Michelucci Antonio相关文献与解析，生知库

Michelucci Antonio, Sforna Luigi, Focaia Riccardo, Leonardi Maria Vittoria, Di Battista Angela, Rastelli Giorgia, Vespa Simone, Boncompagni Simona, Di Cristina Manlio, Catacuzzeno Luigi

炎症/感染

新冠

发表日期：2025

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Piezo1 is an essential player in volume regulation of human glioblastoma cells

Piezo1是人类胶质母细胞瘤细胞体积调节的关键因子。

期刊:Journal of Physiology-London

影响因子:4.4

doi:10.1113/JP289215

Leonardi, Maria Vittoria; Di Battista, Angela; Donati, Leonardo; Sforna, Luigi; Morena, Francesco; Di Cristina, Manlio; Catacuzzeno, Luigi; Michelucci, Antonio

细胞生物学

发表日期：2025

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The crucial decade that ion channels were proven to exist : The vision of Bertil Hille and Clay Armstrong and how it came through

离子通道存在被证实的关键十年：伯蒂尔·希尔和克莱·阿姆斯特朗的远见卓识及其实现历程

期刊:Pflugers Archiv-European Journal of Physiology

影响因子:2.9

doi:10.1007/s00424-025-03085-5

Catacuzzeno, Luigi; Michelucci, Antonio; Franciolini, Fabio

发表日期：2025

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The Long Journey from Animal Electricity to the Discovery of Ion Channels and the Modelling of the Human Brain

从动物电到离子通道的发现和人脑建模的漫长旅程

期刊:Biomolecules

影响因子:4.8

doi:10.3390/biom14060684

Catacuzzeno, Luigi; Michelucci, Antonio; Franciolini, Fabio

Building predictive Markov models of ion channel permeation from molecular dynamics simulations

基于分子动力学模拟构建离子通道渗透的预测性马尔可夫模型

期刊:Biophysical Journal

影响因子:3.1

doi:10.1016/j.bpj.2024.09.030

Catacuzzeno, Luigi; Leonardi, Maria Vittoria; Franciolini, Fabio; Domene, Carmen; Michelucci, Antonio; Furini, Simone

发表日期：2024

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Hypoxia, Ion Channels and Glioblastoma Malignancy

缺氧、离子通道和胶质母细胞瘤恶性肿瘤

期刊:Biomolecules

影响因子:4.8

doi:10.3390/biom13121742

Michelucci, Antonio; Sforna, Luigi; Franciolini, Fabio; Catacuzzeno, Luigi

Cytosolic Ca(2+) gradients and mitochondrial Ca(2+) uptake in resting muscle fibers: A model analysis.

静息肌纤维中胞质 Ca(2+) 梯度和线粒体 Ca(2+) 摄取：模型分析

期刊:Biophysical Reports

影响因子:2.7

doi:10.1016/j.bpr.2023.100117

Marcucci Lorenzo, Michelucci Antonio, Reggiani Carlo

线粒体

发表日期：2023

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Ageing Causes Ultrastructural Modification to Calcium Release Units and Mitochondria in Cardiomyocytes

衰老导致心肌细胞钙释放单元和线粒体的超微结构改变

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms22168364

Di Fonso, Alessia; Pietrangelo, Laura; D'Onofrio, Laura; Michelucci, Antonio; Boncompagni, Simona; Protasi, Feliciano

Long-Term Exercise Reduces Formation of Tubular Aggregates and Promotes Maintenance of Ca(2+) Entry Units in Aged Muscle

长期锻炼可减少老年肌肉中管状聚集体的形成，并促进Ca(2+)内流单元的维持。

期刊:Frontiers in Physiology

影响因子:3.4

doi:10.3389/fphys.2020.601057

Boncompagni, Simona; Pecorai, Claudia; Michelucci, Antonio; Pietrangelo, Laura; Protasi, Feliciano

发表日期：2020

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Pre-assembled Ca2+ entry units and constitutively active Ca2+ entry in skeletal muscle of calsequestrin-1 knockout mice

钙结合蛋白-1敲除小鼠骨骼肌中预组装的Ca2+内流单元和组成型活性Ca2+内流

期刊:Journal of General Physiology

影响因子:2.9

doi:10.1085/jgp.202012617

Michelucci, Antonio; Boncompagni, Simona; Pietrangelo, Laura; Takano, Takahiro; Protasi, Feliciano; Dirksen, Robert T

发表日期：2020

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SARS-CoV-2 ORF3a accessory protein is a water-permeable channel that induces lysosome swelling.

SARS-CoV-2 ORF3a 辅助蛋白是一种水渗透通道，可诱导溶酶体肿胀

Piezo1 is an essential player in volume regulation of human glioblastoma cells

Piezo1是人类胶质母细胞瘤细胞体积调节的关键因子。

The crucial decade that ion channels were proven to exist : The vision of Bertil Hille and Clay Armstrong and how it came through

离子通道存在被证实的关键十年：伯蒂尔·希尔和克莱·阿姆斯特朗的远见卓识及其实现历程

The Long Journey from Animal Electricity to the Discovery of Ion Channels and the Modelling of the Human Brain

从动物电到离子通道的发现和人脑建模的漫长旅程

Building predictive Markov models of ion channel permeation from molecular dynamics simulations

基于分子动力学模拟构建离子通道渗透的预测性马尔可夫模型

Hypoxia, Ion Channels and Glioblastoma Malignancy

缺氧、离子通道和胶质母细胞瘤恶性肿瘤

Cytosolic Ca(2+) gradients and mitochondrial Ca(2+) uptake in resting muscle fibers: A model analysis.

静息肌纤维中胞质 Ca(2+) 梯度和线粒体 Ca(2+) 摄取：模型分析

Ageing Causes Ultrastructural Modification to Calcium Release Units and Mitochondria in Cardiomyocytes

衰老导致心肌细胞钙释放单元和线粒体的超微结构改变

Long-Term Exercise Reduces Formation of Tubular Aggregates and Promotes Maintenance of Ca(2+) Entry Units in Aged Muscle

长期锻炼可减少老年肌肉中管状聚集体的形成，并促进Ca(2+)内流单元的维持。

Pre-assembled Ca2+ entry units and constitutively active Ca2+ entry in skeletal muscle of calsequestrin-1 knockout mice

钙结合蛋白-1敲除小鼠骨骼肌中预组装的Ca2+内流单元和组成型活性Ca2+内流