Matundan相关文献与解析，生知库 | 链接生命科学的每一步

Blocking Autophagy in M1 Macrophages Enhances Virus Replication and Eye Disease in Ocularly Infected Transgenic Mice

阻断 M1 巨噬细胞的自噬可增强眼部感染转基因小鼠的病毒复制和眼部疾病

期刊:Journal of Virology

影响因子:4

doi:10.1128/jvi.01401-22

Ujjaldeep Jaggi, Harry H Matundan, Dhong Hyun Lee, Homayon Ghiasi

Small Noncoding RNA (sncRNA1) within the Latency-Associated Transcript Modulates Herpes Simplex Virus 1 Virulence and the Host Immune Response during Acute but Not Latent Infection

潜伏期相关转录本中的小非编码RNA（sncRNA1）在急性感染期而非潜伏期感染期调节单纯疱疹病毒1型的毒力和宿主免疫反应

期刊:Journal of Virology

影响因子:3.8

doi:10.1128/jvi.00054-22

Tormanen, Kati; Matundan, Harry H; Wang, Shaohui; Jaggi, Ujjaldeep; Mott, Kevin R; Ghiasi, Homayon

Herpes Simplex Virus 1 Small Noncoding RNAs 1 and 2 Activate the Herpesvirus Entry Mediator Promoter

单纯疱疹病毒1型小非编码RNA1和2激活疱疹病毒进入介质启动子

期刊:Journal of Virology

影响因子:3.8

doi:10.1128/JVI.01985-21

Tormanen, Kati; Wang, Shaohui; Matundan, Harry H; Yu, Jack; Jaggi, Ujjaldeep; Ghiasi, Homayon

Herpes Simplex Virus 1 Glycoproteins Differentially Regulate the Activity of Costimulatory Molecules and T Cells

单纯疱疹病毒 1 型糖蛋白以差异方式调节共刺激分子和 T 细胞的活性

期刊:mSphere

影响因子:3.7

doi:10.1128/msphere.00382-22

Harry H Matundan, Ujjaldeep Jaggi, Homayon Ghiasi

Essential role of M1 macrophages in blocking cytokine storm and pathology associated with murine HSV-1 infection

M1型巨噬细胞在阻断与小鼠HSV-1感染相关的细胞因子风暴和病理过程中发挥着至关重要的作用

期刊:PLoS Pathogens

影响因子:5.5

doi:10.1371/journal.ppat.1009999

Ujjaldeep Jaggi ，Harry H Matundan ，Jack Yu ，Satoshi Hirose ，Mathias Mueller ，Floyd L Wormley Jr ，Homayon Ghiasi

Absence of CD28-CTLA4-PD-L1 Costimulatory Molecules Reduces Herpes Simplex Virus 1 Reactivation

CD28-CTLA4-PD-L1共刺激分子的缺失会降低单纯疱疹病毒1型的再激活

期刊:mBio

影响因子:4.7

doi:10.1128/mBio.01176-21

Matundan, Harry H; Jaggi, Ujjaldeep; Yu, Jack; Akbari, Omid; Ghiasi, Homayon

Suppression of CD80 Expression by ICP22 Affects Herpes Simplex Virus Type 1 Replication and CD8+IFN-γ+ Infiltrates in the Eyes of Infected Mice but Not Latency Reactivation

ICP22抑制CD80表达会影响单纯疱疹病毒1型复制和感染小鼠眼部CD8+IFN-γ+浸润，但不会影响潜伏期病毒的激活。

期刊:Journal of Virology

影响因子:4

doi:10.1128/JVI.01036-21

Harry H Matundan ，Shaohui Wang ，Ujjaldeep Jaggi ，Jack Yu ，Homayon Ghiasi

A retrospective real-world analysis of placental-based allografts on pressure ulcers

一项回顾性真实世界研究分析了胎盘来源同种异体移植对压疮的疗效

Absence of CD80 reduces HSV-1 replication in the eye and delays reactivation but not latency levels

CD80 的缺失会降低眼部 HSV-1 的复制并延缓病毒的再激活，但不会降低潜伏期水平。

IL-17A expression by both T cells and non-T cells contribute to HSV-IL-2-induced CNS demyelination

T细胞和非T细胞表达的IL-17A均促进HSV-IL-2诱导的中枢神经系统脱髓鞘。

Blocking Autophagy in M1 Macrophages Enhances Virus Replication and Eye Disease in Ocularly Infected Transgenic Mice

阻断 M1 巨噬细胞的自噬可增强眼部感染转基因小鼠的病毒复制和眼部疾病

Small Noncoding RNA (sncRNA1) within the Latency-Associated Transcript Modulates Herpes Simplex Virus 1 Virulence and the Host Immune Response during Acute but Not Latent Infection

潜伏期相关转录本中的小非编码RNA（sncRNA1）在急性感染期而非潜伏期感染期调节单纯疱疹病毒1型的毒力和宿主免疫反应

Herpes Simplex Virus 1 Small Noncoding RNAs 1 and 2 Activate the Herpesvirus Entry Mediator Promoter

单纯疱疹病毒1型小非编码RNA1和2激活疱疹病毒进入介质启动子

Herpes Simplex Virus 1 Glycoproteins Differentially Regulate the Activity of Costimulatory Molecules and T Cells

单纯疱疹病毒 1 型糖蛋白以差异方式调节共刺激分子和 T 细胞的活性

Essential role of M1 macrophages in blocking cytokine storm and pathology associated with murine HSV-1 infection

M1型巨噬细胞在阻断与小鼠HSV-1感染相关的细胞因子风暴和病理过程中发挥着至关重要的作用

Absence of CD28-CTLA4-PD-L1 Costimulatory Molecules Reduces Herpes Simplex Virus 1 Reactivation

CD28-CTLA4-PD-L1共刺激分子的缺失会降低单纯疱疹病毒1型的再激活

Suppression of CD80 Expression by ICP22 Affects Herpes Simplex Virus Type 1 Replication and CD8+IFN-γ+ Infiltrates in the Eyes of Infected Mice but Not Latency Reactivation

ICP22抑制CD80表达会影响单纯疱疹病毒1型复制和感染小鼠眼部CD8+IFN-γ+浸润，但不会影响潜伏期病毒的激活。