; Vogel, Tiphanie P; Burn, Melisa; Boone, David L

A prescription for improving drug formulary decision making

改进药品目录决策的方案

Schiff, Gordon D; Galanter, William L; Duhig, Jay; Koronkowski, Michael J; Lodolce, Amy E; Pontikes, Pam; Busker, John; Touchette, Daniel; Walton, Surrey; Lambert, Bruce L

TNFAIP3 maintains intestinal barrier function and supports epithelial cell tight junctions

TNFAIP3 维持肠道屏障功能并支持上皮细胞紧密连接

Lauren E Kolodziej, James P Lodolce, Jonathan E Chang, Jeffrey R Schneider, Wesley A Grimm, Sarah J Bartulis, Xiaorong Zhu, Jeannette S Messer, Stephen F Murphy, Nishith Reddy, Jerrold R Turner, David L Boone

African-derived genetic polymorphisms in TNFAIP3 mediate risk for autoimmunity

源自非洲的TNFAIP3基因多态性与自身免疫性疾病的风险相关

Lodolce, James P; Kolodziej, Lauren E; Rhee, Lesley; Kariuki, Silvia N; Franek, Beverly S; McGreal, Nancy M; Logsdon, Mark F; Bartulis, Sarah J; Perera, Minoli A; Ellis, Nathan A; Adams, Erin J; Hanauer, Stephen B; Jolly, Meenakshi; Niewold, Timothy B; Boone, David L

Interleukin (IL)-15R[alpha]-deficient natural killer cells survive in normal but not IL-15R[alpha]-deficient mice

白细胞介素 (IL)-15Rα 缺乏的自然杀伤细胞在正常小鼠中存活,但在 IL-15Rα 缺乏的小鼠中则不能存活

Rima Koka, Patrick R Burkett, Marcia Chien, Sophia Chai, Faye Chan, James P Lodolce, David L Boone, Averil Ma

T cell-independent interleukin 15Ralpha signals are required for bystander proliferation

旁观者增殖需要 T 细胞独立的白细胞介素 15Ralpha 信号

J P Lodolce, P R Burkett, D L Boone, M Chien, A Ma

The pleiotropic functions of interleukin 15: not so interleukin 2-like after all

白细胞介素 15 的多效性功能:并非完全像白细胞介素 2 那样

Ma, A; Boone, D L; Lodolce, J P