Manolson Morris F相关文献与解析，生知库

New Mechanisms for Regulation of Intracellular Collagen Degradation in Fibrotic Lesions of Periodontal Tissues

牙周组织纤维化病变中细胞内胶原降解调控的新机制

期刊:FASEB Journal

影响因子:4.2

doi:10.1096/fj.202502454R

Wong, Wing Hei; Zirngibl, Ralph A; Manolson, Morris F; McCulloch, Christopher A

Characterization of a PIP Binding Site in the N-Terminal Domain of V-ATPase a4 and Its Role in Plasma Membrane Association

V-ATPase a4 N端结构域中PIP结合位点的特征及其在质膜结合中的作用

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms24054867

Chu, Anh; Yao, Yeqi; Saffi, Golam T; Chung, Ji Hyun; Botelho, Roberto J; Glibowicka, Miroslawa; Deber, Charles M; Manolson, Morris F

发表日期：2023

文献求助收藏

The V-ATPase a3 Subunit: Structure, Function and Therapeutic Potential of an Essential Biomolecule in Osteoclastic Bone Resorption

V-ATPase a3 亚基：破骨细胞骨吸收中一种重要生物分子的结构、功能和治疗潜力

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms22136934

Chu, Anh; Zirngibl, Ralph A; Manolson, Morris F

细胞生物学

发表日期：2021

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Osteopetrosis mutation R444L causes endoplasmic reticulum retention and misprocessing of vacuolar H+-ATPase a3 subunit.

骨硬化症突变 R444L 导致内质网滞留和液泡 H+-ATPase a3 亚基的错误加工

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M112.345702

Bhargava Ajay, Voronov Irina, Wang Yongqiang, Glogauer Michael, Kartner Norbert, Manolson Morris F

骨科研究

发表日期：2012

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Regulation of vacuolar H(+)-ATPase in microglia by RANKL

RANKL对小胶质细胞液泡H(+)-ATPase的调控

期刊:Biochemical and Biophysical Research Communications

影响因子:2.2

doi:10.1016/j.bbrc.2009.08.122

Serrano, Eric M; Ricofort, Ryan D; Zuo, Jian; Ochotny, Noelle; Manolson, Morris F; Holliday, L Shannon

Evidence for Rab7b and Its Splice Isoforms Having Distinct Biological Functions from Rab7a

证据表明 Rab7b 及其剪接异构体与 Rab7a 具有不同的生物学功能

New Mechanisms for Regulation of Intracellular Collagen Degradation in Fibrotic Lesions of Periodontal Tissues

牙周组织纤维化病变中细胞内胶原降解调控的新机制

Characterization of a PIP Binding Site in the N-Terminal Domain of V-ATPase a4 and Its Role in Plasma Membrane Association

V-ATPase a4 N端结构域中PIP结合位点的特征及其在质膜结合中的作用

The V-ATPase a3 Subunit: Structure, Function and Therapeutic Potential of an Essential Biomolecule in Osteoclastic Bone Resorption

V-ATPase a3 亚基：破骨细胞骨吸收中一种重要生物分子的结构、功能和治疗潜力

Osteopetrosis mutation R444L causes endoplasmic reticulum retention and misprocessing of vacuolar H+-ATPase a3 subunit.

骨硬化症突变 R444L 导致内质网滞留和液泡 H+-ATPase a3 亚基的错误加工

Regulation of vacuolar H(+)-ATPase in microglia by RANKL

RANKL对小胶质细胞液泡H(+)-ATPase的调控