Mavroidis Manolis相关文献与解析，生知库

Crosstalk between coagulation and complement activation promotes cardiac dysfunction in arrhythmogenic right ventricular cardiomyopathy

凝血和补体激活之间的相互作用促进致心律失常性右室心肌病中的心脏功能障碍。

Ren, Jie; Tsilafakis, Konstantinos; Chen, Liang; Lekkos, Konstantinos; Kostavasili, Ioanna; Varela, Aimilia; Cokkinos, Dennis V; Davos, Constantinos H; Sun, Xiaogang; Song, Jiangping; Mavroidis, Manolis

心血管

Intermediate filaments in cardiomyopathy

心肌病中的中间丝

期刊:Biophysical Reviews

影响因子:3.7

doi:10.1007/s12551-018-0443-2

Tsikitis, Mary; Galata, Zoi; Mavroidis, Manolis; Psarras, Stelios; Capetanaki, Yassemi

Expression of Toll-like receptors (TLRs) in the lungs of an experimental sepsis mouse model

实验性脓毒症小鼠模型肺部 Toll 样受体 (TLR) 的表达

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0188050

Bakopoulos, Anargyros; Kapelouzou, Alkistis; Tsilimigras, Diamantis I; Katsimpoulas, Michalis; Schizas, Dimitrios; Aravanis, Chrysostomos; Balafas, Evaggelos; Mavroidis, Manolis; Pavlakis, Kitty; Machairas, Anastasios; Liakakos, Theodore

A missense mutation in desmin tail domain linked to human dilated cardiomyopathy promotes cleavage of the head domain and abolishes its Z-disc localization.

与人类扩张型心肌病相关的结蛋白尾部结构域的错义突变促进头部结构域的切割，并消除其在 Z 盘中的定位

期刊:FASEB Journal

影响因子:4.2

doi:10.1096/fj.07-088724

Mavroidis Manolis, Panagopoulou Panagiota, Kostavasili Ioanna, Weisleder Noah, Capetanaki Yassemi

Extensive induction of important mediators of fibrosis and dystrophic calcification in desmin-deficient cardiomyopathy.

肌球蛋白缺乏性心肌病中纤维化和营养不良性钙化的重要介质的广泛诱导

期刊:American Journal of Pathology

影响因子:3.6

doi:10.1016/S0002-9440(10)64916-4

Mavroidis Manolis, Capetanaki Yassemi

心血管

发表日期：2002

文献求助收藏

Are the Head and Tail Domains of Intermediate Filaments Really Unstructured Regions?

中间丝的头部和尾部结构域真的是无结构区域吗？

Crosstalk between coagulation and complement activation promotes cardiac dysfunction in arrhythmogenic right ventricular cardiomyopathy

凝血和补体激活之间的相互作用促进致心律失常性右室心肌病中的心脏功能障碍。

Intermediate filaments in cardiomyopathy

心肌病中的中间丝

Expression of Toll-like receptors (TLRs) in the lungs of an experimental sepsis mouse model

实验性脓毒症小鼠模型肺部 Toll 样受体 (TLR) 的表达

A missense mutation in desmin tail domain linked to human dilated cardiomyopathy promotes cleavage of the head domain and abolishes its Z-disc localization.

与人类扩张型心肌病相关的结蛋白尾部结构域的错义突变促进头部结构域的切割，并消除其在 Z 盘中的定位

Extensive induction of important mediators of fibrosis and dystrophic calcification in desmin-deficient cardiomyopathy.

肌球蛋白缺乏性心肌病中纤维化和营养不良性钙化的重要介质的广泛诱导