Mialki相关文献与解析，生知库 | 链接生命科学的每一步

A blocking peptide stabilizes lysophosphatidic acid receptor 1 and promotes lysophosphatidic acid-induced cellular responses

阻断肽可稳定溶血磷脂酸受体1，并促进溶血磷脂酸诱导的细胞反应。

期刊:Journal of Cellular Biochemistry

影响因子:2.8

doi:10.1002/jcb.29919

Taleb, Sarah J; Wei, Jianxin; Mialki, Rachel K; Dong, Su; Li, Yanhui; Zhao, Jing; Zhao, Yutong

SCF E3 ligase F-box protein complex SCF(FBXL19) regulates cell migration by mediating Rac1 ubiquitination and degradation

SCF E3连接酶F-box蛋白复合物SCF(FBXL19)通过介导Rac1泛素化和降解来调控细胞迁移。

期刊:FASEB Journal

影响因子:4.2

doi:10.1096/fj.12-223099

Zhao, Jing; Mialki, Rachel K; Wei, Jianxin; Coon, Tiffany A; Zou, Chunbin; Chen, Bill B; Mallampalli, Rama K; Zhao, Yutong

Overexpression of USP14 protease reduces I-κB protein levels and increases cytokine release in lung epithelial cells

USP14蛋白酶的过表达会降低肺上皮细胞中I-κB蛋白的水平并增加细胞因子的释放。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.C112.446682

Mialki, Rachel K; Zhao, Jing; Wei, Jianxin; Mallampalli, Daniel F; Zhao, Yutong

A new mechanism of RhoA ubiquitination and degradation: roles of SCF(FBXL19) E3 ligase and Erk2

RhoA 泛素化降解的新机制：SCF(FBXL19) E3 连接酶和 Erk2 的作用

期刊:

影响因子:3.3

doi:10.1016/j.bbamcr.2013.07.005

Jianxin Wei, Rachel K Mialki, Su Dong, Andrew Khoo, Rama K Mallampalli, Yutong Zhao, Jing Zhao

F-box protein FBXL19-mediated ubiquitination and degradation of the receptor for IL-33 limits pulmonary inflammation

F-box 蛋白 FBXL19 介导的 IL-33 受体泛素化和降解可限制肺部炎症

期刊:Nature Immunology

影响因子:27.7

doi:10.1038/ni.2341

Jing Zhao, Jianxin Wei, Rachel K Mialki, Daniel F Mallampalli, Bill B Chen, Tiffany Coon, Chunbin Zou, Rama K Mallampalli, Yutong Zhao

信号转导

Immunology/Inflammation

发表日期：2012

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Depletion of Microglia Increases Cortical Oligodendrocyte Density During Remyelination

小胶质细胞减少会增加髓鞘再生过程中皮质少突胶质细胞的密度

A blocking peptide stabilizes lysophosphatidic acid receptor 1 and promotes lysophosphatidic acid-induced cellular responses

阻断肽可稳定溶血磷脂酸受体1，并促进溶血磷脂酸诱导的细胞反应。

SCF E3 ligase F-box protein complex SCF(FBXL19) regulates cell migration by mediating Rac1 ubiquitination and degradation

SCF E3连接酶F-box蛋白复合物SCF(FBXL19)通过介导Rac1泛素化和降解来调控细胞迁移。

Overexpression of USP14 protease reduces I-κB protein levels and increases cytokine release in lung epithelial cells

USP14蛋白酶的过表达会降低肺上皮细胞中I-κB蛋白的水平并增加细胞因子的释放。

A new mechanism of RhoA ubiquitination and degradation: roles of SCF(FBXL19) E3 ligase and Erk2

RhoA 泛素化降解的新机制：SCF(FBXL19) E3 连接酶和 Erk2 的作用

F-box protein FBXL19-mediated ubiquitination and degradation of the receptor for IL-33 limits pulmonary inflammation

F-box 蛋白 FBXL19 介导的 IL-33 受体泛素化和降解可限制肺部炎症