Manfred Beier相关文献与解析，生知库 | 链接生命科学的每一步

Dario Van Meenen, Annika Doege, Emily Alefeld, André Haase, Manfred Beier, Tobias Kiefer, Eva Biewald, Klaus Metz, Oliver Dräger, Maike Anna Busch, Nicole Dünker

肿瘤

其它细胞

发表日期：2022

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TFF1 in Aqueous Humor-A Potential New Biomarker for Retinoblastoma

房水中的 TFF1——视网膜母细胞瘤的潜在新生物标志物

期刊:Cancers

影响因子:

doi:10.3390/cancers14030677

Maike Anna Busch, André Haase, Natalia Miroschnikov, Annika Doege, Eva Biewald, Nikolaos E Bechrakis, Manfred Beier, Deniz Kanber, Dietmar Lohmann, Klaus Metz, Nicole Dünker

Comprehensive Biology and Genetics Compendium of Wilms Tumor Cell Lines with Different WT1 Mutations

具有不同 WT1 突变的 Wilms 肿瘤细胞系的综合生物学和遗传学概要

期刊:Cancers

影响因子:

doi:10.3390/cancers13010060

Brigitte Royer-Pokora, Maike Anna Busch, Sarah Tenbusch, Mathias Schmidt, Manfred Beier, Andrew D Woods, Holger Thiele, Jaume Mora

Distinctive mutational spectrum and karyotype disruption in long-term cisplatin-treated urothelial carcinoma cell lines

长期顺铂治疗的尿路上皮癌细胞系中独特的突变谱和核型破坏

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-019-50891-w

Margaretha A Skowron, Patrick Petzsch, Karin Hardt, Nicholas Wagner, Manfred Beier, Stefanie Stepanow, Matthias Drechsler, Harald Rieder, Karl Köhrer, Günter Niegisch, Michèle J Hoffmann, Wolfgang A Schulz

Reduction of the tumorigenic potential of human retinoblastoma cell lines by TFF1 overexpression involves p53/caspase signaling and miR-18a regulation

TFF1 过表达降低人类视网膜母细胞瘤细胞系的致瘤潜力涉及 p53/caspase 信号传导和 miR-18a 调控

期刊:International Journal of Cancer

影响因子:5.7

doi:10.1002/ijc.30768

Maike Busch, Jan Große-Kreul, Janina Jasmin Wirtz, Manfred Beier, Harald Stephan, Brigitte Royer-Pokora, Klaus Metz, Nicole Dünker

Establishment of a Conditionally Immortalized Wilms Tumor Cell Line with a Homozygous WT1 Deletion within a Heterozygous 11p13 Deletion and UPD Limited to 11p15

建立具有纯合 WT1 缺失、杂合 11p13 缺失和 UPD 限制为 11p15 的条件性永生化 Wilms 肿瘤细胞系

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0155561

Artur Brandt, Katharina Löhers, Manfred Beier, Barbara Leube, Carmen de Torres, Jaume Mora, Parineeta Arora, Parmjit S Jat, Brigitte Royer-Pokora

Classification of a frameshift/extended and a stop mutation in WT1 as gain-of-function mutations that activate cell cycle genes and promote Wilms tumour cell proliferation

将 WT1 中的移码/扩展突变和终止突变归类为获得功能突变，可激活细胞周期基因并促进 Wilms 肿瘤细胞增殖

期刊:Human Molecular Genetics

影响因子:3.1

doi:10.1093/hmg/ddu111

Maike Busch, Heinrich Schwindt, Artur Brandt, Manfred Beier, Nicole Görldt, Paul Romaniuk, Eneda Toska, Stefan Roberts, Hans-Dieter Royer, Brigitte Royer-Pokora

Wilms tumor cells with WT1 mutations have characteristic features of mesenchymal stem cells and express molecular markers of paraxial mesoderm

WT1 突变的 Wilms 肿瘤细胞具有间充质干细胞的特征，并表达近轴中胚层的分子标记

期刊:Human Molecular Genetics

影响因子:3.1

doi:10.1093/hmg/ddq042

Brigitte Royer-Pokora, Maike Busch, Manfred Beier, Constanze Duhme, Carmen de Torres, Jaume Mora, Artur Brandt, Hans-Dieter Royer

Hyperactivation of the insulin-like growth factor receptor I signaling pathway is an essential event for cisplatin resistance of ovarian cancer cells

胰岛素样生长因子受体 I 信号通路过度激活是卵巢癌细胞顺铂耐药的重要事件

期刊:Cancer Research

影响因子:12.5

doi:10.1158/0008-5472.CAN-08-3153

Niels Eckstein, Kati Servan, Barbara Hildebrandt, Anne Pölitz, Georg von Jonquières, Sybille Wolf-Kümmeth, Inge Napierski, Alexandra Hamacher, Matthias U Kassack, Jan Budczies, Manfred Beier, Manfred Dietel, Brigitte Royer-Pokora, Carsten Denkert, Hans-Dieter Royer

ADAM10 and ADAM17-Novel Players in Retinoblastoma Carcinogenesis

ADAM10 和 ADAM17——视网膜母细胞瘤致癌作用中的新参与者

TFF1 in Aqueous Humor-A Potential New Biomarker for Retinoblastoma

房水中的 TFF1——视网膜母细胞瘤的潜在新生物标志物

Comprehensive Biology and Genetics Compendium of Wilms Tumor Cell Lines with Different WT1 Mutations

具有不同 WT1 突变的 Wilms 肿瘤细胞系的综合生物学和遗传学概要

Distinctive mutational spectrum and karyotype disruption in long-term cisplatin-treated urothelial carcinoma cell lines

长期顺铂治疗的尿路上皮癌细胞系中独特的突变谱和核型破坏

Reduction of the tumorigenic potential of human retinoblastoma cell lines by TFF1 overexpression involves p53/caspase signaling and miR-18a regulation

TFF1 过表达降低人类视网膜母细胞瘤细胞系的致瘤潜力涉及 p53/caspase 信号传导和 miR-18a 调控

Establishment of a Conditionally Immortalized Wilms Tumor Cell Line with a Homozygous WT1 Deletion within a Heterozygous 11p13 Deletion and UPD Limited to 11p15

建立具有纯合 WT1 缺失、杂合 11p13 缺失和 UPD 限制为 11p15 的条件性永生化 Wilms 肿瘤细胞系

Classification of a frameshift/extended and a stop mutation in WT1 as gain-of-function mutations that activate cell cycle genes and promote Wilms tumour cell proliferation

将 WT1 中的移码/扩展突变和终止突变归类为获得功能突变，可激活细胞周期基因并促进 Wilms 肿瘤细胞增殖

Wilms tumor cells with WT1 mutations have characteristic features of mesenchymal stem cells and express molecular markers of paraxial mesoderm

WT1 突变的 Wilms 肿瘤细胞具有间充质干细胞的特征，并表达近轴中胚层的分子标记

Hyperactivation of the insulin-like growth factor receptor I signaling pathway is an essential event for cisplatin resistance of ovarian cancer cells

胰岛素样生长因子受体 I 信号通路过度激活是卵巢癌细胞顺铂耐药的重要事件