Lu Yongju相关文献与解析，生知库 | 链接生命科学的每一步

Proteomic Characterization of the World Trade Center dust-activated mdig and c-myc signaling circuit linked to multiple myeloma

世界贸易中心尘埃激活的mdig和c-myc信号通路与多发性骨髓瘤相关的蛋白质组学特征

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/srep36305

Wu, Kai; Li, Lingzhi; Thakur, Chitra; Lu, Yongju; Zhang, Xiangmin; Yi, Zhengping; Chen, Fei

Gene expression profile of human lung epithelial cells chronically exposed to single-walled carbon nanotubes

长期暴露于单壁碳纳米管的人肺上皮细胞的基因表达谱

期刊:Nanoscale Research Letters

影响因子:

doi:10.1186/s11671-014-0707-0

Chen, Dongquan; Stueckle, Todd A; Luanpitpong, Sudjit; Rojanasakul, Yon; Lu, Yongju; Wang, Liying

Carcinogenic metalloid arsenic induces expression of mdig oncogene through JNK and STAT3 activation

致癌类金属砷通过激活 JNK 和 STAT3 诱导 mdig 癌基因的表达。

期刊:Cancer Letters

影响因子:10.1

doi:10.1016/j.canlet.2014.01.002

Sun, Jiaying; Yu, Miaomiao; Lu, Yongju; Thakur, Chitra; Chen, Bailing; Qiu, Ping; Zhao, Hongwen; Chen, Fei

Reactive oxygen species contribute to arsenic-induced EZH2 phosphorylation in human bronchial epithelial cells and lung cancer cells

活性氧促进砷诱导的人支气管上皮细胞和肺癌细胞中EZH2的磷酸化。

期刊:Toxicology and Applied Pharmacology

影响因子:3.4

doi:10.1016/j.taap.2014.02.005

Li, Lingzhi; Qiu, Ping; Chen, Bailing; Lu, Yongju; Wu, Kai; Thakur, Chitra; Chang, Qingshan; Sun, Jiaying; Chen, Fei

Reactive oxygen species-mediated p38 MAPK regulates carbon nanotube-induced fibrogenic and angiogenic responses.

活性氧介导的 p38 MAPK 调节碳纳米管诱导的纤维化和血管生成反应

期刊:Nanotoxicology

影响因子:3.4

doi:10.3109/17435390.2011.647929

Azad Neelam, Iyer Anand Krishnan V, Wang Liying, Liu Yuxin, Lu Yongju, Rojanasakul Yon

Mdig de-represses H19 large intergenic non-coding RNA (lincRNA) by down-regulating H3K9me3 and heterochromatin

Mdig通过下调H3K9me3和异染色质来解除H19大片段基因间非编码RNA（lincRNA）的抑制。

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.1155

Chen, Bailing; Yu, Miaomiao; Chang, Qingshan; Lu, Yongju; Thakur, Chitra; Ma, Danjun; Yi, Zhenping; Chen, Fei

发表日期：2013

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JNK-dependent Stat3 phosphorylation contributes to Akt activation in response to arsenic exposure.

砷暴露后，JNK 依赖的 Stat3 磷酸化促进 Akt 激活

期刊:Toxicological Sciences

影响因子:4.1

doi:10.1093/toxsci/kfs199

Liu Jia, Chen Bailing, Lu Yongju, Guan Youfei, Chen Fei

Chronic occupational exposure to arsenic induces carcinogenic gene signaling networks and neoplastic transformation in human lung epithelial cells

长期职业性接触砷会诱导人肺上皮细胞产生致癌基因信号通路并发生肿瘤转化。

期刊:Toxicology and Applied Pharmacology

影响因子:3.4

doi:10.1016/j.taap.2012.04.003

Stueckle, Todd A; Lu, Yongju; Davis, Mary E; Wang, Liying; Jiang, Bing-Hua; Holaskova, Ida; Schafer, Rosana; Barnett, John B; Rojanasakul, Yon

Carbon nanotubes induce malignant transformation and tumorigenesis of human lung epithelial cells

碳纳米管诱导人肺上皮细胞恶性转化和肿瘤发生

期刊:Nano Letters

影响因子:9.1

doi:10.1021/nl2011214

Wang, Liying; Luanpitpong, Sudjit; Castranova, Vincent; Tse, William; Lu, Yongju; Pongrakhananon, Varisa; Rojanasakul, Yon

Metabolic and epigenetic reprogramming in the arsenic-induced cancer stem cells

砷诱导的癌症干细胞的代谢和表观遗传重编程

Proteomic Characterization of the World Trade Center dust-activated mdig and c-myc signaling circuit linked to multiple myeloma

世界贸易中心尘埃激活的mdig和c-myc信号通路与多发性骨髓瘤相关的蛋白质组学特征

Gene expression profile of human lung epithelial cells chronically exposed to single-walled carbon nanotubes

长期暴露于单壁碳纳米管的人肺上皮细胞的基因表达谱

Carcinogenic metalloid arsenic induces expression of mdig oncogene through JNK and STAT3 activation

致癌类金属砷通过激活 JNK 和 STAT3 诱导 mdig 癌基因的表达。

Reactive oxygen species contribute to arsenic-induced EZH2 phosphorylation in human bronchial epithelial cells and lung cancer cells

活性氧促进砷诱导的人支气管上皮细胞和肺癌细胞中EZH2的磷酸化。

Reactive oxygen species-mediated p38 MAPK regulates carbon nanotube-induced fibrogenic and angiogenic responses.

活性氧介导的 p38 MAPK 调节碳纳米管诱导的纤维化和血管生成反应

Mdig de-represses H19 large intergenic non-coding RNA (lincRNA) by down-regulating H3K9me3 and heterochromatin

Mdig通过下调H3K9me3和异染色质来解除H19大片段基因间非编码RNA（lincRNA）的抑制。

JNK-dependent Stat3 phosphorylation contributes to Akt activation in response to arsenic exposure.

砷暴露后，JNK 依赖的 Stat3 磷酸化促进 Akt 激活

Chronic occupational exposure to arsenic induces carcinogenic gene signaling networks and neoplastic transformation in human lung epithelial cells

长期职业性接触砷会诱导人肺上皮细胞产生致癌基因信号通路并发生肿瘤转化。

Carbon nanotubes induce malignant transformation and tumorigenesis of human lung epithelial cells

碳纳米管诱导人肺上皮细胞恶性转化和肿瘤发生