Masatake Fujimura相关文献与解析，生知库

Environmental stresses suppress nonsense-mediated mRNA decay (NMD) and affect cells by stabilizing NMD-targeted gene expression

环境压力抑制无义介导的 mRNA 衰变 (NMD)，并通过稳定 NMD 靶基因表达来影响细胞

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-018-38015-2

Fusako Usuki, Akio Yamashita, Masatake Fujimura

The selective GSK3 inhibitor, SAR502250, displays neuroprotective activity and attenuates behavioral impairments in models of neuropsychiatric symptoms of Alzheimer's disease in rodents

选择性 GSK3 抑制剂 SAR502250 表现出神经保护活性，并减轻啮齿动物阿尔茨海默病神经精神症状模型中的行为障碍

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-019-54557-5

Guy Griebel, Jeanne Stemmelin, Mati Lopez-Grancha, Denis Boulay, Gerald Boquet, Franck Slowinski, Philippe Pichat, Sandra Beeské, Shinji Tanaka, Akiko Mori, Masatake Fujimura, Junichi Eguchi

神经

发表日期：2019

文献求助收藏

Chemokine CCL4 Induced in Mouse Brain Has a Protective Role against Methylmercury Toxicity

小鼠脑中诱导的趋化因子 CCL4 对甲基汞毒性具有保护作用

期刊:Toxics

影响因子:3.9

doi:10.3390/toxics6030036

Tsutomu Takahashi, Min-Seok Kim, Miyuki Iwai-Shimada, Masatake Fujimura, Takashi Toyama, Akira Naganuma, Gi-Wook Hwang

Endoplasmic reticulum stress preconditioning modifies intracellular mercury content by upregulating membrane transporters

内质网应激预处理通过上调膜转运蛋白改变细胞内汞含量

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-017-09435-3

Fusako Usuki, Masatake Fujimura, Akio Yamashita

Methylmercury induces the expression of TNF-α selectively in the brain of mice

甲基汞选择性诱导小鼠脑组织TNF-α表达

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/srep38294

Miyuki Iwai-Shimada, Tsutomu Takahashi, Min-Seok Kim, Masatake Fujimura, Hitoyasu Ito, Takashi Toyama, Akira Naganuma, Gi-Wook Hwang

信号转导

发表日期：2016

文献求助收藏

Endoplasmic reticulum stress preconditioning attenuates methylmercury-induced cellular damage by inducing favorable stress responses

内质网应激预处理通过诱导有利的应激反应减轻甲基汞诱导的细胞损伤

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/srep02346

Fusako Usuki, Masatake Fujimura, Akio Yamashita

Post-transcriptional defects of antioxidant selenoenzymes cause oxidative stress under methylmercury exposure

抗氧化硒酶的转录后缺陷导致甲基汞暴露下的氧化应激

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1074/jbc.M110.168872

Fusako Usuki, Akio Yamashita, Masatake Fujimura

表观遗传

发表日期：2011

文献求助收藏

Characterization of pathological changes in the olfactory system of mice exposed to methylmercury

甲基汞暴露小鼠嗅觉系统病理变化的特征

Development of a sensor to detect methylmercury toxicity

检测甲基汞毒性的传感器的开发

Alterations in UPR Signaling by Methylmercury Trigger Neuronal Cell Death in the Mouse Brain

甲基汞引起 UPR 信号改变，引发小鼠脑神经细胞死亡

Environmental stresses suppress nonsense-mediated mRNA decay (NMD) and affect cells by stabilizing NMD-targeted gene expression

环境压力抑制无义介导的 mRNA 衰变 (NMD)，并通过稳定 NMD 靶基因表达来影响细胞

The selective GSK3 inhibitor, SAR502250, displays neuroprotective activity and attenuates behavioral impairments in models of neuropsychiatric symptoms of Alzheimer's disease in rodents

选择性 GSK3 抑制剂 SAR502250 表现出神经保护活性，并减轻啮齿动物阿尔茨海默病神经精神症状模型中的行为障碍

Chemokine CCL4 Induced in Mouse Brain Has a Protective Role against Methylmercury Toxicity

小鼠脑中诱导的趋化因子 CCL4 对甲基汞毒性具有保护作用

Endoplasmic reticulum stress preconditioning modifies intracellular mercury content by upregulating membrane transporters

内质网应激预处理通过上调膜转运蛋白改变细胞内汞含量

Methylmercury induces the expression of TNF-α selectively in the brain of mice

甲基汞选择性诱导小鼠脑组织TNF-α表达

Endoplasmic reticulum stress preconditioning attenuates methylmercury-induced cellular damage by inducing favorable stress responses

内质网应激预处理通过诱导有利的应激反应减轻甲基汞诱导的细胞损伤

Post-transcriptional defects of antioxidant selenoenzymes cause oxidative stress under methylmercury exposure

抗氧化硒酶的转录后缺陷导致甲基汞暴露下的氧化应激