Maria-Pia Rastaldi相关文献与解析，生知库

Stefan Zschiedrich, Tillmann Bork, Wei Liang, Nicola Wanner, Kristina Eulenbruch, Stefan Munder, Björn Hartleben, Oliver Kretz, Simon Gerber, Matias Simons, Amandine Viau, Martine Burtin, Changli Wei, Jochen Reiser, Nadja Herbach, Maria-Pia Rastaldi, Clemens D Cohen, Pierre-Louis Tharaux, Fabiola Te

信号转导

mTOR

发表日期：2017

文献求助收藏

Early involvement of cellular stress and inflammatory signals in the pathogenesis of tubulointerstitial kidney disease due to UMOD mutations

细胞应激和炎症信号在 UMOD 突变导致的肾小管间质性肾病发病机制中的早期作用

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-017-07804-6

Matteo Trudu, Celine Schaeffer, Michela Riba, Masami Ikehata, Paola Brambilla, Piergiorgio Messa, Filippo Martinelli-Boneschi, Maria Pia Rastaldi, Luca Rampoldi

信号转导

细胞生物学

其它细胞

Immunology/Inflammation

发表日期：2017

文献求助收藏

A nanoporous surface is essential for glomerular podocyte differentiation in three-dimensional culture

纳米多孔表面对于三维培养中的肾小球足细胞分化至关重要

期刊:International Journal of Nanomedicine

影响因子:6.6

doi:10.2147/IJN.S110201

Cristina Zennaro, Maria Pia Rastaldi, Gerald James Bakeine, Riccarda Delfino, Federica Tonon, Rossella Farra, Gabriele Grassi, Mary Artero, Massimo Tormen, Michele Carraro

LPS nephropathy in mice is ameliorated by IL-2 independently of regulatory T cells activity

IL-2 可独立于调节性 T 细胞活性改善小鼠的 LPS 肾病

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0111285

Roberta Bertelli, Armando Di Donato, Michela Cioni, Fabio Grassi, Masami Ikehata, Alice Bonanni, Maria Pia Rastaldi, Gian Marco Ghiggeri

Defining cell-type specificity at the transcriptional level in human disease

在人类疾病的转录水平上定义细胞类型特异性

期刊:Genome Research

影响因子:6.2

doi:10.1101/gr.155697.113

Wenjun Ju, Casey S Greene, Felix Eichinger, Viji Nair, Jeffrey B Hodgin, Markus Bitzer, Young-Suk Lee, Qian Zhu, Masami Kehata, Min Li, Song Jiang, Maria Pia Rastaldi, Clemens D Cohen, Olga G Troyanskaya, Matthias Kretzler

Podocyte expression of membrane transporters involved in puromycin aminonucleoside-mediated injury

参与嘌呤霉素氨基核苷介导损伤的足细胞膜转运蛋白表达

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0066159

Cristina Zennaro, Maria Pia Rastaldi, Lorella Pascolo, Marco Stebel, Elisa Trevisan, Mary Artero, Claudio Tiribelli, Vittorio Di Maso, Michele Carraro

Urinary exosomal microRNAs in incipient diabetic nephropathy

早期糖尿病肾病中的尿液外泌体微小RNA

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0073798

Federica Barutta, Marinella Tricarico, Alessandro Corbelli, Laura Annaratone, Silvia Pinach, Serena Grimaldi, Graziella Bruno, Daniela Cimino, Daniela Taverna, Maria Chiara Deregibus, Maria Pia Rastaldi, Paolo Cavallo Perin, Gabriella Gruden

Compensatory molecular and functional mechanisms in nervous system of the Grm1(crv4) mouse lacking the mGlu1 receptor: a model for motor coordination deficits

缺乏 mGlu1 受体的 Grm1(crv4) 小鼠神经系统的补偿分子和功能机制：运动协调缺陷的模型

期刊:Cerebral Cortex

影响因子:2.9

doi:10.1093/cercor/bhs200

Pia Irene Anna Rossi, Ilaria Musante, Maria Summa, Anna Pittaluga, Laura Emionite, Masami Ikehata, Maria Pia Rastaldi, Roberto Ravazzolo, Aldamaria Puliti

Crk1/2-dependent signaling is necessary for podocyte foot process spreading in mouse models of glomerular disease

在肾小球疾病小鼠模型中，Crk1/2 依赖性信号传导是足细胞足突扩散所必需的

期刊:Journal of Clinical Investigation

影响因子:13.3

doi:10.1172/JCI60070

Britta George, Rakesh Verma, Abdulsalam A Soofi, Puneet Garg, Jidong Zhang, Tae-Ju Park, Laura Giardino, Larisa Ryzhova, Duncan B Johnstone, Hetty Wong, Deepak Nihalani, David J Salant, Steven K Hanks, Tom Curran, Maria Pia Rastaldi, Lawrence B Holzman

Role of mTOR in podocyte function and diabetic nephropathy in humans and mice

mTOR 在人类和小鼠足细胞功能和糖尿病肾病中的作用

期刊:Journal of Clinical Investigation

影响因子:13.3

doi:10.1172/JCI44774

Markus Gödel, Björn Hartleben, Nadja Herbach, Shuya Liu, Stefan Zschiedrich, Shun Lu, Andrea Debreczeni-Mór, Maja T Lindenmeyer, Maria-Pia Rastaldi, Götz Hartleben, Thorsten Wiech, Alessia Fornoni, Robert G Nelson, Matthias Kretzler, Rüdiger Wanke, Hermann Pavenstädt, Dontscho Kerjaschki, Clemens D

Targeting mTOR Signaling Can Prevent the Progression of FSGS

靶向 mTOR 信号传导可阻止 FSGS 进展

Early involvement of cellular stress and inflammatory signals in the pathogenesis of tubulointerstitial kidney disease due to UMOD mutations

细胞应激和炎症信号在 UMOD 突变导致的肾小管间质性肾病发病机制中的早期作用

A nanoporous surface is essential for glomerular podocyte differentiation in three-dimensional culture

纳米多孔表面对于三维培养中的肾小球足细胞分化至关重要

LPS nephropathy in mice is ameliorated by IL-2 independently of regulatory T cells activity

IL-2 可独立于调节性 T 细胞活性改善小鼠的 LPS 肾病

Defining cell-type specificity at the transcriptional level in human disease

在人类疾病的转录水平上定义细胞类型特异性

Podocyte expression of membrane transporters involved in puromycin aminonucleoside-mediated injury

参与嘌呤霉素氨基核苷介导损伤的足细胞膜转运蛋白表达

Urinary exosomal microRNAs in incipient diabetic nephropathy

早期糖尿病肾病中的尿液外泌体微小RNA

Compensatory molecular and functional mechanisms in nervous system of the Grm1(crv4) mouse lacking the mGlu1 receptor: a model for motor coordination deficits

缺乏 mGlu1 受体的 Grm1(crv4) 小鼠神经系统的补偿分子和功能机制：运动协调缺陷的模型

Crk1/2-dependent signaling is necessary for podocyte foot process spreading in mouse models of glomerular disease

在肾小球疾病小鼠模型中，Crk1/2 依赖性信号传导是足细胞足突扩散所必需的

Role of mTOR in podocyte function and diabetic nephropathy in humans and mice

mTOR 在人类和小鼠足细胞功能和糖尿病肾病中的作用