While systolic cardiomyocyte function is preserved, diastolic myocyte function and recovery from acidosis are impaired in CaMKIIδ-KO mice

CaMKIIδ-KO 小鼠的收缩性心肌细胞功能得以保留，但舒张性心肌细胞功能和酸中毒恢复受到损害