Makris Spyridon相关文献与解析，生知库 | 链接生命科学的每一步

Neutrophil recruitment and activation are differentially dependent on MyD88/TRIF and MAVS signaling during RSV infection.

RSV 感染期间，中性粒细胞的募集和活化对 MyD88/TRIF 和 MAVS 信号传导的依赖性不同

期刊:Mucosal Immunology

影响因子:7.6

doi:10.1038/s41385-019-0190-0

Kirsebom Freja C M, Kausar Fahima, Nuriev Rinat, Makris Spyridon, Johansson Cecilia

Recent advances in understanding rhinovirus immunity

鼻病毒免疫学研究的最新进展

期刊:F1000Research

影响因子:

doi:10.12688/f1000research.15337.1

Makris, Spyridon; Johnston, Sebastian

Alveolar Macrophages Can Control Respiratory Syncytial Virus Infection in the Absence of Type I Interferons

肺泡巨噬细胞在缺乏I型干扰素的情况下也能控制呼吸道合胞病毒感染。

期刊:Journal of Innate Immunity

影响因子:3

doi:10.1159/000446824

Makris, Spyridon; Bajorek, Monika; Culley, Fiona J; Goritzka, Michelle; Johansson, Cecilia

Impaired Antibody-mediated Protection and Defective IgA B-Cell Memory in Experimental Infection of Adults with Respiratory Syncytial Virus

呼吸道合胞病毒感染成人后，抗体介导的保护作用受损，IgA B细胞记忆缺陷

期刊:American Journal of Respiratory and Critical Care Medicine

影响因子:19.4

doi:10.1164/rccm.201412-2256OC

Habibi, Maximillian S; Jozwik, Agnieszka; Makris, Spyridon; Dunning, Jake; Paras, Allan; DeVincenzo, John P; de Haan, Cornelis A M; Wrammert, Jens; Openshaw, Peter J M; Chiu, Christopher

T cell responses are elicited against Respiratory Syncytial Virus in the absence of signalling through TLRs, RLRs and IL-1R/IL-18R

即使没有通过 TLR、RLR 和 IL-1R/IL-18R 信号传导，也能诱导针对呼吸道合胞病毒的 T 细胞反应。

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/srep18533

Goritzka, Michelle; Pereira, Catherine; Makris, Spyridon; Durant, Lydia R; Johansson, Cecilia

Protocol for isolating stromal cells from lymphoid tissue for performing scRNA-seq

用于从淋巴组织中分离基质细胞以进行单细胞RNA测序的方案

Protocol for isolating stromal cells from lymphoid tissue for performing scRNA-seq

用于从淋巴组织中分离基质细胞以进行单细胞RNA测序的方案

Immune function and dysfunction are determined by lymphoid tissue efficacy

免疫功能及其障碍取决于淋巴组织的效能。

Neutrophil recruitment and activation are differentially dependent on MyD88/TRIF and MAVS signaling during RSV infection.

RSV 感染期间，中性粒细胞的募集和活化对 MyD88/TRIF 和 MAVS 信号传导的依赖性不同

Recent advances in understanding rhinovirus immunity

鼻病毒免疫学研究的最新进展

Alveolar Macrophages Can Control Respiratory Syncytial Virus Infection in the Absence of Type I Interferons

肺泡巨噬细胞在缺乏I型干扰素的情况下也能控制呼吸道合胞病毒感染。

Impaired Antibody-mediated Protection and Defective IgA B-Cell Memory in Experimental Infection of Adults with Respiratory Syncytial Virus

呼吸道合胞病毒感染成人后，抗体介导的保护作用受损，IgA B细胞记忆缺陷

T cell responses are elicited against Respiratory Syncytial Virus in the absence of signalling through TLRs, RLRs and IL-1R/IL-18R

即使没有通过 TLR、RLR 和 IL-1R/IL-18R 信号传导，也能诱导针对呼吸道合胞病毒的 T 细胞反应。