Markus Christmann相关文献与解析，生知库

Al-Hassan M Mustafa ，Giuseppe Petrosino ，Marten A Fischer ，Tina M Schnöder ，Désirée Gül ，Yanira Zeyn ，Christoph Hieber ，Johanna Lossa ，Sabine Muth ，Markus P Radsak ，Walburgis Brenner ，Markus Christmann ，Matthias Bros ，Florian H Heidel ，Oliver H Krämer

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HDAC1

The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence

p21CIP1-CDK4-DREAM 轴是基因毒性应激诱导细胞衰老的主要调节器

期刊:Nucleic Acids Research

影响因子:16.6

doi:10.1093/nar/gkae426

Ariane Schmidt, Sebastian Allmann, Christian Schwarzenbach, Petra Snyder, Jia-Xuan Chen, Georg Nagel, Anna Schöneis, Birgit Rasenberger, Petra Beli, Alexander Loewer, Thomas G Hofmann, Maja T Tomicic, Markus Christmann

Genotoxic and Cytotoxic Activity of Fisetin on Glioblastoma Cells

漆黄素对胶质母细胞瘤细胞的基因毒性和细胞毒活性

期刊:Anticancer Research

影响因子:1.6

doi:10.21873/anticanres.16884

Lea Beltzig, Markus Christmann, Minodora Dobreanu, Bernd Kaina

p53 triggers mitochondrial apoptosis following DNA damage-dependent replication stress by the hepatotoxin methyleugenol

p53 在肝毒素甲基丁香酚造成 DNA 损伤依赖性复制压力后引发线粒体凋亡

期刊:Cell Death & Disease

影响因子:8.1

doi:10.1038/s41419-022-05446-9

Max J Carlsson #, Anastasia S Vollmer #, Philipp Demuth, Daniel Heylmann, Diana Reich, Caroline Quarz, Birgit Rasenberger, Teodora Nikolova, Thomas G Hofmann, Markus Christmann, Julia A Fuhlbrueck, Simone Stegmüller, Elke Richling, Alexander T Cartus, Jörg Fahrer

Abrogation of Cellular Senescence Induced by Temozolomide in Glioblastoma Cells: Search for Senolytics

消除替莫唑胺诱导的胶质母细胞瘤细胞衰老：寻找抗衰老药物

期刊:Cells

影响因子:5.1

doi:10.3390/cells11162588

Lea Beltzig, Markus Christmann, Bernd Kaina

Senescence Is the Main Trait Induced by Temozolomide in Glioblastoma Cells

衰老是替莫唑胺诱导胶质母细胞瘤细胞的主要特征

期刊:Cancers

影响因子:

doi:10.3390/cancers14092233

Lea Beltzig, Christian Schwarzenbach, Petra Leukel, Katrin B M Frauenknecht, Clemens Sommer, Alessandro Tancredi, Monika E Hegi, Markus Christmann, Bernd Kaina

Localization matters: nuclear-trapped Survivin sensitizes glioblastoma cells to temozolomide by elevating cellular senescence and impairing homologous recombination

定位很重要：核捕获的 Survivin 通过加速细胞衰老和损害同源重组使胶质母细胞瘤细胞对替莫唑胺敏感

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-021-03864-0

Thomas R Reich #, Christian Schwarzenbach, Juliana Brandstetter Vilar, Sven Unger, Fabian Mühlhäusler, Teodora Nikolova, Alicia Poplawski, H Irem Baymaz, Petra Beli, Markus Christmann, Maja T Tomicic #

Oxaliplatin-Induced Senescence in Colorectal Cancer Cells Depends on p14ARF-Mediated Sustained p53 Activation

奥沙利铂诱导的结肠直肠癌细胞衰老依赖于 p14ARF 介导的持续 p53 激活

期刊:Cancers

影响因子:

doi:10.3390/cancers13092019

Maja T Tomicic, Franziska Krämer, Alexandra Nguyen, Christian Schwarzenbach, Markus Christmann

Targeting c-IAP1, c-IAP2, and Bcl-2 Eliminates Senescent Glioblastoma Cells Following Temozolomide Treatment

以 c-IAP1、c-IAP2 和 Bcl-2 为靶点，在替莫唑胺治疗后消除衰老的胶质母细胞瘤细胞

期刊:Cancers

影响因子:

doi:10.3390/cancers13143585

Christian Schwarzenbach, Larissa Tatsch, Juliana Brandstetter Vilar, Birgit Rasenberger, Lea Beltzig, Bernd Kaina, Maja T Tomicic, Markus Christmann

Natural Merosesquiterpenes Activate the DNA Damage Response via DNA Strand Break Formation and Trigger Apoptotic Cell Death in p53-Wild-type and Mutant Colorectal Cancer

天然半萜烯通过 DNA 链断裂形成激活 DNA 损伤反应并引发 p53 野生型和突变型结直肠癌的细胞凋亡

期刊:Cancers

影响因子:

doi:10.3390/cancers13133282

Apisada Jiso, Philipp Demuth, Madeleine Bachowsky, Manuel Haas, Nina Seiwert, Daniel Heylmann, Birgit Rasenberger, Markus Christmann, Lea Dietrich, Thomas Brunner, Riyanti, Till F Schäberle, Anuchit Plubrukarn, Jörg Fahrer

The deacetylases HDAC1/HDAC2 control JAK2V617F-STAT signaling through the ubiquitin ligase SIAH2

去乙酰化酶HDAC1/HDAC2通过泛素连接酶SIAH2调控JAK2V617F-STAT信号通路。

The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence

p21CIP1-CDK4-DREAM 轴是基因毒性应激诱导细胞衰老的主要调节器

Genotoxic and Cytotoxic Activity of Fisetin on Glioblastoma Cells

漆黄素对胶质母细胞瘤细胞的基因毒性和细胞毒活性

p53 triggers mitochondrial apoptosis following DNA damage-dependent replication stress by the hepatotoxin methyleugenol

p53 在肝毒素甲基丁香酚造成 DNA 损伤依赖性复制压力后引发线粒体凋亡

Abrogation of Cellular Senescence Induced by Temozolomide in Glioblastoma Cells: Search for Senolytics

消除替莫唑胺诱导的胶质母细胞瘤细胞衰老：寻找抗衰老药物

Senescence Is the Main Trait Induced by Temozolomide in Glioblastoma Cells

衰老是替莫唑胺诱导胶质母细胞瘤细胞的主要特征

Localization matters: nuclear-trapped Survivin sensitizes glioblastoma cells to temozolomide by elevating cellular senescence and impairing homologous recombination

定位很重要：核捕获的 Survivin 通过加速细胞衰老和损害同源重组使胶质母细胞瘤细胞对替莫唑胺敏感

Oxaliplatin-Induced Senescence in Colorectal Cancer Cells Depends on p14ARF-Mediated Sustained p53 Activation

奥沙利铂诱导的结肠直肠癌细胞衰老依赖于 p14ARF 介导的持续 p53 激活

Targeting c-IAP1, c-IAP2, and Bcl-2 Eliminates Senescent Glioblastoma Cells Following Temozolomide Treatment

以 c-IAP1、c-IAP2 和 Bcl-2 为靶点，在替莫唑胺治疗后消除衰老的胶质母细胞瘤细胞

Natural Merosesquiterpenes Activate the DNA Damage Response via DNA Strand Break Formation and Trigger Apoptotic Cell Death in p53-Wild-type and Mutant Colorectal Cancer

天然半萜烯通过 DNA 链断裂形成激活 DNA 损伤反应并引发 p53 野生型和突变型结直肠癌的细胞凋亡