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The soluble G protein of respiratory syncytial virus promotes viral dissemination via TLR2-mediated NLRP3 priming and pyroptosis.

呼吸道合胞病毒的可溶性 G 蛋白通过 TLR2 介导的 NLRP3 启动和细胞焦亡促进病毒传播。

期刊:npj Viruses

影响因子:

doi:10.1038/s44298-026-00172-x

Meineke Robert, Agac Ayse, Knittler Marie-Christin, Osterhaus Albert D M E, Ludlow Martin, Rimmelzwaan Guus F

STAT1 signaling controls cholesterol metabolism in epithelial cells and RSV-induced syncytia formation.

STAT1 信号传导控制上皮细胞中的胆固醇代谢和 RSV 诱导的合胞体形成。

期刊:npj Viruses

影响因子:

doi:10.1038/s44298-026-00173-w

Agac Ayse, Ludlow Martin, Knittler Marie-Christin, Osterhaus Albert D M E, Rimmelzwaan Guus F, Meineke Robert

Selective pressure mediated by influenza virus M1(58-66) epitope-specific CD8(+)T cells promotes accumulation of extra-epitopic amino acid substitutions associated with viral resistance to these T cells

由流感病毒M1(58-66)表位特异性CD8(+)T细胞介导的选择压力促进了与病毒对这些T细胞的耐药性相关的表位外氨基酸替换的积累。

期刊:Virus Research

影响因子:2.7

doi:10.1016/j.virusres.2024.199355

Jansen, Janina M; Meineke, Robert; Molle, Antonia; van de Sandt, Carolien E; Saletti, Giulietta; Rimmelzwaan, Guus F

Respiratory syncytial virus glycoprotein G impedes CX(3)CR1-activation by CX(3)CL1 and monocyte function.

呼吸道合胞病毒糖蛋白 G 抑制 CX(3)CL1 激活 CX(3)CR1 和单核细胞功能

期刊:npj Viruses

影响因子:

doi:10.1038/s44298-024-00075-9

Meineke Robert, Agac Ayse, Knittler Marie-Christin, Ludlow Martin, Osterhaus Albert D M E, Rimmelzwaan Guus F

细胞生物学

发表日期：2024

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Host Responses to Respiratory Syncytial Virus Infection

宿主对呼吸道合胞病毒感染的反应

期刊:Viruses-Basel

影响因子:3.5

doi:10.3390/v15101999

Agac, Ayse; Kolbe, Sophie M; Ludlow, Martin; Osterhaus, Albert D M E; Meineke, Robert; Rimmelzwaan, Guus F

Influenza Virus Infections and Cellular Kinases

流感病毒感染与细胞激酶

期刊:Viruses-Basel

影响因子:3.5

doi:10.3390/v11020171

Meineke, Robert; Rimmelzwaan, Guus F; Elbahesh, Husni

Roles of the Chemokine Receptor CX3CR1 in the Pathogenesis of RSV Infections

趋化因子受体CX3CR1在呼吸道合胞病毒感染发病机制中的作用

The soluble G protein of respiratory syncytial virus promotes viral dissemination via TLR2-mediated NLRP3 priming and pyroptosis.

呼吸道合胞病毒的可溶性 G 蛋白通过 TLR2 介导的 NLRP3 启动和细胞焦亡促进病毒传播。

STAT1 signaling controls cholesterol metabolism in epithelial cells and RSV-induced syncytia formation.

STAT1 信号传导控制上皮细胞中的胆固醇代谢和 RSV 诱导的合胞体形成。

Selective pressure mediated by influenza virus M1(58-66) epitope-specific CD8(+)T cells promotes accumulation of extra-epitopic amino acid substitutions associated with viral resistance to these T cells

由流感病毒M1(58-66)表位特异性CD8(+)T细胞介导的选择压力促进了与病毒对这些T细胞的耐药性相关的表位外氨基酸替换的积累。

Respiratory syncytial virus glycoprotein G impedes CX(3)CR1-activation by CX(3)CL1 and monocyte function.

呼吸道合胞病毒糖蛋白 G 抑制 CX(3)CL1 激活 CX(3)CR1 和单核细胞功能

Host Responses to Respiratory Syncytial Virus Infection

宿主对呼吸道合胞病毒感染的反应

Influenza Virus Infections and Cellular Kinases

流感病毒感染与细胞激酶