Mathias Richard T相关文献与解析，生知库

Shihan Mahbubul H, Balasubramanian Ramachandran, Wang Yan, Rafi Rabiul, Faranda Adam P, Parreno Justin, Varadaraj Kulandaiappan, Gao Junyuan, Mathias Richard T, Nie Xingju, Duncan Melinda K

CD24

CD2

发表日期：2025

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Effect of hydrogen peroxide on lens transparency, intracellular pH, gap junction coupling, hydrostatic pressure and membrane water permeability

过氧化氢对晶状体透明度、细胞内pH值、间隙连接耦合、静水压力和膜水通透性的影响

期刊:Experimental Eye Research

影响因子:2.7

doi:10.1016/j.exer.2024.109957

Varadaraj, Kulandaiappan; Gao, Junyuan; Mathias, Richard T; Kumari, Sindhu

细胞生物学

发表日期：2024

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Loss of fiber cell communication may contribute to the development of cataracts of many different etiologies

纤维细胞通讯的丧失可能导致多种不同病因引起的白内障的发生。

期刊:Frontiers in Physiology

影响因子:3.4

doi:10.3389/fphys.2022.989524

Beyer, Eric C; Mathias, Richard T; Berthoud, Viviana M

Connexin Mutants Compromise the Lens Circulation and Cause Cataracts through Biomineralization

连接蛋白突变体通过生物矿化作用损害晶状体循环并导致白内障。

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms21165822

Berthoud, Viviana M; Gao, Junyuan; Minogue, Peter J; Jara, Oscar; Mathias, Richard T; Beyer, Eric C

白内障

发表日期：2020

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The Connexin50D47A Mutant Causes Cataracts by Calcium Precipitation

连接蛋白50D47A突变体通过钙沉淀引起白内障

期刊:Investigative Ophthalmology & Visual Science

影响因子:4.7

doi:10.1167/iovs.18-26459

Berthoud, Viviana M; Gao, Junyuan; Minogue, Peter J; Jara, Oscar; Mathias, Richard T; Beyer, Eric C

白内障

发表日期：2019

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C-Terminal End of Aquaporin 0 Regulates Lens Gap Junction Channel Function

水通道蛋白0的C端调节晶状体间隙连接通道的功能

期刊:Investigative Ophthalmology & Visual Science

影响因子:4.7

doi:10.1167/iovs.19-26787

Varadaraj, Kulandaiappan; Gao, Junyuan; Mathias, Richard T; Kumari, Sindhu

发表日期：2019

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Aquaporin 0 Modulates Lens Gap Junctions in the Presence of Lens-Specific Beaded Filament Proteins

水通道蛋白0在晶状体特异性串珠丝状蛋白存在的情况下调节晶状体间隙连接

期刊:Investigative Ophthalmology & Visual Science

影响因子:4.7

doi:10.1167/iovs.17-22153

Kumari, Sindhu; Gao, Junyuan; Mathias, Richard T; Sun, Xiurong; Eswaramoorthy, Amizhdini; Browne, Nicholas; Zhang, Nigel; Varadaraj, Kulandaiappan

发表日期：2017

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Lens ion homeostasis relies on the assembly and/or stability of large connexin 46 gap junction plaques on the broad sides of differentiating fiber cells.

晶状体离子稳态依赖于分化纤维细胞宽面上的大型连接蛋白 46 间隙连接斑块的组装和/或稳定性

期刊:American Journal of Physiology-Cell Physiology

影响因子:4.7

doi:10.1152/ajpcell.00372.2014

Cheng Catherine, Nowak Roberta B, Gao Junyuan, Sun Xiurong, Biswas Sondip K, Lo Woo-Kuen, Mathias Richard T, Fowler Velia M

细胞生物学

发表日期：2015

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Feedback Regulation of Intracellular Hydrostatic Pressure in Surface Cells of the Lens

晶状体表面细胞内静水压的反馈调节

期刊:Biophysical Journal

影响因子:3.1

doi:10.1016/j.bpj.2015.09.018

Gao, Junyuan; Sun, Xiurong; White, Thomas W; Delamere, Nicholas A; Mathias, Richard T

细胞生物学

发表日期：2015

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Studies on Hydrostatic Pressure Responses to Piezo1 and TRPV4 in the Mouse Lens

小鼠晶状体对 Piezo1 和 TRPV4 的静水压力反应研究

CD24 is required for sustained transparency of the adult lens.

CD24 是维持成人晶状体透明度所必需的

Effect of hydrogen peroxide on lens transparency, intracellular pH, gap junction coupling, hydrostatic pressure and membrane water permeability

过氧化氢对晶状体透明度、细胞内pH值、间隙连接耦合、静水压力和膜水通透性的影响

Loss of fiber cell communication may contribute to the development of cataracts of many different etiologies

纤维细胞通讯的丧失可能导致多种不同病因引起的白内障的发生。

Connexin Mutants Compromise the Lens Circulation and Cause Cataracts through Biomineralization

连接蛋白突变体通过生物矿化作用损害晶状体循环并导致白内障。

The Connexin50D47A Mutant Causes Cataracts by Calcium Precipitation

连接蛋白50D47A突变体通过钙沉淀引起白内障

C-Terminal End of Aquaporin 0 Regulates Lens Gap Junction Channel Function

水通道蛋白0的C端调节晶状体间隙连接通道的功能

Aquaporin 0 Modulates Lens Gap Junctions in the Presence of Lens-Specific Beaded Filament Proteins

水通道蛋白0在晶状体特异性串珠丝状蛋白存在的情况下调节晶状体间隙连接

Lens ion homeostasis relies on the assembly and/or stability of large connexin 46 gap junction plaques on the broad sides of differentiating fiber cells.

晶状体离子稳态依赖于分化纤维细胞宽面上的大型连接蛋白 46 间隙连接斑块的组装和/或稳定性

Feedback Regulation of Intracellular Hydrostatic Pressure in Surface Cells of the Lens

晶状体表面细胞内静水压的反馈调节