日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Lipofuscin accumulation in aging and neurodegeneration: a potential "timebomb" overlooked in Alzheimer's disease

衰老和神经退行性疾病中的脂褐素积累:阿尔茨海默病中被忽视的潜在“定时炸弹”

Dougnon, Godfried; Matsui, Hideaki

Behavioral and molecular insights into anxiety in ube3a and fmr1 zebrafish models of autism spectrum disorders

ube3a 和 fmr1 斑马鱼自闭症谱系障碍模型中焦虑的行为和分子机制研究

Dougnon, Godfried; Matsui, Hideaki

Exploring α-Syn's Functions Through Ablation Models: Physiological and Pathological Implications

通过消融模型探索α-突触核蛋白的功能:生理和病理意义

Praveen, Anjali; Dougnon, Godfried; Matsui, Hideaki

Three decades of neuroscience research using animal models of ADHD and ASD: a bibliometric analysis

利用动物模型研究多动症和自闭症谱系障碍的神经科学研究三十年:文献计量分析

Dougnon, Godfried; Matsui, Hideaki

Lipofuscin autofluorescence confounds intracellular amyloid β detection in the aged mouse brain.

老年小鼠脑内脂褐素自发荧光会干扰细胞内淀粉样蛋白β的检测

Dougnon Godfried, Matsui Hideaki

A robust protocol for proteomic profiling of secreted proteins in conditioned culture medium.

一种用于对条件培养基中分泌蛋白进行蛋白质组学分析的可靠方案。

Otsuka Takayoshi, Hatano Atsushi, Matsumoto Masaki, Matsui Hideaki

Age-related TFEB downregulation in proximal tubules causes systemic metabolic disorders and occasional apolipoprotein A4-related amyloidosis

近端肾小管中与年龄相关的TFEB下调会导致全身代谢紊乱,并偶发载脂蛋白A4相关的淀粉样变性。

Jun Nakamura ,Takeshi Yamamoto ,Yoshitsugu Takabatake ,Tomoko Namba-Hamano ,Atsushi Takahashi ,Jun Matsuda ,Satoshi Minami ,Shinsuke Sakai ,Hiroaki Yonishi ,Shihomi Maeda ,Sho Matsui ,Hideaki Kawai ,Isao Matsui ,Tadashi Yamamuro ,Ryuya Edahiro ,Seiji Takashima ,Akira Takasawa ,Yukinori Okada ,Tamotsu Yoshimori ,Andrea Ballabio ,Yoshitaka Isaka

Current trends in basic research on Parkinson's disease: from mitochondria, lysosome to α-synuclein

帕金森病基础研究的最新进展:从线粒体、溶酶体到α-突触核蛋白

Matsui, Hideaki; Takahashi, Ryosuke

Homozygous slc25a20 zebrafish mutant reveals insights into carnitine-acylcarnitine translocase deficiency pathogenesis

纯合slc25a20斑马鱼突变体揭示了肉碱-酰基肉碱转位酶缺乏症的发病机制

Hishida, Ryuichi; Ishiguro, Kohei; Yamanaka, Tomoyuki; Toyokuni, Shinya; Matsui, Hideaki

Phosphorylation of α-synuclein at T64 results in distinct oligomers and exerts toxicity in models of Parkinson's disease

α-突触核蛋白在T64位点的磷酸化会导致不同的寡聚体形成,并在帕金森病模型中发挥毒性作用。

Matsui, Hideaki; Ito, Shinji; Matsui, Hideki; Ito, Junko; Gabdulkhaev, Ramil; Hirose, Mika; Yamanaka, Tomoyuki; Koyama, Akihide; Kato, Taisuke; Tanaka, Maiko; Uemura, Norihito; Matsui, Noriko; Hirokawa, Sachiko; Yoshihama, Maki; Shimozawa, Aki; Kubo, Shin-Ichiro; Iwasaki, Kenji; Hasegawa, Masato; Takahashi, Ryosuke; Hirai, Keisuke; Kakita, Akiyoshi; Onodera, Osamu