Mathéa相关文献与解析，生知库 | 链接生命科学的每一步

Titanium dioxide and carbon black nanoparticles disrupt neuronal homeostasis via excessive activation of cellular prion protein signaling

二氧化钛和炭黑纳米颗粒通过过度激活细胞朊病毒蛋白信号破坏神经元稳态

Luiz W Ribeiro, Mathéa Pietri, Hector Ardila-Osorio, Anne Baudry, François Boudet-Devaud, Chloé Bizingre, Zaira E Arellano-Anaya, Anne-Marie Haeberlé, Nicolas Gadot, Sonja Boland, Stéphanie Devineau, Yannick Bailly, Odile Kellermann, Anna Bencsik, Benoit Schneider

信号转导

Loss of prion protein control of glucose metabolism promotes neurodegeneration in model of prion diseases

朊病毒蛋白对葡萄糖代谢的控制丧失会促进朊病毒疾病模型中的神经退行性变。

期刊:PLoS Pathogens

影响因子:5.5

doi:10.1371/journal.ppat.1009991.

Hélène Arnould,Vincent Baudouin,Anne Baudry,Luiz W Ribeiro,Hector Ardila-Osorio,Mathéa Pietri,Cédric Caradeuc,Cynthia Soultawi,Declan Williams,Marjorie Alvarez,Carole Crozet,Fatima Djouadi,Mireille Laforge,Gildas Bertho,Odile Kellermann,Jean-Marie Launay,Gerold Schmitt-Ulms,Benoit Schneider

The Cellular Prion Protein-ROCK Connection: Contribution to Neuronal Homeostasis and Neurodegenerative Diseases

细胞朊蛋白与ROCK蛋白的关联：对神经元稳态和神经退行性疾病的影响

期刊:Frontiers in Cellular Neuroscience

影响因子:4

doi:10.3389/fncel.2021.660683

Schneider, Benoit; Baudry, Anne; Pietri, Mathéa; Alleaume-Butaux, Aurélie; Bizingre, Chloé; Nioche, Pierre; Kellermann, Odile; Launay, Jean-Marie

Production of seedable Amyloid-β peptides in model of prion diseases upon PrPSc-induced PDK1 overactivation

PrPSc 诱导 PDK1 过度激活后，朊病毒疾病模型中可产生可播种的淀粉样β肽

期刊:Nature Communications

影响因子:14.7

doi:10.1038/s41467-019-11333-3

Juliette Ezpeleta, Vincent Baudouin, Zaira E Arellano-Anaya, François Boudet-Devaud, Mathéa Pietri, Anne Baudry, Anne-Marie Haeberlé, Yannick Bailly, Odile Kellermann, Jean-Marie Launay, Benoit Schneider

微生物学

发表日期：2019

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Protective role of cellular prion protein against TNFα-mediated inflammation through TACE α-secretase

细胞朊病毒蛋白通过 TACE α-分泌酶对 TNFα 介导的炎症的保护作用

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-017-08110-x

Juliette Ezpeleta, François Boudet-Devaud, Mathéa Pietri, Anne Baudry, Vincent Baudouin, Aurélie Alleaume-Butaux, Nathalie Dagoneau, Odile Kellermann, Jean-Marie Launay, Benoit Schneider

Immunology/Inflammation

发表日期：2017

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Double-Edge Sword of Sustained ROCK Activation in Prion Diseases through Neuritogenesis Defects and Prion Accumulation

朊病毒疾病中持续的 ROCK 激活是一把双刃剑，它通过神经突生成缺陷和朊病毒积累发挥作用

期刊:PLoS Pathogens

影响因子:4.9

doi:10.1371/journal.ppat.1005073

Alleaume-Butaux, Aurélie; Nicot, Simon; Pietri, Mathéa; Baudry, Anne; Dakowski, Caroline; Tixador, Philippe; Ardila-Osorio, Hector; Haeberlé, Anne-Marie; Bailly, Yannick; Peyrin, Jean-Michel; Launay, Jean-Marie; Kellermann, Odile; Schneider, Benoit

Neuritogenesis: the prion protein controls β1 integrin signaling activity

神经突发生：朊病毒蛋白控制 β1 整合素信号传导活性

期刊:FASEB Journal

影响因子:4.4

doi:10.1096/fj.11-185579

Damien Loubet, Caroline Dakowski, Mathéa Pietri, Elodie Pradines, Sophie Bernard, Jacques Callebert, Hector Ardila-Osorio, Sophie Mouillet-Richard, Jean-Marie Launay, Odile Kellermann, Benoit Schneider

The cellular prion protein interacts with the tissue non-specific alkaline phosphatase in membrane microdomains of bioaminergic neuronal cells

细胞朊病毒蛋白与生物胺能神经元细胞膜微区中的组织非特异性碱性磷酸酶相互作用

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0006497

Myriam Ermonval, Anne Baudry, Florence Baychelier, Elodie Pradines, Mathéa Pietri, Kimimitsu Oda, Benoît Schneider, Sophie Mouillet-Richard, Jean-Marie Launay, Odile Kellermann

NADPH oxidase and extracellular regulated kinases 1/2 are targets of prion protein signaling in neuronal and nonneuronal cells

NADPH氧化酶和细胞外信号调节激酶1/2是朊病毒蛋白信号传导在神经元和非神经元细胞中的靶点。

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.2235648100

Schneider, Benoît; Mutel, Vincent; Pietri, Mathéa; Ermonval, Myriam; Mouillet-Richard, Sophie; Kellermann, Odile

Post-translational modifications in prion diseases

朊病毒疾病中的翻译后修饰

Titanium dioxide and carbon black nanoparticles disrupt neuronal homeostasis via excessive activation of cellular prion protein signaling

二氧化钛和炭黑纳米颗粒通过过度激活细胞朊病毒蛋白信号破坏神经元稳态

Loss of prion protein control of glucose metabolism promotes neurodegeneration in model of prion diseases

朊病毒蛋白对葡萄糖代谢的控制丧失会促进朊病毒疾病模型中的神经退行性变。

The Cellular Prion Protein-ROCK Connection: Contribution to Neuronal Homeostasis and Neurodegenerative Diseases

细胞朊蛋白与ROCK蛋白的关联：对神经元稳态和神经退行性疾病的影响

Production of seedable Amyloid-β peptides in model of prion diseases upon PrPSc-induced PDK1 overactivation

PrPSc 诱导 PDK1 过度激活后，朊病毒疾病模型中可产生可播种的淀粉样β肽

Protective role of cellular prion protein against TNFα-mediated inflammation through TACE α-secretase

细胞朊病毒蛋白通过 TACE α-分泌酶对 TNFα 介导的炎症的保护作用

Double-Edge Sword of Sustained ROCK Activation in Prion Diseases through Neuritogenesis Defects and Prion Accumulation

朊病毒疾病中持续的 ROCK 激活是一把双刃剑，它通过神经突生成缺陷和朊病毒积累发挥作用

Neuritogenesis: the prion protein controls β1 integrin signaling activity

神经突发生：朊病毒蛋白控制 β1 整合素信号传导活性

The cellular prion protein interacts with the tissue non-specific alkaline phosphatase in membrane microdomains of bioaminergic neuronal cells

细胞朊病毒蛋白与生物胺能神经元细胞膜微区中的组织非特异性碱性磷酸酶相互作用

NADPH oxidase and extracellular regulated kinases 1/2 are targets of prion protein signaling in neuronal and nonneuronal cells

NADPH氧化酶和细胞外信号调节激酶1/2是朊病毒蛋白信号传导在神经元和非神经元细胞中的靶点。