McRae Steven相关文献与解析，生知库 | 链接生命科学的每一步

Withdrawal: The hepatitis C virus-induced NLRP3 inflammasome activates the sterol regulatory element-binding protein (SREBP) and regulates lipid metabolism

撤药反应：丙型肝炎病毒诱导的NLRP3炎症小体激活甾醇调节元件结合蛋白（SREBP），从而调节脂质代谢。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.W118.006979

McRae, Steven; Iqbal, Jawed; Sarkar-Dutta, Mehuli; Lane, Samantha; Nagaraj, Abhiram; Ali, Naushad; Waris, Gulam

Withdrawal: Mechanism of hepatitis C virus (HCV)-induced osteopontin and its role in epithelial to mesenchymal transition of hepatocytes

撤退：丙型肝炎病毒（HCV）诱导的骨桥蛋白的机制及其在肝细胞上皮间质转化中的作用

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.W118.006978

Iqbal, Jawed; McRae, Steven; Banaudha, Krishna; Mai, Thi; Waris, Gulam

The Hepatitis C Virus-induced NLRP3 Inflammasome Activates the Sterol Regulatory Element-binding Protein (SREBP) and Regulates Lipid Metabolism.

丙型肝炎病毒诱导的 NLRP3 炎症小体激活甾醇调节元件结合蛋白 (SREBP) 并调节脂质代谢

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M115.694059

McRae Steven, Iqbal Jawed, Sarkar-Dutta Mehuli, Lane Samantha, Nagaraj Abhiram, Ali Naushad, Waris Gulam

Hepatitis C virus activates interleukin-1Î² via caspase-1-inflammasome complex.

丙型肝炎病毒通过 caspase-1-炎症小体复合物激活白细胞介素-1β

期刊:Journal of General Virology

影响因子:4.3

doi:10.1099/vir.0.034033-0

Burdette Dylan, Haskett Adam, Presser Lance, McRae Steven, Iqbal Jawed, Waris Gulam

Hyperuricemia as a mediator of the proinflammatory endocrine imbalance in the adipose tissue in a murine model of the metabolic syndrome

在代谢综合征小鼠模型中，高尿酸血症是脂肪组织中促炎性内分泌失衡的介质。

期刊:Diabetes

影响因子:7.5

doi:10.2337/db10-0916

Baldwin, William; McRae, Steven; Marek, George; Wymer, David; Pannu, Varinderpal; Baylis, Chris; Johnson, Richard J; Sautin, Yuri Y

An RNA guanine quadruplex regulated pathway to TRAIL-sensitization by DDX21

RNA 鸟嘌呤四链体调控 DDX21 对 TRAIL 敏感的通路

Withdrawal: The hepatitis C virus-induced NLRP3 inflammasome activates the sterol regulatory element-binding protein (SREBP) and regulates lipid metabolism

撤药反应：丙型肝炎病毒诱导的NLRP3炎症小体激活甾醇调节元件结合蛋白（SREBP），从而调节脂质代谢。

Withdrawal: Mechanism of hepatitis C virus (HCV)-induced osteopontin and its role in epithelial to mesenchymal transition of hepatocytes

撤退：丙型肝炎病毒（HCV）诱导的骨桥蛋白的机制及其在肝细胞上皮间质转化中的作用

The Hepatitis C Virus-induced NLRP3 Inflammasome Activates the Sterol Regulatory Element-binding Protein (SREBP) and Regulates Lipid Metabolism.

丙型肝炎病毒诱导的 NLRP3 炎症小体激活甾醇调节元件结合蛋白 (SREBP) 并调节脂质代谢

Hepatitis C virus activates interleukin-1Î² via caspase-1-inflammasome complex.

丙型肝炎病毒通过 caspase-1-炎症小体复合物激活白细胞介素-1β

Hyperuricemia as a mediator of the proinflammatory endocrine imbalance in the adipose tissue in a murine model of the metabolic syndrome

在代谢综合征小鼠模型中，高尿酸血症是脂肪组织中促炎性内分泌失衡的介质。