日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Structure-based identification of a new IAP-targeting compound that induces cancer cell death inducing NF-κB pathway

基于结构鉴定一种诱导癌细胞死亡诱导 NF-κB 通路的新型 IAP 靶向化合物

Federica Cossu, Simone Camelliti, Daniele Lecis, Luca Sorrentino, Maria Teresa Majorini, Mario Milani, Eloise Mastrangelo

Infiltrating Mast Cell-Mediated Stimulation of Estrogen Receptor Activity in Breast Cancer Cells Promotes the Luminal Phenotype

浸润性肥大细胞介导乳腺癌细胞中雌激素受体活性的刺激促进了管腔表型

Maria Teresa Majorini, Valeria Cancila, Alice Rigoni, Laura Botti, Matteo Dugo, Tiziana Triulzi, Loris De Cecco, Enrico Fontanella, Elena Jachetti, Elda Tagliabue, Claudia Chiodoni, Claudio Tripodo, Mario P Colombo #, Daniele Lecis #

Cancer bio-immunotherapy XVI annual NIBIT-(Italian Network for Tumor Biotherapy) meeting, October 11-13 2018, Milan, Italy

第十六届意大利肿瘤生物疗法网络(NIBIT)年会,癌症生物免疫疗法,2018年10月11-13日,意大利米兰

Bellone, Matteo; Brevi, Arianna; Bruzzì, Stefania; Consonni, Michela; De Santis, Francesca; Di Lullo, Giulia; Majorini, Maria Teresa; Pastò, Anna; Amadori, Alberto; Bregni, Marco; Di Nicola, Massimo; Calabrò, Luana; Ferrucci, Pier Francesco; Proietti, Enrico; Colombo, Mario Paolo; Russo, Vincenzo

MiR-16 regulates the pro-tumorigenic potential of lung fibroblasts through the inhibition of HGF production in an FGFR-1- and MEK1-dependent manner

MiR-16 以 FGFR-1 和 MEK1 依赖的方式抑制 HGF 产生,从而调节肺成纤维细胞的促肿瘤发生潜力

Francesca Andriani, Maria Teresa Majorini, Miguel Mano, Elena Landoni, Rosalba Miceli, Federica Facchinetti, Mavis Mensah, Enrico Fontanella, Matteo Dugo, Mauro Giacca, Ugo Pastorino, Gabriella Sozzi, Domenico Delia, Luca Roz, Daniele Lecis

cIAP1 regulates the EGFR/Snai2 axis in triple-negative breast cancer cells

cIAP1 调节三阴性乳腺癌细胞中的 EGFR/Snai2 轴

Maria Teresa Majorini, Giacomo Manenti, Miguel Mano, Loris De Cecco, Annalisa Conti, Patrizia Pinciroli, Enrico Fontanella, Elda Tagliabue, Claudia Chiodoni, Mario Paolo Colombo, Domenico Delia, Daniele Lecis

Oncogenic KRAS sensitizes premalignant, but not malignant cells, to Noxa-dependent apoptosis through the activation of the MEK/ERK pathway

致癌基因 KRAS 通过激活 MEK/ERK 通路,使癌前细胞(而非恶性细胞)对 Noxa 依赖性细胞凋亡敏感

Annalisa Conti, Maria Teresa Majorini, Richard Elliott, Alan Ashworth, Christopher J Lord, Carlotta Cancelliere, Alberto Bardelli, Pierfausto Seneci, Henning Walczak, Domenico Delia, Daniele Lecis