日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Attomolar fecal cytokine profiling reveals gut immune dynamics and disease states

阿托摩尔级粪便细胞因子谱分析揭示肠道免疫动态和疾病状态

Zhang, Stephanie J; Sharma, Utkarsh; Senussi, Yasmeen; Dayao, Arvin; Sveen, Mia; Brown, Madison; Lomphithak, Thanpisit; Nguyen, My; Lawless, Aleigha R; Briskin, Camille A; Sharova, Tatyana; Boland, Genevieve M; Cohen, Sonia; Snapper, Scott; Gazzaniga, Francesca S; Bry, Lynn; Walt, David R; Gibson, Travis E

Necroptosis-driven T cell activation promotes IL-6-mediated PD-L1 upregulation in cholangiocarcinoma cells: IL-6 gene signature as a biomarker for chemo-immunotherapy response

坏死性凋亡驱动的T细胞活化促进胆管癌细胞中IL-6介导的PD-L1上调:IL-6基因特征作为化疗-免疫治疗反应的生物标志物

Thanpisit Lomphithak ,Nattaya Duangthim ,Sasiprapa Sonkaew ,Siriporn Jitkaew

Prognostic significance and response to immune checkpoint inhibitors of RIPK3, MLKL and necroptosis in non-small cell lung cancer

非小细胞肺癌中 RIPK3、MLKL 和坏死凋亡的免疫检查点抑制剂的预后意义及反应

Nattaya Duangthim, Thanpisit Lomphithak, Ryoko Saito-Koyama, Yasuhiro Miki, Chihiro Inoue, Ikuro Sato, Eisaku Miyauchi, Jiro Abe, Hironobu Sasano, Siriporn Jitkaew

Natural Flavonoids Quercetin and Kaempferol Targeting G2/M Cell Cycle-Related Genes and Synergize with Smac Mimetic LCL-161 to Induce Necroptosis in Cholangiocarcinoma Cells

天然黄酮类化合物槲皮素和山奈酚靶向 G2/M 细胞周期相关基因并与 Smac 模拟物 LCL-161 协同作用,诱导胆管癌细胞坏死性凋亡

Thanpisit Lomphithak, Patthorn Jaikla, Apiwit Sae-Fung, Sasiprapa Sonkaew, Siriporn Jitkaew

Die hard: cell death mechanisms and their implications in nanotoxicology

顽固不化:细胞死亡机制及其在纳米毒理学中的意义

Lomphithak, Thanpisit; Fadeel, Bengt

High-Dose Exposure to Polymer-Coated Iron Oxide Nanoparticles Elicits Autophagy-Dependent Ferroptosis in Susceptible Cancer Cells

高剂量暴露于聚合物包覆的氧化铁纳米粒子会在易感癌细胞中引发自噬依赖性铁死亡

Thanpisit Lomphithak, Selin Helvacioglu, Ilaria Armenia, Sandeep Keshavan, Jesús G Ovejero, Giovanni Baldi, Costanza Ravagli, Valeria Grazú, Bengt Fadeel

Tumor necroptosis is correlated with a favorable immune cell signature and programmed death-ligand 1 expression in cholangiocarcinoma

肿瘤坏死性凋亡与胆管癌中的有利免疫细胞特征和程序性死亡配体 1 表达相关

Thanpisit Lomphithak, Perawatt Akara-Amornthum, Keigo Murakami, Masatoshi Hashimoto, Hajime Usubuchi, Erina Iwabuchi, Michiaki Unno, Zhenyu Cai, Hironobu Sasano, Siriporn Jitkaew

Receptor-interacting protein kinase 1 is a key mediator in TLR3 ligand and Smac mimetic-induced cell death and suppresses TLR3 ligand-promoted invasion in cholangiocarcinoma

受体相互作用蛋白激酶 1 是 TLR3 配体和 Smac 模拟物诱导细胞死亡的关键介质,并抑制 TLR3 配体促进的胆管癌侵袭

Thanpisit Lomphithak, Swati Choksi, Apiwat Mutirangura, Rutaiwan Tohtong, Tewin Tencomnao, Hajime Usubuchi, Michiaki Unno, Hironobu Sasano, Siriporn Jitkaew

Key necroptotic proteins are required for Smac mimetic-mediated sensitization of cholangiocarcinoma cells to TNF-α and chemotherapeutic gemcitabine-induced necroptosis

关键的坏死性凋亡蛋白是 Smac 模拟物介导的胆管癌细胞对 TNF-α 的敏感化以及化疗药物吉西他滨诱导的坏死性凋亡所必需的

Perawatt Akara-Amornthum, Thanpisit Lomphithak, Swati Choksi, Rutaiwan Tohtong, Siriporn Jitkaew