日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Molecular and functional characterization of GMP-manufactured neural stem cells and their extracellular vesicles for innovative therapeutic applications

对符合GMP标准的神经干细胞及其细胞外囊泡进行分子和功能表征,以用于创新治疗应用。

Guzzetti, Martina; Mezzasoma, Letizia; Chiasserini, Davide; Macchioni, Lara; Davidescu, Magdalena; di Michele, Alessandro; Gargaro, Marco; Di-Iacovo, Nicola; Manni, Giorgia; Muzi, Gianmarco; Proietti, Ilaria; Bevacqua, Giuseppina; Becattini, Eleonora; Conti, Carlo; Talesa, Vincenzo Nicola; Romani, Rita; Bellezza, Ilaria; Grespi, Valentina

Crosstalk between Long-Term Sublethal Oxidative Stress and Detrimental Inflammation as Potential Drivers for Age-Related Retinal Degeneration

长期亚致死性氧化应激与有害炎症之间的相互作用可能是年龄相关性视网膜变性的潜在驱动因素

Macchioni, Lara; Chiasserini, Davide; Mezzasoma, Letizia; Davidescu, Magdalena; Orvietani, Pier Luigi; Fettucciari, Katia; Salviati, Leonardo; Cellini, Barbara; Bellezza, Ilaria

Acetamidine-Based iNOS Inhibitors as Molecular Tools to Counteract Inflammation in BV2 Microglial Cells

基于乙酰胺的iNOS抑制剂作为分子工具对抗BV2小胶质细胞的炎症

Grottelli, Silvia; Amoroso, Rosa; Macchioni, Lara; D'Onofrio, Fiorella; Fettucciari, Katia; Bellezza, Ilaria; Maccallini, Cristina

The efficacy of the anticancer 3-bromopyruvate is potentiated by antimycin and menadione by unbalancing mitochondrial ROS production and disposal in U118 glioblastoma cells

抗癌药物3-溴丙酮酸的疗效可通过抗霉素和甲萘醌增强,其机制是通过扰乱U118胶质母细胞瘤细胞中线粒体活性氧(ROS)的产生和清除平衡。

Petricciuolo, Maya; Davidescu, Magdalena; Fettucciari, Katia; Gatticchi, Leonardo; Brancorsini, Stefano; Roberti, Rita; Corazzi, Lanfranco; Macchioni, Lara

Clostridium difficile-related postinfectious IBS: a case of enteroglial microbiological stalking and/or the solution of a conundrum?

艰难梭菌感染后肠易激综合征:肠神经胶质微生物跟踪的案例和/或难题的解决?

Bassotti, Gabrio; Macchioni, Lara; Corazzi, Lanfranco; Marconi, Pierfrancesco; Fettucciari, Katia

Enteric glial cells counteract Clostridium difficile Toxin B through a NADPH oxidase/ROS/JNK/caspase-3 axis, without involving mitochondrial pathways

肠道神经胶质细胞通过 NADPH 氧化酶/ROS/JNK/caspase-3 轴对抗艰难梭菌毒素 B,而不涉及线粒体途径。

Macchioni, Lara; Davidescu, Magdalena; Fettucciari, Katia; Petricciuolo, Maya; Gatticchi, Leonardo; Gioè, Davide; Villanacci, Vincenzo; Bellini, Massimo; Marconi, Pierfrancesco; Roberti, Rita; Bassotti, Gabrio; Corazzi, Lanfranco

Lethal digenic mutations in the K(+) channels Kir4.1 (KCNJ10) and SLACK (KCNT1) associated with severe-disabling seizures and neurodevelopmental delay.

K(+)通道Kir4.1 (KCNJ10)和SLACK (KCNT1)的致命双基因突变与严重的致残性癫痫和神经发育迟缓有关

Hasan Sonia, Balobaid Ameera, Grottesi Alessandro, Dabbagh Omar, Cenciarini Marta, Rawashdeh Rifaat, Al-Sagheir Afaf, Bove Cecilia, Macchioni Lara, Pessia Mauro, Al-Owain Mohammed, D'Adamo Maria Cristina