日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Retraction Note: Dclk1, a tumor stem cell marker, regulates pro-survival signaling and self-renewal of intestinal tumor cells

撤稿声明:Dclk1是一种肿瘤干细胞标志物,可调节肠道肿瘤细胞的促生存信号传导和自我更新。

Chandrakesan, Parthasarathy; Yao, Jiannan; Qu, Dongfeng; May, Randal; Weygant, Nathaniel; Ge, Yang; Ali, Naushad; Sureban, Sripathi M; Gude, Modhi; Vega, Kenneth; Bannerman-Menson, Eddie; Xia, Lijun; Bronze, Michael; An, Guangyu; Houchen, Courtney W

A novel D-peptide modulates DCLK1 gelsolin interactions, reducing PDAC tumor growth.

一种新型 D 肽调节 DCLK1 凝溶胶蛋白相互作用,从而减少 PDAC 肿瘤生长。

Moore Landon L, Qu Dongfeng, Chandrekesan Parthasarathy, Pitts Kamille, May Randal, Anderson Byron E, Brown Milton L, Houchen Courtney W

A Novel D-peptide modulates DCLK1 Gelsolin interactions, reducing PDAC tumor growth.

一种新型D肽调节DCLK1与凝溶胶蛋白的相互作用,从而减少PDAC肿瘤的生长

Moore Landon L, Qu Dongfeng, Chandrekesan Parthasarathy, Pitts Kamille, May Randal, Anderson Byron E, Brown Milton, Houchen Courtney W

DCLK1-Isoform2 Alternative Splice Variant Promotes Pancreatic Tumor Immunosuppressive M2-Macrophage Polarization

DCLK1-Isoform2 选择性剪接变体促进胰腺肿瘤免疫抑制性 M2 巨噬细胞极化

Chandrakesan, Parthasarathy; Panneerselvam, Janani; May, Randal; Weygant, Nathaniel; Qu, Dongfeng; Berry, William R; Pitts, Kamille; Stanger, Ben Z; Rao, Chinthalapally V; Bronze, Michael S; Houchen, Courtney W

Alternative splice variants of DCLK1 mark cancer stem cells, promote self-renewal and drug-resistance, and can be targeted to inhibit tumorigenesis in kidney cancer

DCLK1的替代剪接变体可标记癌症干细胞,促进自我更新和耐药性,并可作为抑制肾癌肿瘤发生的靶点。

Ge, Yang; Weygant, Nathaniel; Qu, Dongfeng; May, Randal; Berry, William L; Yao, Jiannan; Chandrakesan, Parthasarathy; Zheng, Wei; Zhao, Lichao; Zhao, Karena L; Drake, Michael; Vega, Kenneth J; Bronze, Michael S; Tomasek, James J; An, Guangyu; Houchen, Courtney W

Dclk1, a tumor stem cell marker, regulates pro-survival signaling and self-renewal of intestinal tumor cells

Dclk1是一种肿瘤干细胞标志物,它调控肠道肿瘤细胞的促生存信号传导和自我更新。

Chandrakesan, Parthasarathy; Yao, Jiannan; Qu, Dongfeng; May, Randal; Weygant, Nathaniel; Ge, Yang; Ali, Naushad; Sureban, Sripathi M; Gude, Modhi; Vega, Kenneth; Bannerman-Menson, Eddie; Xia, Lijun; Bronze, Michael; An, Guangyu; Houchen, Courtney W

Dclk1 Defines Quiescent Pancreatic Progenitors that Promote Injury-Induced Regeneration and Tumorigenesis

Dclk1 定义了静止的胰腺祖细胞,这些祖细胞促进损伤诱导的再生和肿瘤发生

Westphalen, C Benedikt; Takemoto, Yoshihiro; Tanaka, Takayuki; Macchini, Marina; Jiang, Zhengyu; Renz, Bernhard W; Chen, Xiaowei; Ormanns, Steffen; Nagar, Karan; Tailor, Yagnesh; May, Randal; Cho, Youngjin; Asfaha, Samuel; Worthley, Daniel L; Hayakawa, Yoku; Urbanska, Aleksandra M; Quante, Michael; Reichert, Maximilian; Broyde, Joshua; Subramaniam, Prem S; Remotti, Helen; Su, Gloria H; Rustgi, Anil K; Friedman, Richard A; Honig, Barry; Califano, Andrea; Houchen, Courtney W; Olive, Kenneth P; Wang, Timothy C

Survival of Patients with Gastrointestinal Cancers Can Be Predicted by a Surrogate microRNA Signature for Cancer Stem-like Cells Marked by DCLK1 Kinase

胃肠道癌症患者的生存期可通过DCLK1激酶标记的癌症干细胞样细胞的替代microRNA特征进行预测

Weygant, Nathaniel; Ge, Yang; Qu, Dongfeng; Kaddis, John S; Berry, William L; May, Randal; Chandrakesan, Parthasarathy; Bannerman-Menson, Edwin; Vega, Kenneth J; Tomasek, James J; Bronze, Michael S; An, Guangyu; Houchen, Courtney W

DCLK1 is detectable in plasma of patients with Barrett's esophagus and esophageal adenocarcinoma

在巴雷特食管和食管腺癌患者的血浆中可以检测到DCLK1。

Whorton, Joshua; Sureban, Sripathi M; May, Randal; Qu, Dongfeng; Lightfoot, Stan A; Madhoun, Mohammad; Johnson, Milton; Tierney, William M; Maple, John T; Vega, Kenneth J; Houchen, Courtney W

Plasma DCLK1 is a marker of hepatocellular carcinoma (HCC): Targeting DCLK1 prevents HCC tumor xenograft growth via a microRNA-dependent mechanism

血浆DCLK1是肝细胞癌(HCC)的标志物:靶向DCLK1可通过microRNA依赖性机制抑制HCC肿瘤异种移植瘤的生长。

Sureban, Sripathi M; Madhoun, Mohammad F; May, Randal; Qu, Dongfeng; Ali, Naushad; Fazili, Javid; Weygant, Nathaniel; Chandrakesan, Parthasarathy; Ding, Kai; Lightfoot, Stanley A; Houchen, Courtney W