Marcet Marcus M相关文献与解析，生知库 | 链接生命科学的每一步

Delayed administration of bone marrow mesenchymal stem cell conditioned medium significantly improves outcome after retinal ischemia in rats.

延迟给予骨髓间充质干细胞条件培养基可显著改善大鼠视网膜缺血后的预后

期刊:Investigative Ophthalmology & Visual Science

Dreixler John C, Poston Jacqueline N, Balyasnikova Irina, Shaikh Afzhal R, Tupper Kelsey Y, Conway Sineadh, Boddapati Venkat, Marcet Marcus M, Lesniak Maciej S, Roth Steven

Full-thickness eyelid lesions in sarcoidosis

结节病引起的全层眼睑病变

期刊:Case Reports in Ophthalmological Medicine

影响因子:0.4

doi:10.1155/2013/579121

Collins, Megan E; Petronic-Rosic, Vesna; Sweiss, Nadera J; Marcet, Marcus M

发表日期：2013

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Delayed post-ischemic conditioning significantly improves the outcome after retinal ischemia

延迟缺血后适应可显著改善视网膜缺血后的预后

期刊:Experimental Eye Research

影响因子:2.7

doi:10.1016/j.exer.2011.03.015

Dreixler, John C; Poston, Jacqueline N; Shaikh, Afzhal R; Alexander, Michael; Tupper, Kelsey Y; Marcet, Marcus M; Bernaudin, Myriam; Roth, Steven

发表日期：2011

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Mitogen-activated protein kinase phosphatase-1 (MKP-1) in retinal ischemic preconditioning.

视网膜缺血预适应中的丝裂原活化蛋白激酶磷酸酶-1 (MKP-1)

期刊:Experimental Eye Research

影响因子:2.7

doi:10.1016/j.exer.2010.10.011

Dreixler John C, Bratton Anthony, Du Eugenie, Shaikh Afzhal R, Savoie Brian, Alexander Michael, Marcet Marcus M, Roth Steven

免疫/内分泌

发表日期：2011

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Protein kinase B (Akt) and mitogen-activated protein kinase p38Î± in retinal ischemic post-conditioning.

视网膜缺血后处理中的蛋白激酶 B (Akt) 和丝裂原活化蛋白激酶 p38α

期刊:Journal of Molecular Neuroscience

影响因子:2.7

doi:10.1007/s12031-011-9523-5

Dreixler John C, Sampat Ajay, Shaikh Afzhal R, Alexander Michael, Marcet Marcus M, Roth Steven

Antimetabolites as an adjunct to dacryocystorhinostomy for nasolacrimal duct obstruction

抗代谢药物作为鼻泪管阻塞泪囊鼻腔吻合术的辅助治疗

Delayed administration of bone marrow mesenchymal stem cell conditioned medium significantly improves outcome after retinal ischemia in rats.

延迟给予骨髓间充质干细胞条件培养基可显著改善大鼠视网膜缺血后的预后

Full-thickness eyelid lesions in sarcoidosis

结节病引起的全层眼睑病变

Delayed post-ischemic conditioning significantly improves the outcome after retinal ischemia

延迟缺血后适应可显著改善视网膜缺血后的预后

Mitogen-activated protein kinase phosphatase-1 (MKP-1) in retinal ischemic preconditioning.

视网膜缺血预适应中的丝裂原活化蛋白激酶磷酸酶-1 (MKP-1)

Protein kinase B (Akt) and mitogen-activated protein kinase p38Î± in retinal ischemic post-conditioning.

视网膜缺血后处理中的蛋白激酶 B (Akt) 和丝裂原活化蛋白激酶 p38α